The role of diacylglycerol kinase ζ and phosphatidic acid in the mechanical activation of mammalian target of rapamycin (mTOR) signaling and skeletal muscle hypertrophy
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Regulation of mTORC1 by growth factors, energy status, amino acids and mechanical stimuli at a glanceThe molecular basis for load-induced skeletal muscle hypertrophyAutophagy is essential to support skeletal muscle plasticity in response to endurance exerciseDiacylglycerol Kinases: Shaping Diacylglycerol and Phosphatidic Acid Gradients to Control Cell PolarityLipid modulation of skeletal muscle mass and functionMechanosensitive Molecular Networks Involved in Transducing Resistance Exercise-Signals into Muscle Protein AccretionThe role of mTOR signaling in the regulation of protein synthesis and muscle mass during immobilization in micePhosphatidic acid enhances mTOR signaling and resistance exercise induced hypertrophy.Absence of γ-sarcoglycan alters the response of p70S6 kinase to mechanical perturbation in murine skeletal muscle.G protein-coupled receptor 56 regulates mechanical overload-induced muscle hypertrophyAlcohol impairs skeletal muscle protein synthesis and mTOR signaling in a time-dependent manner following electrically stimulated muscle contractionPhospholipase D1 facilitates second-phase myoblast fusion and skeletal muscle regenerationRNA sequencing identifies upregulated kyphoscoliosis peptidase and phosphatidic acid signaling pathways in muscle hypertrophy generated by transgenic expression of myostatin propeptideThe Prion Protein N1 and N2 Cleavage Fragments Bind to Phosphatidylserine and Phosphatidic Acid; Relevance to Stress-Protection ResponsesBone and skeletal muscle: Key players in mechanotransduction and potential overlapping mechanisms.Diacylglycerol kinase-ζ regulates mTORC1 and lipogenic metabolism in cancer cells through SREBP-1The effects of phosphatidic acid supplementation on strength, body composition, muscular endurance, power, agility, and vertical jump in resistance trained men.New roles for Smad signaling and phosphatidic acid in the regulation of skeletal muscle mass.The role of mTORC1 in regulating protein synthesis and skeletal muscle mass in response to various mechanical stimuli.Role of phospholipase d in g-protein coupled receptor functionNutritional strategies to support concurrent training.Redundant and specialized roles for diacylglycerol kinases α and ζ in the control of T cell functions.The mechanistic and ergogenic effects of phosphatidic acid in skeletal muscle.Ginsenoside Rg1 prevents starvation-induced muscle protein degradation via regulation of AKT/mTOR/FoxO signaling in C2C12 myotubesDo metabolites that are produced during resistance exercise enhance muscle hypertrophy?Intramuscular Anabolic Signaling and Endocrine Response Following Resistance Exercise: Implications for Muscle Hypertrophy.Resistance exercise initiates mechanistic target of rapamycin (mTOR) translocation and protein complex co-localisation in human skeletal muscle.Cause and management of muscle wasting in chronic liver disease.AMPKγ3 is dispensable for skeletal muscle hypertrophy induced by functional overload.Phosphatidic acid: biosynthesis, pharmacokinetics, mechanisms of action and effect on strength and body composition in resistance-trained individuals.Effects of oral phosphatidic acid feeding with or without whey protein on muscle protein synthesis and anabolic signaling in rodent skeletal muscle.Perspective: The Potential Role of Essential Amino Acids and the Mechanistic Target of Rapamycin Complex 1 (mTORC1) Pathway in the Pathogenesis of Child Stunting.Identification of mechanically regulated phosphorylation sites on tuberin (TSC2) that control mechanistic target of rapamycin (mTOR) signaling.Treatment to improve nutrition and functional capacity evaluation in liver transplant candidates.mTOR as a Key Regulator in Maintaining Skeletal Muscle Mass.Increased hypertrophic response with increased mechanical load in skeletal muscles receiving identical activity patterns.DGKζ deficiency protects against peripheral insulin resistance and improves energy metabolism.How many phosphoproteins does it take to make muscle grow?Akt-dependent and Akt-independent pathways are involved in protein synthesis activation during reloading of disused soleus muscle.Mechanosensitive Ion Channels: TRPV4 and P2X7 in Disseminating Cancer Cells.
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The role of diacylglycerol kinase ζ and phosphatidic acid in the mechanical activation of mammalian target of rapamycin (mTOR) signaling and skeletal muscle hypertrophy
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 03 December 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The role of diacylglycerol kin ...... nd skeletal muscle hypertrophy
@en
The role of diacylglycerol kin ...... mammalian target of rapamycin
@nl
type
label
The role of diacylglycerol kin ...... nd skeletal muscle hypertrophy
@en
The role of diacylglycerol kin ...... mammalian target of rapamycin
@nl
prefLabel
The role of diacylglycerol kin ...... nd skeletal muscle hypertrophy
@en
The role of diacylglycerol kin ...... mammalian target of rapamycin
@nl
P2093
P2860
P356
P1476
The role of diacylglycerol kin ...... nd skeletal muscle hypertrophy
@en
P2093
Chan-Ran Kim
Hannah C Lincoln
Jae-Sung You
John W Frey
Xiao-Ping Zhong
P2860
P304
P356
10.1074/JBC.M113.531392
P407
P577
2013-12-03T00:00:00Z