Stress and IL-1beta contribute to the development of depressive-like behavior following peripheral nerve injury.
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Comorbidity Factors and Brain Mechanisms Linking Chronic Stress and Systemic IllnessHigh Times for Painful Blues: The Endocannabinoid System in Pain-Depression ComorbidityCognitive and emotional control of pain and its disruption in chronic painAlcoholism and inflammation: neuroimmunology of behavioral and mood disordersActivation of P2X7 promotes cerebral edema and neurological injury after traumatic brain injury in miceNeuronal nitric oxide synthase and NADPH oxidase interact to affect cognitive, affective, and social behaviors in mice.Alterations in the inflammatory cytokines and brain-derived neurotrophic factor contribute to depression-like phenotype after spared nerve injury: improvement by ketamine.Spared Nerve Injury Increases the Expression of Microglia M1 Markers in the Prefrontal Cortex of Rats and Provokes Depression-Like Behaviors.Chronic Social Stress Time-Dependently Affects Neuropathic Pain-Related Cold Allodynia and Leads to Altered Expression of Spinal Biochemical Mediators.Inflammation: The Common Pathway of Stress-Related Diseases.Astrocyte Activation in Locus Coeruleus Is Involved in Neuropathic Pain Exacerbation Mediated by Maternal Separation and Social Isolation Stress.Effects of tianeptine on symptoms of fibromyalgia via BDNF signaling in a fibromyalgia animal model.Neuropathic pain promotes adaptive changes in gene expression in brain networks involved in stress and depressionProlonged depression-like behavior caused by immune challenge: influence of mouse strain and social environment.Neuropathic pain-induced depressive-like behavior and hippocampal neurogenesis and plasticity are dependent on TNFR1 signalingPain and depression comorbidity: a preclinical perspective.Toward an anti-inflammatory strategy for depressionModulation of cortisol responses to the DEX/CRH test by polymorphisms of the interleukin-1beta gene in healthy adults.Effect of environment on the long-term consequences of chronic painIntracerebroventricular administration of HIV-1 Tat induces brain cytokine and indoleamine 2,3-dioxygenase expression: a possible mechanism for AIDS comorbid depression.Chronic neuropathic pain-like behavior correlates with IL-1β expression and disrupts cytokine interactions in the hippocampus.Peripheral indoleamine 2,3-dioxygenase 1 is required for comorbid depression-like behavior but does not contribute to neuropathic pain in mice.Neurotrophins role in depression neurobiology: a review of basic and clinical evidence.Microglia Transcriptome Changes in a Model of Depressive Behavior after Immune Challenge.Peripheral and central effects of repeated social defeat stress: monocyte trafficking, microglial activation, and anxiety.Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depressionExacerbated mechanical hyperalgesia in rats with genetically predisposed depressive behavior: role of melatonin and NMDA receptors.Glucocorticoids, cytokines and brain abnormalities in depression.Effects of hydrogen-rich water on depressive-like behavior in mice.Interleukin-1 beta converting enzyme is necessary for development of depression-like behavior following intracerebroventricular administration of lipopolysaccharide to mice.Alleviation of chronic neuropathic pain by environmental enrichment in mice well after the establishment of chronic painInterleukin-1β overproduction is a common cause for neuropathic pain, memory deficit, and depression following peripheral nerve injury in rodentsUncoupling protein 2 modulation of the NLRP3 inflammasome in astrocytes and its implications in depression"Bedside-to-Bench" Behavioral Outcomes in Animal Models of Pain: Beyond the Evaluation of Reflexes.Neuroinflammation and comorbidity of pain and depression.Chronic neuropathic pain-like behavior and brain-borne IL-1β.Targeting IL-1 in depression.An altered spinal serotonergic system contributes to increased thermal nociception in an animal model of depression.Altered neuropathic pain behaviour in a rat model of depression is associated with changes in inflammatory gene expression in the amygdala.Integrative neurobiology of metabolic diseases, neuroinflammation, and neurodegeneration.
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P2860
Stress and IL-1beta contribute to the development of depressive-like behavior following peripheral nerve injury.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 22 September 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Stress and IL-1beta contribute ...... owing peripheral nerve injury.
@en
Stress and IL-1beta contribute ...... owing peripheral nerve injury.
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type
label
Stress and IL-1beta contribute ...... owing peripheral nerve injury.
@en
Stress and IL-1beta contribute ...... owing peripheral nerve injury.
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prefLabel
Stress and IL-1beta contribute ...... owing peripheral nerve injury.
@en
Stress and IL-1beta contribute ...... owing peripheral nerve injury.
@nl
P2093
P2860
P356
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P1476
Stress and IL-1beta contribute ...... owing peripheral nerve injury.
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P2093
P2860
P2888
P304
P356
10.1038/MP.2009.91
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P577
2009-09-22T00:00:00Z