Premature senescence of endothelial cells upon chronic exposure to TNFα can be prevented by N-acetyl cysteine and plumericin.
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Ionizing Radiation-Induced Immune and Inflammatory Reactions in the Brain.Role of Kallistatin Treatment in Aging and Cancer by Modulating miR-34a and miR-21 Expression.Association between corneal endothelial cell densities and elevated cytokine levels in the aqueous humor.Activation of Nrf2-mediated anti-oxidant genes by antrodin C prevents hyperglycemia-induced senescence and apoptosis in human endothelial cells.miR‑342‑5p promotes Zmpste24‑deficient mouse embryonic fibroblasts proliferation by suppressing GAS2.Ganglioside GM1 contributes to extracellular/intracellular regulation of insulin resistance, impairment of insulin signaling and down-stream eNOS activation, in human aortic endothelial cells after short- or long-term exposure to TNFα.
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Premature senescence of endothelial cells upon chronic exposure to TNFα can be prevented by N-acetyl cysteine and plumericin.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 03 January 2017
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Premature senescence of endoth ...... cetyl cysteine and plumericin.
@en
Premature senescence of endoth ...... cetyl cysteine and plumericin.
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type
label
Premature senescence of endoth ...... cetyl cysteine and plumericin.
@en
Premature senescence of endoth ...... cetyl cysteine and plumericin.
@nl
prefLabel
Premature senescence of endoth ...... cetyl cysteine and plumericin.
@en
Premature senescence of endoth ...... cetyl cysteine and plumericin.
@nl
P2093
P2860
P50
P356
P1433
P1476
Premature senescence of endoth ...... cetyl cysteine and plumericin.
@en
P2093
Andre Oszwald
Ezzat M Awad
Markus Lipovac
Shafaat Y Khan
P2860
P2888
P356
10.1038/SREP39501
P407
P577
2017-01-03T00:00:00Z