One pyrimidine dimer inactivates expression of a transfected gene in xeroderma pigmentosum cells.
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Human ERCC5 cDNA-cosmid complementation for excision repair and bipartite amino acid domains conserved with RAD proteins of Saccharomyces cerevisiae and Schizosaccharomyces pombeChromosomal protein HMGN1 enhances the rate of DNA repair in chromatinUltraviolet light selection assay to optimize oligonucleotide correction of mutations in endogenous xeroderma pigmentosum genes.Joining of linear plasmid DNA is reduced and error-prone in Bloom's syndrome cells.Decreased transcription-coupled nucleotide excision repair capacity is associated with increased p53- and MLH1-independent apoptosis in response to cisplatin.Photoproduct frequency is not the major determinant of UV base substitution hot spots or cold spots in human cellsThe tumor suppressor p53 can both stimulate and inhibit ultraviolet light-induced apoptosisSequence specificity of cyclobutane pyrimidine dimers in DNA treated with solar (ultraviolet B) radiation.The xeroderma pigmentosum group C gene leads to selective repair of cyclobutane pyrimidine dimers rather than 6-4 photoproducts.DNA repair phenotype and cancer susceptibility--a mini review.Forty years of research on xeroderma pigmentosum at the US National Institutes of Health.Ultraviolet light-induced chromosomal aberrations in cultured cells from Cockayne syndrome and complementation group C xeroderma pigmentosum patients: lack of correlation with cancer susceptibility.Abnormal ultraviolet mutagenic spectrum in plasmid DNA replicated in cultured fibroblasts from a patient with the skin cancer-prone disease, xeroderma pigmentosumShining a light on xeroderma pigmentosum.Acquired cisplatin resistance in human ovarian cancer cells is associated with enhanced repair of cisplatin-DNA lesions and reduced drug accumulation.Rapid repair kinetics of pyrimidine(6-4)pyrimidone photoproducts in human cells are due to excision rather than conformational change.DNA repair in a small yeast plasmid folded into chromatin.The residual repair capacity of xeroderma pigmentosum complementation group C fibroblasts is highly specific for transcriptionally active DNAIdentification and Functional Testing of ERCC2 Mutations in a Multi-national Cohort of Patients with Familial Breast- and Ovarian Cancer.DNA repair and aging in basal cell carcinoma: a molecular epidemiology study.The nucleosome-binding protein HMGN2 modulates global genome repairPhotorepair mutants of Arabidopsis.Cross-resistance to UV radiation of a cisplatin-resistant human cell line: overexpression of cellular factors that recognize UV-modified DNAA constitutive damage-specific DNA-binding protein is synthesized at higher levels in UV-irradiated primate cellsEpstein-Barr virus latent membrane protein 1 represses DNA repair through the PI3K/Akt/FOXO3a pathway in human epithelial cells.UV light-induced cyclobutane pyrimidine dimers are mutagenic in mammalian cells.In vitro expression levels of cell-cycle checkpoint proteins are associated with cellular DNA repair capacity in peripheral blood lymphocytes: a multivariate analysisRestricted ultraviolet mutational spectrum in a shuttle vector propagated in xeroderma pigmentosum cells.Differential expression of pyrimidine dimer-binding proteins in normal and UV light-treated vertebrate cells.The biology of the (6-4) photoproduct.Analysis of actively transcribed DNA repair using a transfection-based systemHow are base excision DNA repair pathways deployed in vivo?Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress.Cyclosporin A, but not everolimus, inhibits DNA repair mediated by calcineurin: implications for tumorigenesis under immunosuppression.The use of recombinant DNA techniques to study radiation-induced damage, repair and genetic change in mammalian cells.Genome-wide variation of the somatic mutation frequency in transgenic plants.Role of the AtRad1p endonuclease in homologous recombination in plants.Epstein-Barr virus latent membrane protein 1 induces micronucleus formation, represses DNA repair and enhances sensitivity to DNA-damaging agents in human epithelial cells.Analysis of DNA repair using transfection-based host cell reactivation.Variations in transcription-repair coupling in mouse cells.
P2860
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P2860
One pyrimidine dimer inactivates expression of a transfected gene in xeroderma pigmentosum cells.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 1985
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
One pyrimidine dimer inactivat ...... n xeroderma pigmentosum cells.
@en
One pyrimidine dimer inactivat ...... n xeroderma pigmentosum cells.
@nl
type
label
One pyrimidine dimer inactivat ...... n xeroderma pigmentosum cells.
@en
One pyrimidine dimer inactivat ...... n xeroderma pigmentosum cells.
@nl
prefLabel
One pyrimidine dimer inactivat ...... n xeroderma pigmentosum cells.
@en
One pyrimidine dimer inactivat ...... n xeroderma pigmentosum cells.
@nl
P2860
P356
P1476
One pyrimidine dimer inactivat ...... in xeroderma pigmentosum cells
@en
P2093
M Protić-Sabljić
P2860
P304
P356
10.1073/PNAS.82.19.6622
P407
P577
1985-10-01T00:00:00Z