Alternative Lengthening of Telomeres Mediated by Mitotic DNA Synthesis Engages Break-Induced Replication Processes.
about
Diagnosis and treatment of ALT tumors: is Trabectedin a new therapeutic option?A method for measuring the distribution of the shortest telomeres in cells and tissues.RAD51 and RTEL1 compensate telomere loss in the absence of telomerase.Telomere sequence content can be used to determine ALT activity in tumours.Pathways for maintenance of telomeres and common fragile sites during DNA replication stress.Human cancer cells utilize mitotic DNA synthesis to resist replication stress at telomeres regardless of their telomere maintenance mechanism.Preserving replication fork integrity and competence via the homologous recombination pathwayATRX loss induces multiple hallmarks of the alternative lengthening of telomeres (ALT) phenotype in human glioma cell lines in a cell line-specific manner
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P2860
Alternative Lengthening of Telomeres Mediated by Mitotic DNA Synthesis Engages Break-Induced Replication Processes.
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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name
Alternative Lengthening of Tel ...... Induced Replication Processes.
@en
Alternative Lengthening of Tel ...... Induced Replication Processes.
@nl
type
label
Alternative Lengthening of Tel ...... Induced Replication Processes.
@en
Alternative Lengthening of Tel ...... Induced Replication Processes.
@nl
prefLabel
Alternative Lengthening of Tel ...... Induced Replication Processes.
@en
Alternative Lengthening of Tel ...... Induced Replication Processes.
@nl
P2860
P356
P1476
Alternative Lengthening of Tel ...... Induced Replication Processes.
@en
P2093
Jaewon Min
Jerry W Shay
P2860
P356
10.1128/MCB.00226-17
P407
P577
2017-09-26T00:00:00Z