Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias. - Wikidata - awgldk - TriplyDB

Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

http://www.wikidata.org/entity/Q37550949

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X chromosome inactivation in human pluripotent stem cells as a model for human development: back to the drawing board?Medulloblastoma: Molecular Classification-Based Personal Therapeutics.Epigenome Aberrations: Emerging Driving Factors of the Clear Cell Renal Cell Carcinoma.Avian W and mammalian Y chromosomes convergently retained dosage-sensitive regulators.When the Lyon(ized chromosome) roars: ongoing expression from an inactive X chromosome.An integrative approach to assess X-chromosome inactivation using allele-specific expression with applications to epithelial ovarian cancer.Regulation of X-chromosome dosage compensation in human: mechanisms and model systems.Kdm6a overexpression improves the development of cloned mouse embryos.Targeting RNA helicases in cancer: The translation trap.JARID1 Histone Demethylases: Emerging Targets in Cancer.Sex disparity in cancer: roles of microRNAs and related functional players.Genetics: Sex and the single cell.Cooperative p16 and p21 action protects female astrocytes from transformation.The deubiquitinase USP9X regulates FBW7 stability and suppresses colorectal cancer.Paternal lineage early onset hereditary ovarian cancers: A Familial Ovarian Cancer Registry study.Targeting JARID1B's demethylase activity blocks a subset of its functions in oral cancer.Sex Differences in HIV Infection.X chromosome protects against bladder cancer in females via a KDM6A-dependent epigenetic mechanism.New generation sequencing of targeted genes in the classical and the variant form of hairy cell leukemia highlights mutations in epigenetic regulation genes.Sex Differences in HIV Infection: Mystique Versus Machismo

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Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

http://www.wikidata.org/entity/Q37550949

description

2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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2016年學術文章

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name

Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

@en

Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

@nl

type

label

Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

@en

Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

@nl

altLabel

Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias

@en

prefLabel

Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

@en

Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

@nl

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Nature Genetics

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Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.

@en

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Akinori Yoda

http://www.w3.org/2001/XMLSchema#string

Alexander A Gimelbrant

http://www.w3.org/2001/XMLSchema#string

Andrew A Lane

http://www.w3.org/2001/XMLSchema#string

Andrew Dunford

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David M Weinstock

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John P Cleary

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Julian M Hess

http://www.w3.org/2001/XMLSchema#string

Michael S Lawrence

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Rameen Beroukhim

http://www.w3.org/2001/XMLSchema#string

Steven E Schumacher

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P2860

X-inactivation profile reveals extensive variability in X-linked gene expression in females

Mutations in the JARID1C gene, which is involved in transcriptional regulation and chromatin remodeling, cause X-linked mental retardation.

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles.

Biological insights from 108 schizophrenia-associated genetic loci

Functional impact of global rare copy number variation in autism spectrum disorders

Comprehensive, Integrative Genomic Analysis of Diffuse Lower-Grade Gliomas.

Cancer statistics, 2013

Antioncogenes and human cancer.

Discovery and saturation analysis of cancer genes across 21 tumour types

UTX and UTY demonstrate histone demethylase-independent function in mouse embryonic development

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10-16

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10.1038/NG.3726

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