Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury. - Wikidata - awgldk - TriplyDB

Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury.

Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury.

http://www.wikidata.org/entity/Q37617674

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An ErChen and YinChen Decoction Ameliorates High-Fat-Induced Nonalcoholic Steatohepatitis in Rats by Regulating JNK1 Signaling Pathway.Mechanisms of acetaminophen hepatotoxicity and their translation to the human pathophysiology.Low-dose acetaminophen induces early disruption of cell-cell tight junctions in human hepatic cells and mouse liver.JNK at the crossroad of obesity, insulin resistance, and cell stress response.Schisandra Lignan Extract Protects against Carbon Tetrachloride-Induced Liver Injury in Mice by Inhibiting Oxidative Stress and Regulating the NF-κB and JNK Signaling Pathways.The novel intracellular protein CREG inhibits hepatic steatosis, obesity, and insulin resistance.Krüppel-like factor 6 is a transcriptional activator of autophagy in acute liver injury.The role of MAP2 kinases and p38 kinase in acute murine liver injury models Lipopolysaccharide downregulates macrophage-derived IL-22 to modulate alcohol-induced hepatocyte cell death.New insights into the role and mechanism of c-Jun-N-terminal kinase signaling in the pathobiology of liver diseases.The protective effect of the natural compound hesperetin against fulminant hepatitis in vivo and in vitro.EXTRAHEPATIC TOXICITY OF ACETAMINOPHEN: CRITICAL EVALUATION OF THE EVIDENCE AND PROPOSED MECHANISMS.Phosphatidylcholine transfer protein/StarD2 promotes microvesicular steatosis and liver injury in murine experimental steatohepatitis.Dissecting the molecular pathophysiology of drug-induced liver injury.Mitochondrial-Lysosomal Axis in Acetaminophen Hepatotoxicity.Mechanisms of acetaminophen-induced liver injury and its implications for therapeutic interventions.

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Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury.

http://www.wikidata.org/entity/Q37617674

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 18 December 2015

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury.

@en

Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury.

@nl

type

label

Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury.

@en

Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury.

@nl

prefLabel

Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury.

@en

Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury.

@nl

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J.GASTRO.2015.12.019

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Gastroenterology

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Combined Activities of JNK1 and JNK2 in Hepatocytes Protect Against Toxic Liver Injury

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Christian Preisinger

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Christian Trautwein

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Gang Zhao

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Jin Peng

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Malika Al Masaoudi

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Miguel Eugenio Zoubek

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Nikolaus Gassler

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Roger J Davis

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Tom Luedde

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Wei Hu

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P2860

Apoptosis, pyroptosis, and necrosis: mechanistic description of dead and dying eukaryotic cells.

Acetylcysteine for acetaminophen poisoning

Signal transduction by the JNK group of MAP kinases

A liver full of JNK: signaling in regulation of cell function and disease pathogenesis, and clinical approaches

Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosis.

Disruption of the Jnk2 (Mapk9) gene reduces destructive insulitis and diabetes in a mouse model of type I diabetes.

Molecular forms of HMGB1 and keratin-18 as mechanistic biomarkers for mode of cell death and prognosis during clinical acetaminophen hepatotoxicity

Mitochondria-targeted cytochrome P450 2E1 induces oxidative damage and augments alcohol-mediated oxidative stress.

Acetaminophen-induced acute liver failure: results of a United States multicenter, prospective study.

Suppression of p53-dependent senescence by the JNK signal transduction pathway

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scholarly article

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10.1053/J.GASTRO.2015.12.019

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Mark V. Boekschoten

Michael Müller

Lars P. Bechmann

Yulia A Nevzorova

Francisco Javier Cubero

Christian Liedtke

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2015-12-19T00:00:00Z

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