Deletion of tumor necrosis factor-alpha receptor 1 (TNFR1) protects against diet-induced obesity by means of increased thermogenesis. - Wikidata - awgldk - TriplyDB

Deletion of tumor necrosis factor-alpha receptor 1 (TNFR1) protects against diet-induced obesity by means of increased thermogenesis.

Deletion of tumor necrosis factor-alpha receptor 1 (TNFR1) protects against diet-induced obesity by means of increased thermogenesis.

http://www.wikidata.org/entity/Q37479267

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High-fat diet: bacteria interactions promote intestinal inflammation which precedes and correlates with obesity and insulin resistance in mouse Of mice and men: novel insights regarding constitutive and recruitable brown adipocytes Influencing Factors of Thermogenic Adipose Tissue Activity Adipose tissue and adipokines: the association with and application of adipokines in obesity IL-6 and IL-10 anti-inflammatory activity links exercise to hypothalamic insulin and leptin sensitivity through IKKbeta and ER stress inhibition Unsaturated fatty acids revert diet-induced hypothalamic inflammation in obesity The cellular and molecular bases of leptin and ghrelin resistance in obesity.Priming of metabolic dysfunctions by prenatal immune activation in mice: relevance to schizophrenia.Cinnamon induces browning in subcutaneous adipocytes.Circulating levels of tumor necrosis factor-alpha receptor 2 are increased in heart failure with preserved ejection fraction relative to heart failure with reduced ejection fraction: evidence for a divergence in pathophysiology Capsaicin induces "brite" phenotype in differentiating 3T3-L1 preadipocytes.KSRP ablation enhances brown fat gene program in white adipose tissue through reduced miR-150 expression.Negative regulators of brown adipose tissue (BAT)-mediated thermogenesis Adaptive thermogenesis in adipocytes: is beige the new brown?Prdm16 determines the thermogenic program of subcutaneous white adipose tissue in mice.Central IKKβ inhibition prevents air pollution mediated peripheral inflammation and exaggeration of type II diabetes.Moderate intensity exercise inhibits macrophage infiltration and attenuates adipocyte inflammation in ovariectomized rats Metformin attenuates the exacerbation of the allergic eosinophilic inflammation in high fat-diet-induced obesity in mice.Alzheimer-associated Aβ oligomers impact the central nervous system to induce peripheral metabolic deregulation.Cold tolerance, cold-induced hyperphagia, and nonshivering thermogenesis are normal in α₁-AMPK-/- mice Saturated fatty acids modulate autophagy's proteins in the hypothalamus Hypothalamic inflammation: a double-edged sword to nutritional diseases.Deficiency for costimulatory receptor 4-1BB protects against obesity-induced inflammation and metabolic disorders Death receptor 5 signaling promotes hepatocyte lipoapoptosis FGF21 regulates PGC-1α and browning of white adipose tissues in adaptive thermogenesis.Inflammatory cause of metabolic syndrome via brain stress and NF-κB.Inverse association between brown adipose tissue activation and white adipose tissue accumulation in successfully treated pediatric malignancy.Developments in the imaging of brown adipose tissue and its associations with muscle, puberty, and health in children.Development of insulin resistance in mice lacking PGC-1α in adipose tissues.Relevance of brown adipose tissue in infancy and adolescence.IEX-1 deficiency induces browning of white adipose tissue and resists diet-induced obesity.A high-fat/high-cholesterol diet inhibits growth of fetal hippocampal transplants via increased inflammation.TWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity.Central Leptin and Tumor Necrosis Factor-α (TNFα) in Diurnal Control of Blood Pressure and Hypertension Neuroinflammatory and autonomic mechanisms in diabetes and hypertension.Diabetic myopathy: impact of diabetes mellitus on skeletal muscle progenitor cells.Resolvin RvD2 reduces hypothalamic inflammation and rescues mice from diet-induced obesity.The Leptin Receptor Complex: Heavier Than Expected?Leptin Resistance and the Neuro-Adipose Connection.Relationship between oxidative stress and inflammatory cytokines in diabetic nephropathy.

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Deletion of tumor necrosis factor-alpha receptor 1 (TNFR1) protects against diet-induced obesity by means of increased thermogenesis.

http://www.wikidata.org/entity/Q37479267

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 26 October 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Deletion of tumor necrosis fac ...... ns of increased thermogenesis.

@en

Deletion of tumor necrosis factor-alpha receptor 1

@nl

type

label

Deletion of tumor necrosis fac ...... ns of increased thermogenesis.

@en

Deletion of tumor necrosis factor-alpha receptor 1

@nl

prefLabel

Deletion of tumor necrosis fac ...... ns of increased thermogenesis.

@en

Deletion of tumor necrosis factor-alpha receptor 1

@nl

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JBC.M109.030874

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Journal of Biological Chemistry

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Deletion of tumor necrosis fac ...... ns of increased thermogenesis.

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Ana P Arruda

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Antonio C Boschero

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Carina Solon

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Erika A Roman

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Giovanna R Degasperi

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Juliana C Moraes

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Licio A Velloso

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Marciane Milanski

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Maria L Bonfleur

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Paty K Picardi

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P2860

The intriguing biology of the tumour necrosis factor/tumour necrosis factor receptor superfamily: players, rules and the games

Inflammation and metabolic disorders

The TNF and TNF receptor superfamilies: integrating mammalian biology

Obesity wars: molecular progress confronts an expanding epidemic

Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection

Saturated fatty acids produce an inflammatory response predominantly through the activation of TLR4 signaling in hypothalamus: implications for the pathogenesis of obesity

Endoplasmic reticulum stress links obesity, insulin action, and type 2 diabetes

Protection from obesity-induced insulin resistance in mice lacking TNF-alpha function

TLR4 links innate immunity and fatty acid-induced insulin resistance

Leptin activates anorexigenic POMC neurons through a neural network in the arcuate nucleus

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36213-36222

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scholarly article

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10.1074/JBC.M109.030874

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52

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284

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Sandro M Hirabara

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2009-10-26T00:00:00Z

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19858212

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