T cell-activation in neuromyelitis optica lesions plays a role in their formation. - Wikidata - awgldk - TriplyDB

T cell-activation in neuromyelitis optica lesions plays a role in their formation.

T cell-activation in neuromyelitis optica lesions plays a role in their formation.

http://www.wikidata.org/entity/Q39039987

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Antibodies as Mediators of Brain Pathology Treatment of neuromyelitis optica: state-of-the-art and emerging therapies Advances in Autoimmune Epilepsy Associated with Antibodies, Their Potential Pathogenic Molecular Mechanisms, and Current Recommended Immunotherapies Efficacy and safety of tacrolimus treatment for neuromyelitis optica spectrum disorder Expanding Role of T Cells in Human Autoimmune Diseases of the Central Nervous System.Systemic inflammation and the brain: novel roles of genetic, molecular, and environmental cues as drivers of neurodegeneration.Neuromyelitis optica pathology in rats following intraperitoneal injection of NMO-IgG and intracerebral needle injury.Markedly increased IP-10 production by blood-brain barrier in neuromyelitis optica Experimental Neuromyelitis Optica Induces a Type I Interferon Signature in the Spinal Cord.Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNS.Severely exacerbated neuromyelitis optica rat model with extensive astrocytopathy by high affinity anti-aquaporin-4 monoclonal antibody.Aquaporin 4-specific T cells and NMO-IgG cause primary retinal damage in experimental NMO/SD Multiple sclerosis: experimental models and reality.Astrocyte barriers to neurotoxic inflammation.Neuromyelitis optica: a positive appraisal of seronegative cases.OX40, OX40L and Autoimmunity: a Comprehensive Review.Neurodegeneration in multiple sclerosis and neuromyelitis optica.Müller cells and retinal axons can be primary targets in experimental neuromyelitis optica spectrum disorder.The Role of Brain-Reactive Autoantibodies in Brain Pathology and Cognitive Impairment.Neuromyelitis optica study model based on chronic infusion of autoantibodies in rat cerebrospinal fluid.IgG and complement deposition and neuronal loss in cats and humans with epilepsy and voltage-gated potassium channel complex antibodies.Human antibodies against the myelin oligodendrocyte glycoprotein can cause complement-dependent demyelination.Association of TNFSF4 Polymorphisms with Neuromyelitis Optica Spectrum Disorders in a Chinese Population.Impact of Autologous Mesenchymal Stem Cell Infusion on Neuromyelitis Optica Spectrum Disorder: A Pilot, 2-Year Observational Study.Mechanisms for lesion localization in neuromyelitis optica spectrum disorders.Early and extensive spinal white matter involvement in neuromyelitis optica.OX40 Gene and Serum Protein Expression Profiles in Patients with Parkinson's Disease.Prediction of disease severity in neuromyelitis optica by the levels of interleukin (IL)-6 produced during remission phase.The compartmentalized inflammatory response in the multiple sclerosis brain is composed of tissue-resident CD8+ T lymphocytes and B cells.

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T cell-activation in neuromyelitis optica lesions plays a role in their formation.

http://www.wikidata.org/entity/Q39039987

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

@zh-tw

2013年论文

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2013年论文

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2013年论文

@zh-cn

name

T cell-activation in neuromyelitis optica lesions plays a role in their formation.

@en

T cell-activation in neuromyelitis optica lesions plays a role in their formation.

@nl

type

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T cell-activation in neuromyelitis optica lesions plays a role in their formation.

@en

T cell-activation in neuromyelitis optica lesions plays a role in their formation.

@nl

prefLabel

T cell-activation in neuromyelitis optica lesions plays a role in their formation.

@en

T cell-activation in neuromyelitis optica lesions plays a role in their formation.

@nl

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Acta neuropathologica communications

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T cell-activation in neuromyelitis optica lesions plays a role in their formation

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Hartmut Wekerle

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Joachim W Ellwart

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Joana Machado-Santos

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Kazuo Fujihara

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Maja Kitic

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Maria Pohl

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Marie-Therese Fischer

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Naoto Kawakami

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Tatsuro Misu

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P2860

IgG marker of optic-spinal multiple sclerosis binds to the aquaporin-4 water channel

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

A case of NMO seropositive for aquaporin-4 antibody more than 10 years before onset

A serum autoantibody marker of neuromyelitis optica: distinction from multiple sclerosis

Phenotypic and functional features of human Th17 cells

A role for humoral mechanisms in the pathogenesis of Devic's neuromyelitis optica.

Control of immunity by the TNFR-related molecule OX40 (CD134)

Intravenous neuromyelitis optica autoantibody in mice targets aquaporin-4 in peripheral organs and area postrema

Intrarenal synthesis of complement.

Presence of six different lesion types suggests diverse mechanisms of tissue injury in neuromyelitis optica

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