Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.
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Pathogenesis, immunology, and diagnosis of latent Mycobacterium tuberculosis infectionVaccines for TB: Lessons from the Past Translating into Future PotentialsCurrent efforts and future prospects in the development of live mycobacteria as vaccinesPre-exposure of Mycobacterium tuberculosis-infected macrophages to crystalline silica impairs control of bacterial growth by deregulating the balance between apoptosis and necrosisImproving Mycobacterium bovis bacillus Calmette-Guèrin as a vaccine delivery vector for viral antigens by incorporation of glycolipid activators of NKT cellsTo achieve an earlier IFN-γ response is not sufficient to control Mycobacterium tuberculosis infection in mice.Cell-to-cell transfer of M. tuberculosis antigens optimizes CD4 T cell primingSuboptimal activation of antigen-specific CD4+ effector cells enables persistence of M. tuberculosis in vivo.The three Mycobacterium tuberculosis antigen 85 isoforms have unique substrates and activities determined by non-active site regions.Networked T cell death following macrophage infection by Mycobacterium tuberculosis.Mycobacterial membrane vesicles administered systemically in mice induce a protective immune response to surface compartments of Mycobacterium tuberculosisBeyond macrophages: the diversity of mononuclear cells in tuberculosis.New approaches in the diagnosis and treatment of latent tuberculosis infectionThe ESAT-6 protein of Mycobacterium tuberculosis interacts with beta-2-microglobulin (β2M) affecting antigen presentation function of macrophage.Role of PPE18 protein in intracellular survival and pathogenicity of Mycobacterium tuberculosis in miceMemory T cells in latent Mycobacterium tuberculosis infection are directed against three antigenic islands and largely contained in a CXCR3+CCR6+ Th1 subsetExogenous control of the expression of Group I CD1 molecules competent for presentation of microbial nonpeptide antigens to human T lymphocytesImmunological consequences of intragenus conservation of Mycobacterium tuberculosis T-cell epitopesMicroRNA-155 promotes autophagy to eliminate intracellular mycobacteria by targeting RhebRecent Advances in Defining the Immunoproteome of Mycobacterium tuberculosisESAT-6 Targeting to DEC205+ Antigen Presenting Cells Induces Specific-T Cell Responses against ESAT-6 and Reduces Pulmonary Infection with Virulent Mycobacterium tuberculosis.Evasion of innate immunity by Mycobacterium tuberculosis: is death an exit strategy?Human and Murine Clonal CD8+ T Cell Expansions Arise during Tuberculosis Because of TCR SelectionA recombinant Mycobacterium smegmatis induces potent bactericidal immunity against Mycobacterium tuberculosis.A Quantitative Analysis of Complexity of Human Pathogen-Specific CD4 T Cell Responses in Healthy M. tuberculosis Infected South Africans.Imaging flow cytometry analysis of intracellular pathogens.Epigenetics and Proteomics Join Transcriptomics in the Quest for Tuberculosis Biomarkers.A recombinant attenuated Mycobacterium tuberculosis vaccine strain is safe in immunosuppressed simian immunodeficiency virus-infected infant macaquesEnhanced control of Mycobacterium tuberculosis extrapulmonary dissemination in mice by an arabinomannan-protein conjugate vaccine.MicroRNA-144-3p inhibits autophagy activation and enhances Bacillus Calmette-Guérin infection by targeting ATG4a in RAW264.7 macrophage cellsDifferential trafficking of TLR1 I602S underlies host protection against pathogenic mycobacteria.Antigen Export Reduces Antigen Presentation and Limits T Cell Control of M. tuberculosis.Enhanced Direct Major Histocompatibility Complex Class I Self-Antigen Presentation Induced by Chlamydia InfectionM. tuberculosis T Cell Epitope Analysis Reveals Paucity of Antigenic Variation and Identifies Rare Variable TB Antigens.PPE26 induces TLR2-dependent activation of macrophages and drives Th1-type T-cell immunity by triggering the cross-talk of multiple pathways involved in the host response.ESAT-6-dependent cytosolic pattern recognition drives noncognate tuberculosis control in vivoMycobacterium tuberculosis Cell Wall Fragments Released upon Bacterial Contact with the Human Lung Mucosa Alter the Neutrophil Response to Infection.Disruption of immune regulation by microbial pathogens and resulting chronic inflammation.Immune Responses to Bacillus Calmette-Guérin Vaccination: Why Do They Fail to Protect against Mycobacterium tuberculosis?Serving the new masters - dendritic cells as hosts for stealth intracellular bacteria.
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Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 25 June 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.
@en
Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.
@nl
type
label
Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.
@en
Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.
@nl
prefLabel
Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.
@en
Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.
@nl
P2860
P1433
P1476
Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.
@en
P2093
P2860
P304
P356
10.1111/J.1399-0039.2009.01301.X
P577
2009-06-25T00:00:00Z