Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy? - Wikidata - awgldk - TriplyDB

Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?

Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?

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Neuroprotective Effects of Protein Tyrosine Phosphatase 1B Inhibition against ER Stress-Induced Toxicity.Quantitative and Systems Pharmacology 3. Network-Based Identification of New Targets for Natural Products Enables Potential Uses in Aging-Associated Disorders.

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Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?

http://www.wikidata.org/entity/Q37614594

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 31 January 2017

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?

@en

type

label

Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?

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prefLabel

Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?

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FNAGI.2017.00007

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Frontiers in Aging Neuroscience

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Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?

@en

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Fernanda G De Felice

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Natalia M Lyra E Silva

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Sergio T Ferreira

http://www.w3.org/2001/XMLSchema#string

P2860

Tyrosine dephosphorylation and deactivation of insulin receptor substrate-1 by protein-tyrosine phosphatase 1B. Possible facilitation by the formation of a ternary complex with the Grb2 adaptor protein

A Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Disease

Positional cloning of the mouse obese gene and its human homologue

PTP1B regulates leptin signal transduction in vivo

Protein tyrosine phosphatases--from housekeeping enzymes to master regulators of signal transduction

Endoplasmic reticulum stress and the inflammatory basis of metabolic disease

Leptin regulates tau phosphorylation and amyloid through AMPK in neuronal cells

The amyloid hypothesis of Alzheimer's disease at 25 years

Role of pro-inflammatory cytokines released from microglia in Alzheimer's disease

Advances in Alzheimer's disease drug development

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7

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scholarly article

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10.3389/FNAGI.2017.00007

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9

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Marcelo N N Vieira

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2017-01-31T00:00:00Z

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28197094

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Alzheimer's disease

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5281585

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