Transcription could be the key to the selection advantage of mitochondrial deletion mutants in aging.
about
MPV17L2 is required for ribosome assembly in mitochondriaQuality matters: how does mitochondrial network dynamics and quality control impact on mtDNA integrity?Mitochondrial function in hypoxic ischemic injury and influence of agingEvolutionary dynamics of the mitochondrial genome in the evaniomorpha (hymenoptera)—a group with an intermediate rate of gene rearrangementMitochondrial DNA Sequence and Lack of Response to Anoxia in the Annual Killifish Austrofundulus limnaeusThe road to rack and ruin: selecting deleterious mitochondrial DNA variantsContext-Dependent Role of Mitochondrial Fusion-Fission in Clonal Expansion of mtDNA MutationsUnderstanding the physiology of the ageing individual: computational modelling of changes in metabolism and endurance.Latent mitochondrial DNA deletion mutations drive muscle fiber loss at old age.Homeostatic Responses Regulate Selfish Mitochondrial Genome Dynamics in C. elegans.Mitochondria, muscle health, and exercise with advancing age.Modelling the molecular mechanisms of aging.Digital PCR Quantitation of Muscle Mitochondrial DNA: Age, Fiber Type, and Mutation-Induced Changes.Postnatal Hyperoxia Exposure Durably Impairs Right Ventricular Function and Mitochondrial Biogenesis.Intracellular evolution of mitochondrial DNA (mtDNA) and the tragedy of the cytoplasmic commons.Resolving the Enigma of the Clonal Expansion of mtDNA Deletions.Subcellular origin of mitochondrial DNA deletions in human skeletal muscle
P2860
Q24299607-77C9A66E-E36B-4DF3-9E50-863618B88368Q26862395-68AE448F-5112-4F57-8FBD-0E6B9AAC3B54Q28396663-43BEB2CE-D947-4ECE-A4DB-AAFC5B2A624BQ28654892-1F3A3256-9263-4DB6-9C31-0CA8B8741BF1Q28828431-6591C4BF-D214-400B-AE64-F15BCFB81B5CQ33655994-973F0EDA-1D57-4180-9F6B-92C59C37B312Q35634191-1BCB4F66-92C3-4DD4-B7B4-C00618A40F49Q36596036-09DA126A-3D31-49B7-BBAB-36BAF5321579Q37422383-06076763-537D-445E-8030-DCA066654DC4Q37619344-A4E62CBE-C0DE-47CF-9583-65CA8AF896BEQ38453882-1ECBAF0B-0ED8-4CE9-9BCE-892C12B78576Q38769316-CA3B486E-D738-4AAC-802D-5C9360E0A091Q38811014-545DE347-550F-4D26-8034-04BD9929D57BQ38995063-95ECF14C-8F3E-48FF-B022-75B3A74084E0Q50910700-13AF17E8-5615-4D4A-B655-46E9FD229F38Q51766326-977EEFF7-C7CF-4D4C-86F9-FB7D091C83D8Q57416781-2FF4FB16-8288-4EAC-B612-0C4428B76381
P2860
Transcription could be the key to the selection advantage of mitochondrial deletion mutants in aging.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 03 February 2014
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Transcription could be the key ...... ial deletion mutants in aging.
@en
Transcription could be the key ...... ial deletion mutants in aging.
@nl
type
label
Transcription could be the key ...... ial deletion mutants in aging.
@en
Transcription could be the key ...... ial deletion mutants in aging.
@nl
prefLabel
Transcription could be the key ...... ial deletion mutants in aging.
@en
Transcription could be the key ...... ial deletion mutants in aging.
@nl
P2860
P356
P1476
Transcription could be the key ...... ial deletion mutants in aging.
@en
P2093
Axel Kowald
P2860
P304
P356
10.1073/PNAS.1314970111
P407
P50
P577
2014-02-03T00:00:00Z