Hydrogen sulfide offers neuroprotection on traumatic brain injury in parallel with reduced apoptosis and autophagy in mice.
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Physiological Importance of Hydrogen Sulfide: Emerging Potent Neuroprotector and NeuromodulatorCathepsin B is a New Drug Target for Traumatic Brain Injury Therapeutics: Evidence for E64d as a Promising Lead Drug CandidateHydrogen Sulfide as a Potential Therapeutic Target in FibrosisModes of physiologic H2S signaling in the brain and peripheral tissuesAmmonium tetrathiomolybdate following ischemia/reperfusion injury: Chemistry, pharmacology, and impact of a new class of sulfide donor in preclinical injury models.The expression of Toll-like receptor 8 and its relationship with VEGF and Bcl-2 in cervical cancer.Effect of apoptosis in neural stem cells treated with sevoflurane.BDNF-TrkB pathway mediates neuroprotection of hydrogen sulfide against formaldehyde-induced toxicity to PC12 cells.Apelin-13 as a novel target for intervention in secondary injury after traumatic brain injury.L-Cysteine metabolism and its nutritional implications.Oxygen tension, H2S, and NO bioavailability: is there an interaction?Neuroprotective effects of hydrogen sulfide on sodium azide‑induced autophagic cell death in PC12 cells.Autophagy in acute brain injury.Functional and Molecular Insights of Hydrogen Sulfide Signaling and Protein Sulfhydration.Upregulation of 3-MST Relates to Neuronal Autophagy After Traumatic Brain Injury in Mice.H2S protects PC12 cells against toxicity of corticosterone by modulation of BDNF-TrkB pathway.Hydrogen sulfide improves neural function in rats following cardiopulmonary resuscitation.Neuroprotective effects of hydrogen sulfide on sodium azide-induced oxidative stress in PC12 cells.Hydrogen sulfide alleviates myocardial fibrosis in mice with alcoholic cardiomyopathy by downregulating autophagy.IL-33 Provides Neuroprotection through Suppressing Apoptotic, Autophagic and NF-κB-Mediated Inflammatory Pathways in a Rat Model of Recurrent Neonatal Seizure.IL-33 Exerts Neuroprotective Effect in Mice Intracerebral Hemorrhage Model Through Suppressing Inflammation/Apoptotic/Autophagic Pathway.Hydrogen sulfide, nitric oxide, and neurodegenerative disorders.Impaired autophagic flux is associated with the severity of trauma and the role of A2AR in brain cells after traumatic brain injury.Exogenous hydrogen sulfide attenuates cerebral ischemia-reperfusion injury by inhibiting autophagy in mice.Autophagy in Traumatic Brain Injury: A New Target for Therapeutic Intervention.IL-33/ST2L Signaling Provides Neuroprotection Through Inhibiting Autophagy, Endoplasmic Reticulum Stress, and Apoptosis in a Mouse Model of Traumatic Brain Injury.The Role of Gaseous Molecules in Traumatic Brain Injury: An Updated Review.
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Hydrogen sulfide offers neuroprotection on traumatic brain injury in parallel with reduced apoptosis and autophagy in mice.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 23 January 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Hydrogen sulfide offers neurop ...... optosis and autophagy in mice.
@en
Hydrogen sulfide offers neurop ...... optosis and autophagy in mice.
@nl
type
label
Hydrogen sulfide offers neurop ...... optosis and autophagy in mice.
@en
Hydrogen sulfide offers neurop ...... optosis and autophagy in mice.
@nl
prefLabel
Hydrogen sulfide offers neurop ...... optosis and autophagy in mice.
@en
Hydrogen sulfide offers neurop ...... optosis and autophagy in mice.
@nl
P2093
P2860
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P1433
P1476
Hydrogen sulfide offers neurop ...... optosis and autophagy in mice.
@en
P2093
Haiyan Shan
Luyang Tao
Mingyang Zhang
Wenwen Dong
Xiping Chen
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P304
P356
10.1371/JOURNAL.PONE.0087241
P407
P577
2014-01-23T00:00:00Z