Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury. - Wikidata - awgldk - TriplyDB

Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

http://www.wikidata.org/entity/Q37591248

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Cell cycle-dependent chromatin shuttling of HBO1-JADE1 histone acetyl transferase (HAT) complex.Stimulating healthy tissue regeneration by targeting the 5-HT₂B receptor in chronic liver disease.Biomarkers of acute kidney injury and associations with short- and long-term outcomes The cell cycle biomarkers: promising research, but do not oversell them Wnt/β-catenin signaling in kidney injury and repair: a double-edged sword EGFR signaling in renal fibrosis The FOXD1 lineage of kidney perivascular cells and myofibroblasts: functions and responses to injury Primary proximal tubule injury leads to epithelial cell cycle arrest, fibrosis, vascular rarefaction, and glomerulosclerosis Alteration of Fatty Acid Oxidation in Tubular Epithelial Cells: From Acute Kidney Injury to Renal Fibrogenesis The aging kidney: increased susceptibility to nephrotoxicity Evidence for the involvement of epigenetics in the progression of renal fibrogenesis Recent advances in understanding of chronic kidney disease Inhibitors/antagonists of TGF-β system in kidney fibrosis Host responses in tissue repair and fibrosis Renal endothelial dysfunction in acute kidney ischemia reperfusion injury Recent developments in myofibroblast biology: paradigms for connective tissue remodeling How the Innate Immune System Senses Trouble and Causes Trouble TGF-β signaling in tissue fibrosis: redox controls, target genes and therapeutic opportunities Dendritic cells and macrophages in the kidney: a spectrum of good and evil Negative Regulation of TGFβ Signaling by Stem Cell Antigen-1 Protects against Ischemic Acute Kidney Injury Group IVA Cytosolic Phospholipase A2 Regulates the G2-to-M Transition by Modulating the Activity of Tumor Suppressor SIRT2.Pulmonary fibrosis: patterns and perpetrators Diagnostic Criteria for Acute Kidney Injury: Present and Future The roles of CD147 and/or cyclophilin A in kidney diseases Cell-cycle arrest and acute kidney injury: the light and the dark sides A proteomics approach to identifying key protein targets involved in VEGF inhibitor mediated attenuation of bleomycin-induced pulmonary fibrosis Severe renal mass reduction impairs recovery and promotes fibrosis after AKI Hypertension-related, calcium-regulated gene (HCaRG/COMMD5) and kidney diseases: HCaRG accelerates tubular repair Endothelial Sphingosine 1‑Phosphate Receptor‑1 Mediates Protection and Recovery from Acute Kidney Injury.Sphingosine Kinase 2 Deficiency Attenuates Kidney Fibrosis via IFN-γ.Cellular and Molecular Mechanisms of AKI.Progression after AKI: Understanding Maladaptive Repair Processes to Predict and Identify Therapeutic Treatments.Multiple Mechanisms are Involved in Salt-Sensitive Hypertension-Induced Renal Injury and Interstitial Fibrosis Cellular mechanisms of tissue fibrosis. 1. Common and organ-specific mechanisms associated with tissue fibrosis.Retinoic Acid Signaling Coordinates Macrophage-Dependent Injury and Repair after AKI.High-resolution renal perfusion mapping using contrast-enhanced ultrasonography in ischemia-reperfusion injury monitors changes in renal microperfusion.Renin-Angiotensin System Inhibitor is Associated with Lower Risk of Ensuing Chronic Kidney Disease after Functional Recovery from Acute Kidney Injury.Silencing SMOC2 ameliorates kidney fibrosis by inhibiting fibroblast to myofibroblast transformation Melatonin promoted renal regeneration in folic acid-induced acute kidney injury via inhibiting nucleocytoplasmic translocation of HMGB1 in tubular epithelial cells.Response gene to complement 32 regulates the G2/M phase checkpoint during renal tubular epithelial cell repair.

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Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

http://www.wikidata.org/entity/Q37591248

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 02 May 2010

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

@en

Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

@nl

type

label

Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

@en

Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

@nl

prefLabel

Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

@en

Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

@nl

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Nature Medicine

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Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury.

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Craig R Brooks

http://www.w3.org/2001/XMLSchema#string

Jagesh V Shah

http://www.w3.org/2001/XMLSchema#string

Joseph V Bonventre

http://www.w3.org/2001/XMLSchema#string

Li Yang

http://www.w3.org/2001/XMLSchema#string

Tatiana Y Besschetnova

http://www.w3.org/2001/XMLSchema#string

P2860

Epithelial-mesenchymal transition and its implications for fibrosis

Mitotic phosphorylation of histone H3: spatio-temporal regulation by mammalian Aurora kinases

Cellular and molecular mechanisms of fibrosis

Control of the G2/M transition

Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells

Requirement for p53 and p21 to sustain G2 arrest after DNA damage

A p53-mediated DNA damage response limits reprogramming to ensure iPS cell genomic integrity

Identification and characterization of a novel and specific inhibitor of the ataxia-telangiectasia mutated kinase ATM

Linking the p53 tumour suppressor pathway to somatic cell reprogramming

Cell cycle checkpoint signaling through the ATM and ATR kinases

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535-43, 1p following 143

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scholarly article

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10.1038/NM.2144

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5

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16

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2010-05-02T00:00:00Z

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43535442

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20436483

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3928013

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