Systematic inactivation and phenotypic characterization of two-component signal transduction systems of Enterococcus faecalis V583.
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Stress Physiology of Lactic Acid BacteriaStructure of the response regulator VicR DNA-binding domainMultiple posttranscriptional regulatory mechanisms partner to control ethanolamine utilization in Enterococcus faecalisEthanolamine activates a sensor histidine kinase regulating its utilization in Enterococcus faecalisA sensor histidine kinase co-ordinates cell wall architecture with cell division in Bacillus subtilisPopulation Density Modulates Drug Inhibition and Gives Rise to Potential Bistability of Treatment Outcomes for Bacterial InfectionsThe transcriptome of the nosocomial pathogen Enterococcus faecalis V583 reveals adaptive responses to growth in blood.Screening of in vivo activated genes in Enterococcus faecalis during insect and mouse infections and growth in urineDevelopment of genetic tools for in vivo virulence analysis of Streptococcus sanguinis.YycH regulates the activity of the essential YycFG two-component system in Bacillus subtilisMechanism of chromosomal transfer of Enterococcus faecalis pathogenicity island, capsule, antimicrobial resistance, and other traits.Whole-genome analyses of Enterococcus faecium isolates with diverse daptomycin MICs.Regulation of the pspA virulence factor and essential pcsB murein biosynthetic genes by the phosphorylated VicR (YycF) response regulator in Streptococcus pneumoniae.MurAA is required for intrinsic cephalosporin resistance of Enterococcus faecalisFull activation of Enterococcus faecalis gelatinase by a C-terminal proteolytic cleavage.The two-component system GrvRS (EtaRS) regulates ace expression in Enterococcus faecalis OG1RF.Oxidative stress enhances cephalosporin resistance of Enterococcus faecalis through activation of a two-component signaling system.Regulation of virulence by the RevR response regulator in Clostridium perfringens.Genomic comparative analysis of the environmental Enterococcus mundtii against enterococcal representative species.Reciprocal regulation of cephalosporin resistance in Enterococcus faecalis.Mechanisms of antibiotic resistance in enterococci.YycH and YycI interact to regulate the essential YycFG two-component system in Bacillus subtilis.Mutations in the β subunit of RNA polymerase alter intrinsic cephalosporin resistance in Enterococci.Comparative genomics of Enterococcus spp. isolated from bovine feces.New insights into the WalK/WalR (YycG/YycF) essential signal transduction pathway reveal a major role in controlling cell wall metabolism and biofilm formation in Staphylococcus aureusInvolvement of sensor kinases in the stress tolerance response of Streptococcus mutans.Whole-genome analysis of a daptomycin-susceptible enterococcus faecium strain and its daptomycin-resistant variant arising during therapyModes and modulations of antibiotic resistance gene expression.Two-component signal transduction proteins as potential drug targets in medically important fungi.Enterococcal Rgg-like regulator ElrR activates expression of the elrA operon.A fratricidal mechanism is responsible for eDNA release and contributes to biofilm development of Enterococcus faecalis.Nutritional control of antibiotic resistance via an interface between the phosphotransferase system and a two-component signaling system.Genetic basis for vancomycin-enhanced cephalosporin susceptibility in vancomycin-resistant enterococci revealed using counterselection with dominant-negative thymidylate synthase.New class of competitive inhibitor of bacterial histidine kinases.Functional Dissection of the CroRS Two-Component System Required for Resistance to Cell Wall Stressors in Enterococcus faecalis.Extracellular SalB contributes to intrinsic cephalosporin resistance and cell envelope integrity in Enterococcus faecalis.Involvement of WalK (VicK) phosphatase activity in setting WalR (VicR) response regulator phosphorylation level and limiting cross-talk in Streptococcus pneumoniae D39 cells.Tracing the Enterococci from Paleozoic Origins to the Hospital.Requirement of the CroRS Two-Component System for Resistance to Cell Wall-Targeting Antimicrobials in Enterococcus faecium.The Response Regulator YycF Inhibits Expression of the Fatty Acid Biosynthesis Repressor FabT in Streptococcus pneumoniae.
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Systematic inactivation and phenotypic characterization of two-component signal transduction systems of Enterococcus faecalis V583.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2004
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Systematic inactivation and ph ...... of Enterococcus faecalis V583.
@en
Systematic inactivation and ph ...... of Enterococcus faecalis V583.
@nl
type
label
Systematic inactivation and ph ...... of Enterococcus faecalis V583.
@en
Systematic inactivation and ph ...... of Enterococcus faecalis V583.
@nl
prefLabel
Systematic inactivation and ph ...... of Enterococcus faecalis V583.
@en
Systematic inactivation and ph ...... of Enterococcus faecalis V583.
@nl
P2860
P1476
Systematic inactivation and ph ...... of Enterococcus faecalis V583.
@en
P2093
Lynn E Hancock
Marta Perego
P2860
P304
P356
10.1128/JB.186.23.7951-7958.2004
P407
P577
2004-12-01T00:00:00Z