Mammalian RAD52 Functions in Break-Induced Replication Repair of Collapsed DNA Replication Forks.
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Human RAD52 interactions with replication protein A and the RAD51 presynaptic complex.Human RAD52 Captures and Holds DNA Strands, Increases DNA Flexibility, and Prevents Melting of Duplex DNA: Implications for DNA RecombinationThe "enemies within": regions of the genome that are inherently difficult to replicate.Human somatic cells deficient for RAD52 are impaired for viral integration and compromised for most aspects of homology-directed repair.DNA Replication Origins and Fork Progression at Mammalian TelomeresBreak-induced replication links microsatellite expansion to complex genome rearrangements.Control of Mus81 nuclease during the cell cycle.Fragile sites in cancer: more than meets the eye.Inter-Fork Strand Annealing causes genomic deletions during the termination of DNA replication.Lingering single-strand breaks trigger Rad51-independent homology-directed repair of collapsed replication forks in the polynucleotide kinase/phosphatase mutant of fission yeast.Replication fork reversal triggers fork degradation in BRCA2-defective cells.The endonuclease EEPD1 mediates synthetic lethality in RAD52-depleted BRCA1 mutant breast cancer cells.Regulation of mitotic recombination between DNA repeats in centromeres.The end-joining factor Ku acts in the end-resection of double strand break-free arrested replication forks.53BP1 and BRCA1 control pathway choice for stalled replication restart.Small-Molecule Inhibitors Targeting DNA Repair and DNA Repair Deficiency in Research and Cancer Therapy.Alternative Lengthening of Telomeres Mediated by Mitotic DNA Synthesis Engages Break-Induced Replication Processes.Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place.Mutational signatures reveal the role of RAD52 in p53-independent p21-driven genomic instability.Break induced replication in eukaryotes: mechanisms, functions, and consequences.Human cancer cells utilize mitotic DNA synthesis to resist replication stress at telomeres regardless of their telomere maintenance mechanism.Rad52-Rad51 association is essential to protect Rad51 filaments against Srs2, but facultative for filament formationSpatiotemporal dynamics of homologous recombination repair at single collapsed replication forksPreserving replication fork integrity and competence via the homologous recombination pathway
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P2860
Mammalian RAD52 Functions in Break-Induced Replication Repair of Collapsed DNA Replication Forks.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on December 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Mammalian RAD52 Functions in B ...... llapsed DNA Replication Forks.
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Mammalian RAD52 Functions in B ...... llapsed DNA Replication Forks.
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type
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Mammalian RAD52 Functions in B ...... llapsed DNA Replication Forks.
@en
Mammalian RAD52 Functions in B ...... llapsed DNA Replication Forks.
@nl
prefLabel
Mammalian RAD52 Functions in B ...... llapsed DNA Replication Forks.
@en
Mammalian RAD52 Functions in B ...... llapsed DNA Replication Forks.
@nl
P2093
P2860
P50
P1433
P1476
Mammalian RAD52 Functions in B ...... llapsed DNA Replication Forks.
@en
P2093
Caterina Da-Ré
Florian Huber
Irene Kamileri
Konstantinos Evangelou
Laura Padayachy
Leonardo Scapozza
Natalia Lugli
Noemie L Nicati
Samia Barriot
P2860
P304
P356
10.1016/J.MOLCEL.2016.10.038
P577
2016-12-01T00:00:00Z