Subfragments of the large terminal repeat cause glucocorticoid-responsive expression of mouse mammary tumor virus and of an adjacent gene.
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Androgen regulated expression of a spermine binding protein gene in mouse ventral prostateA secretory protease inhibitor requires androgens for its expression in male sex accessory tissues but is expressed constitutively in pancreascDNA clones for the ecdysone-inducible polypeptide (EIP) mRNAs of Drosophila Kc cellsThe position and length of the steroid-dependent hypersensitive region in the mouse mammary tumor virus long terminal repeat are invariant despite multiple nucleosome B frames.Prolactin and glucocorticoid hormones synergistically induce expression of transfected rat beta-casein gene promoter constructs in a mammary epithelial cell line.Binding of hormone accelerates the kinetics of glucocorticoid and progesterone receptor binding to DNA.Steroid hormone dependence of four DNase I-hypersensitive regions located within the 7000-bp 5'-flanking segment of the ovalbumin geneThe v-mos and H-ras oncogene expression represses glucocorticoid hormone-dependent transcription from the mouse mammary tumor virus LTR.Sequence-specific DNA binding of the progesterone receptor to the uteroglobin gene: effects of hormone, antihormone and receptor phosphorylationNuclear factor I acts as a transcription factor on the MMTV promoter but competes with steroid hormone receptors for DNA binding.Programmed killing of human cells by means of an inducible clone of parvoviral genes encoding non-structural proteins.The hormone response element of the mouse mammary tumour virus DNA mediates the progestin and androgen induction of transcription in the proviral long terminal repeat region.Glucocorticoid induction of the rat tryptophan oxygenase gene is mediated by two widely separated glucocorticoid-responsive elements.The upstream regulatory region of the human papilloma virus-16 contains an E2 protein-independent enhancer which is specific for cervical carcinoma cells and regulated by glucocorticoid hormones.Modulation of enhancer activity by the hormone responsive regulatory element from mouse mammary tumor virus.Cross-talk between glucocorticoid receptor and AP-1.Application of a protein-blotting procedure to the study of human glucocorticoid receptor interactions with DNA.Transcriptional repression of a hormone-responsive promoterFunctional redundancy of octamer elements in the mouse mammary tumor virus promoter.Dual function of a nuclear factor I binding site in MMTV transcription regulationFactors controlling the expression of mouse mammary tumour virus.Expression of the mouse mammary tumor virus ORF gene in cultured cells.Sequence homologies in the region preceding the transcription initiation site of the liver estrogen-responsive vitellogenin and apo-VLDLII genes.A comparison between mammalian and avian fast skeletal muscle alkali myosin light chain genes: regulatory implicationsHuman HSP70 promoter contains at least two distinct regulatory domains.A complete library of point substitution mutations in the glucocorticoid response element of mouse mammary tumor virus.Hepatitis B virus DNA contains a glucocorticoid-responsive element.Nucleotide sequence of the 5' noncoding region and part of the gag gene of mouse mammary tumor virus; identification of the 5' splicing site for subgenomic mRNAs.Nuclear factor-I and activator protein-2 bind in a mutually exclusive way to overlapping promoter sequences and trans-activate the human growth hormone gene.The long terminal repeat region of the mouse mammary tumour virus contains multiple regulatory elementsTissue-specific and ubiquitous factors binding next to the glucocorticoid receptor modulate transcription from the mouse mammary tumor virus promoterStimulation of basal transcription from the mouse mammary tumor virus promoter by Oct proteins.A redundant nuclear protein binding site contributes to negative regulation of the mouse mammary tumor virus long terminal repeat.Cooperation between structural elements in hormono-regulated transcription from the mouse mammary tumor virus promoter.Identification of sequences responsible for acute-phase induction of human C-reactive protein.A model for hormone receptor binding to the mouse mammary tumour virus regulatory element based on hydroxyl radical footprinting.Regulatory DNA elements localized remotely upstream from the drug-metabolizing cytochrome P-450c gene.Contacts between hormone receptor and DNA double helix within a glucocorticoid regulatory element of mouse mammary tumor virusIsolation and characterization of the human prolactin gene.A mouse mammary tumor virus promoter element near the transcription initiation site
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P2860
Subfragments of the large terminal repeat cause glucocorticoid-responsive expression of mouse mammary tumor virus and of an adjacent gene.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 1983
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Subfragments of the large term ...... virus and of an adjacent gene.
@en
Subfragments of the large term ...... virus and of an adjacent gene.
@nl
type
label
Subfragments of the large term ...... virus and of an adjacent gene.
@en
Subfragments of the large term ...... virus and of an adjacent gene.
@nl
prefLabel
Subfragments of the large term ...... virus and of an adjacent gene.
@en
Subfragments of the large term ...... virus and of an adjacent gene.
@nl
P2093
P2860
P356
P1476
Subfragments of the large term ...... virus and of an adjacent gene.
@en
P2093
P2860
P304
P356
10.1073/PNAS.80.12.3637
P407
P577
1983-06-01T00:00:00Z