Cathepsin B contributes to traumatic brain injury-induced cell death through a mitochondria-mediated apoptotic pathway - Wikidata - awgldk - TriplyDB

Cathepsin B contributes to traumatic brain injury-induced cell death through a mitochondria-mediated apoptotic pathway

Cathepsin B contributes to traumatic brain injury-induced cell death through a mitochondria-mediated apoptotic pathway

http://www.wikidata.org/entity/Q28288643

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The cysteine protease cathepsin B is a key drug target and cysteine protease inhibitors are potential therapeutics for traumatic brain injury Progesterone increases circulating endothelial progenitor cells and induces neural regeneration after traumatic brain injury in aged rats Cysteine proteases as therapeutic targets: does selectivity matter? A systematic review of calpain and cathepsin inhibitors Cathepsin B is a New Drug Target for Traumatic Brain Injury Therapeutics: Evidence for E64d as a Promising Lead Drug Candidate Macrophage derived cystatin B/cathepsin B in HIV replication and neuropathogenesis Mitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategies Mitochondrial injury after mechanical stretch of cortical neurons in vitro: biomarkers of apoptosis and selective peroxidation of anionic phospholipids bFGF Protects Against Blood-Brain Barrier Damage Through Junction Protein Regulation via PI3K-Akt-Rac1 Pathway Following Traumatic Brain Injury Cocaine potentiates cathepsin B secretion and neuronal apoptosis from HIV-infected macrophages.Hemorrhagic progression of a contusion after traumatic brain injury: a review.Loss of lysosomal ion channel transient receptor potential channel mucolipin-1 (TRPML1) leads to cathepsin B-dependent apoptosis.Cathepsin B and cystatin B in HIV-seropositive women are associated with infection and HIV-1-associated neurocognitive disorders.Genetic and pharmacological evidence implicates cathepsins in Niemann-Pick C cerebellar degeneration.Long-term expression of zinc transporters in hippocampus following penicillin-induced developmental seizures and its regulation by E-64d.Apelin-13 as a novel target for intervention in secondary injury after traumatic brain injury.Hydrogen sulfide offers neuroprotection on traumatic brain injury in parallel with reduced apoptosis and autophagy in mice.Insights from molecular modeling into the selective inhibition of cathepsin S by its inhibitor.Cystatin C has a dual role in post-traumatic brain injury recovery.Neutrophils in traumatic brain injury (TBI): friend or foe?IL-33/ST2L Signaling Provides Neuroprotection Through Inhibiting Autophagy, Endoplasmic Reticulum Stress, and Apoptosis in a Mouse Model of Traumatic Brain Injury.Moderate hypothermia inhibits microglial activation after traumatic brain injury by modulating autophagy/apoptosis and the MyD88-dependent TLR4 signaling pathway

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Cathepsin B contributes to traumatic brain injury-induced cell death through a mitochondria-mediated apoptotic pathway

http://www.wikidata.org/entity/Q28288643

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Cathepsin B contributes to tra ...... ria-mediated apoptotic pathway

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Cathepsin B contributes to tra ...... ria-mediated apoptotic pathway

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Cathepsin B contributes to tra ...... ria-mediated apoptotic pathway

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type

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Cathepsin B contributes to tra ...... ria-mediated apoptotic pathway

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Cathepsin B contributes to tra ...... ria-mediated apoptotic pathway

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prefLabel

Cathepsin B contributes to tra ...... ria-mediated apoptotic pathway

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P356

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Journal of Neuroscience Research

P1476

Cathepsin B contributes to tra ...... ria-mediated apoptotic pathway

@en

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Cheng-Liang Luo

http://www.w3.org/2001/XMLSchema#string

Hai-Jun Bao

http://www.w3.org/2001/XMLSchema#string

Hong Ni

http://www.w3.org/2001/XMLSchema#string

Lu-Yang Tao

http://www.w3.org/2001/XMLSchema#string

Qian-Qian Li

http://www.w3.org/2001/XMLSchema#string

Qiang-Qiang Cao

http://www.w3.org/2001/XMLSchema#string

Rui Yang

http://www.w3.org/2001/XMLSchema#string

Xi-Ping Chen

http://www.w3.org/2001/XMLSchema#string

Yu-Xia Sun

http://www.w3.org/2001/XMLSchema#string

Zheng-Hong Qin

http://www.w3.org/2001/XMLSchema#string

P2860

An autophagic mechanism is involved in apoptotic death of rat striatal neurons induced by the non-N-methyl-D-aspartate receptor agonist kainic acid

Mitochondria and cell death.

Release of cytochrome c and activation of pro-caspase-9 following lysosomal photodamage involves Bid cleavage.

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2847-58

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scholarly article

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2187300

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10.1002/JNR.22453

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13

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88

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2010-10-01T00:00:00Z

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45284005

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P698

20653046

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apoptotic process

traumatic brain injury