Ginsenoside Rg1 prevents starvation-induced muscle protein degradation via regulation of AKT/mTOR/FoxO signaling in C2C12 myotubes

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Ginsenoside Rg1 prevents starvation-induced muscle protein degradation via regulation of AKT/mTOR/FoxO signaling in C2C12 myotubes

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Ginsenoside Rg1 prevents starv ...... xO signaling in C2C12 myotubes

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Ginsenoside Rg1 prevents starv ...... xO signaling in C2C12 myotubes

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Ginsenoside Rg1 prevents starv ...... xO signaling in C2C12 myotubes

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P1476

Ginsenoside Rg1 prevents starv ...... xO signaling in C2C12 myotubes

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Fengyu Li

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Hongliang Huo

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Hongyan Zhao

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Lili Sun

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Ping Wang

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Qingmiao Yu

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Shengnan Jia

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Shuang Ma

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Xiaoxue Li

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Xuewei Peng

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P2860

Muscle-specific RING finger 1 is a bona fide ubiquitin ligase that degrades cardiac troponin I

Myosin accumulation and striated muscle myopathy result from the loss of muscle RING finger 1 and 3

The ubiquitin system

Ginseng pharmacology: multiple constituents and multiple actions

Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression

Identification of ubiquitin ligases required for skeletal muscle atrophy

Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

Transgenic overexpression of locally acting insulin-like growth factor-1 inhibits ubiquitin-mediated muscle atrophy in chronic left-ventricular dysfunction.

The translation regulatory subunit eIF3f controls the kinase-dependent mTOR signaling required for muscle differentiation and hypertrophy in mouse.

Muscle inactivation of mTOR causes metabolic and dystrophin defects leading to severe myopathy.

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1241-1247

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scholarly article

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10.3892/ETM.2017.4615

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P433

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2017-06-15T00:00:00Z

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28781621

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5526193

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