Mitogen-activated Protein Kinase Kinase Activity Maintains Acinar-to-Ductal Metaplasia and Is Required for Organ Regeneration in Pancreatitis.
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Epithelial-Myeloid cell crosstalk regulates acinar cell plasticity and pancreatic remodeling in mice.Metaplasia: tissue injury adaptation and a precursor to the dysplasia-cancer sequence.Krüppel-like Factor 5, Increased in Pancreatic Ductal Adenocarcinoma, Promotes Proliferation, Acinar to Ductal Metaplasia, Pancreatic Intraepithelial Neoplasia, and Tumor Growth in Mice.TUMOR CROSSTALK NETWORKS PROMOTE GROWTH AND SUPPORT IMMUNE EVASION IN PANCREATIC CANCER.Pancreatic HIF2α Stabilization Leads to Chronic Pancreatitis and Predisposes to Mucinous Cystic Neoplasm.
P2860
Mitogen-activated Protein Kinase Kinase Activity Maintains Acinar-to-Ductal Metaplasia and Is Required for Organ Regeneration in Pancreatitis.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on January 2017
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Mitogen-activated Protein Kina ...... Regeneration in Pancreatitis.
@en
Mitogen-activated Protein Kina ...... Regeneration in Pancreatitis.
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type
label
Mitogen-activated Protein Kina ...... Regeneration in Pancreatitis.
@en
Mitogen-activated Protein Kina ...... Regeneration in Pancreatitis.
@nl
prefLabel
Mitogen-activated Protein Kina ...... Regeneration in Pancreatitis.
@en
Mitogen-activated Protein Kina ...... Regeneration in Pancreatitis.
@nl
P2093
P2860
P1476
Mitogen-activated Protein Kina ...... Regeneration in Pancreatitis.
@en
P2093
Christopher J Halbrook
Howard C Crawford
Hui-Ju Wen
Jeanine M Ruggeri
Kenneth K Takeuchi
Marina Pasca di Magliano
Yaqing Zhang
P2860
P304
P356
10.1016/J.JCMGH.2016.09.009
P577
2017-01-01T00:00:00Z