Chronic carbamazepine administration attenuates dopamine D2-like receptor-initiated signaling via arachidonic acid in rat brain. - Wikidata - awgldk - TriplyDB

Chronic carbamazepine administration attenuates dopamine D2-like receptor-initiated signaling via arachidonic acid in rat brain.

Chronic carbamazepine administration attenuates dopamine D2-like receptor-initiated signaling via arachidonic acid in rat brain.

http://www.wikidata.org/entity/Q37590122

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Bipolar disorder and mechanisms of action of mood stabilizers Lithium and the other mood stabilizers effective in bipolar disorder target the rat brain arachidonic acid cascade Second messenger/signal transduction pathways in major mood disorders: moving from membrane to mechanism of action, part II: bipolar disorder Characterization of ST14A Cells for Studying Modulation of Voltage-Gated Calcium Channels.Chronic NMDA administration to rats increases brain pro-apoptotic factors while decreasing anti-Apoptotic factors and causes cell death.Extracellular-derived calcium does not initiate in vivo neurotransmission involving docosahexaenoic acid.Chronic valproate treatment blocks D2-like receptor-mediated brain signaling via arachidonic acid in rats.Lamotrigine blocks NMDA receptor-initiated arachidonic acid signalling in rat brain: implications for its efficacy in bipolar disorder Imaging brain signal transduction and metabolism via arachidonic and docosahexaenoic acid in animals and humans.Gabapentin's minimal action on markers of rat brain arachidonic acid metabolism agrees with its inefficacy against bipolar disorder.Effects of chronic clozapine administration on markers of arachidonic acid cascade and synaptic integrity in rat brain Chronic administration of valproic acid reduces brain NMDA signaling via arachidonic acid in unanesthetized rats.Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.The emerging role of dopamine-glutamate interaction and of the postsynaptic density in bipolar disorder pathophysiology: Implications for treatment.Oculogyric crises: A review of phenomenology, etiology, pathogenesis, and treatment.

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Chronic carbamazepine administration attenuates dopamine D2-like receptor-initiated signaling via arachidonic acid in rat brain.

http://www.wikidata.org/entity/Q37590122

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 27 February 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Chronic carbamazepine administ ...... arachidonic acid in rat brain.

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Chronic carbamazepine administ ...... arachidonic acid in rat brain.

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Chronic carbamazepine administ ...... arachidonic acid in rat brain.

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Chronic carbamazepine administ ...... arachidonic acid in rat brain.

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Chronic carbamazepine administ ...... arachidonic acid in rat brain.

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Chronic carbamazepine administ ...... arachidonic acid in rat brain.

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S11064-008-9595-Y

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Neurochemical Research

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Chronic carbamazepine administ ...... arachidonic acid in rat brain.

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Jane M Bell

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Lisa Chang

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Mei Chen

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Mireille Basselin

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P2860

Dopamine dysregulation syndrome: implications for a dopamine hypothesis of bipolar disorder

Haloperidol alone or in combination for acute mania

A simple method for the isolation and purification of total lipides from animal tissues

Molecular cloning and characterization of a novel dopamine receptor (D3) as a target for neuroleptics

Immunohistochemical and immunoblot analysis of Dopamine and cyclic AMP-regulated phosphoprotein, relative molecular mass 32,000 (DARPP-32) in the prefrontal cortex of subjects with schizophrenia and bipolar disorder

Chronic administration of carbamazepine down-regulates AP-2 DNA-binding activity and AP-2alpha protein expression in rat frontal cortex

Decrease in the AP-2 DNA-binding activity and in the protein expression of AP-2 alpha and AP-2 beta in frontal cortex of rats treated with lithium for 6 weeks

Cortical localization of dopamine D4 receptors in the rat brain--immunocytochemical study

Chronic treatment with mood stabilizers increases membrane GRK3 in rat frontal cortex

85 kDa cytosolic phospholipase A2 is a target for chronic lithium in rat brain

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10.1007/S11064-008-9595-Y

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Stanley I. Rapoport

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