Monomethylarsonous acid induces transformation of human bladder cells. - Wikidata - awgldk - TriplyDB

Monomethylarsonous acid induces transformation of human bladder cells.

Monomethylarsonous acid induces transformation of human bladder cells.

http://www.wikidata.org/entity/Q33775357

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Activation of Nrf2 by arsenite and monomethylarsonous acid is independent of Keap1-C151: enhanced Keap1-Cul3 interaction Developmental and genetic modulation of arsenic biotransformation: a gene by environment interaction?Epigenetic changes during cell transformation Inorganic arsenic and human prostate cancer Nutritional status has marginal influence on the metabolism of inorganic arsenic in pregnant Bangladeshi women Breast-feeding protects against arsenic exposure in Bangladeshi infants Arsenic exposure and cancer mortality in a US-based prospective cohort: the strong heart study Low level exposure to monomethyl arsonous acid-induced the over-production of inflammation-related cytokines and the activation of cell signals associated with tumor progression in a urothelial cell model Interleukin-8 (IL-8) over-production and autocrine cell activation are key factors in monomethylarsonous acid [MMA(III)]-induced malignant transformation of urothelial cells The Use of Chemical-Chemical Interaction and Chemical Structure to Identify New Candidate Chemicals Related to Lung Cancer Methylarsonous acid causes oxidative DNA damage in cells independent of the ability to biomethylate inorganic arsenic Monomethylarsonous acid produces irreversible events resulting in malignant transformation of a human bladder cell line following 12 weeks of low-level exposure Influence of arsenate and arsenite on signal transduction pathways: an update Epigenetic mediated transcriptional activation of WNT5A participates in arsenical-associated malignant transformation A transgenic Drosophila model for arsenic methylation suggests a metabolic rationale for differential dose-dependent toxicity endpoints.Involvement of N-6 adenine-specific DNA methyltransferase 1 (N6AMT1) in arsenic biomethylation and its role in arsenic-induced toxicity Arsenic toxicology: translating between experimental models and human pathology.Global gene expression changes in human urothelial cells exposed to low-level monomethylarsonous acid.Impact of trivalent arsenicals on selenoprotein synthesis Agglomerates of aberrant DNA methylation are associated with toxicant-induced malignant transformation.Expression of AS3MT alters transcriptional profiles in human urothelial cells exposed to arsenite.Transcriptional Modulation of the ERK1/2 MAPK and NF-κB Pathways in Human Urothelial Cells After Trivalent Arsenical Exposure: Implications for Urinary Bladder Cancer.Arsenite activates NFκB through induction of C-reactive protein.Epigenetic remodeling during arsenical-induced malignant transformation.Arsenic-based antineoplastic drugs and their mechanisms of action.Hedgehog signaling regulates bladder cancer growth and tumorigenicity Exposure to monomethylarsonous acid (MMA(III)) leads to altered selenoprotein synthesis in a primary human lung cell model.Oral exposure to inorganic arsenic: evaluation of its carcinogenic and non-carcinogenic effects.Arsenicals produce stable progressive changes in DNA methylation patterns that are linked to malignant transformation of immortalized urothelial cells.Acetylated H4K16 by MYST1 protects UROtsa cells from arsenic toxicity and is decreased following chronic arsenic exposure.Requirement of arsenic biomethylation for oxidative DNA damage.Differential binding of monomethylarsonous acid compared to arsenite and arsenic trioxide with zinc finger peptides and proteins.Expression Of Selected Pathway-Marker Genes In Human Urothelial Cells Exposed Chronically To A Non-Cytotoxic Concentration Of Monomethylarsonous Acid.Oxidative DNA damage after acute exposure to arsenite and monomethylarsonous acid in biomethylation-deficient human cells.Interdependent genotoxic mechanisms of monomethylarsonous acid: role of ROS-induced DNA damage and poly(ADP-ribose) polymerase-1 inhibition in the malignant transformation of urothelial cells Cell damage and death by autoschizis in human bladder (RT4) carcinoma cells resulting from treatment with ascorbate and menadione.Persistence of DNA damage following exposure of human bladder cells to chronic monomethylarsonous acid Reactive oxygen species regulate properties of transformation in UROtsa cells exposed to monomethylarsonous acid by modulating MAPK signaling The role of reactive oxygen species in arsenite and monomethylarsonous acid-induced signal transduction in human bladder cells: acute studies.Oxidative DNA damage enhances the carcinogenic potential of in vitro chronic arsenic exposures.

P2860

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P2860

Monomethylarsonous acid induces transformation of human bladder cells.

http://www.wikidata.org/entity/Q33775357

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2006 nî lūn-bûn

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2006 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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Monomethylarsonous acid induces transformation of human bladder cells.

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Monomethylarsonous acid induces transformation of human bladder cells.

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Monomethylarsonous acid induces transformation of human bladder cells.

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J.TAAP.2006.04.011

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Toxicology and Applied Pharmacology

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Monomethylarsonous acid induces transformation of human bladder cells.

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A Jay Gandolfi

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Bhumasamudram Jagadish

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Eugene A Mash

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Kylee E Eblin

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Tiffany G Bredfeldt

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P2860

Association of chromosomal alterations with arsenite-induced tumorigenicity of human HaCaT keratinocytes in nude mice.

Determination of monomethylarsonous acid, a key arsenic methylation intermediate, in human urine

Urinary trivalent methylated arsenic species in a population chronically exposed to inorganic arsenic.

Arsenite cocarcinogenesis: an animal model derived from genetic toxicology studies

The immortalized UROtsa cell line as a potential cell culture model of human urothelium

Biokinetics and subchronic toxic effects of oral arsenite, arsenate, monomethylarsonic acid, and dimethylarsinic acid in v-Ha-ras transgenic (Tg.AC) mice.

Enzymatic reduction of arsenic compounds in mammalian systems: the rate-limiting enzyme of rabbit liver arsenic biotransformation is MMA(V) reductase

A comparative study of the sub-chronic toxic effects of three organic arsenical compounds on the urothelium in F344 rats; gender-based differences in response.

Cancers related to exposure to arsenic at a copper smelter

Monomethylarsonous acid (MMA(III)) is more toxic than arsenite in Chang human hepatocytes.

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