Paclitaxel-resistant cancer cell-derived secretomes elicit ABCB1-associated docetaxel cross-resistance and escape from apoptosis through FOXO3a-driven glycolytic regulation.
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Paraoxonase-1 (PON1) induces metastatic potential and apoptosis escape via its antioxidative function in lung cancer cells.Combined Inhibitions of Glycolysis and AKT/autophagy Can Overcome Resistance to EGFR-targeted Therapy of Lung Cancer.Pharmacological treatment with inhibitors of nuclear export enhances the antitumor activity of docetaxel in human prostate cancer.Variable Cell Line Pharmacokinetics Contribute to Non-Linear Treatment Response in Heterogeneous Cell Populations.
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Paclitaxel-resistant cancer cell-derived secretomes elicit ABCB1-associated docetaxel cross-resistance and escape from apoptosis through FOXO3a-driven glycolytic regulation.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 20 January 2017
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Paclitaxel-resistant cancer ce ...... -driven glycolytic regulation.
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Paclitaxel-resistant cancer ce ...... -driven glycolytic regulation.
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type
label
Paclitaxel-resistant cancer ce ...... -driven glycolytic regulation.
@en
Paclitaxel-resistant cancer ce ...... -driven glycolytic regulation.
@nl
prefLabel
Paclitaxel-resistant cancer ce ...... -driven glycolytic regulation.
@en
Paclitaxel-resistant cancer ce ...... -driven glycolytic regulation.
@nl
P2860
P356
P1476
Paclitaxel-resistant cancer ce ...... -driven glycolytic regulation.
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P2093
Ji-Young Hong
P2860
P2888
P356
10.1038/EMM.2016.131
P577
2017-01-20T00:00:00Z
P6179
1053895254