IL-1 receptor blockade restores autophagy and reduces inflammation in chronic granulomatous disease in mice and in humans. - Wikidata - awgldk - TriplyDB

IL-1 receptor blockade restores autophagy and reduces inflammation in chronic granulomatous disease in mice and in humans.

IL-1 receptor blockade restores autophagy and reduces inflammation in chronic granulomatous disease in mice and in humans.

http://www.wikidata.org/entity/Q37627905

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The Role of Autophagy-Related Proteins in Candida albicans Infections Roles of Autophagy and Autophagy-Related Proteins in Antifungal Immunity Genetic disorders coupled to ROS deficiency The cooperation between the autophagy machinery and the inflammasome to implement an appropriate innate immune response: do they regulate each other?Paving the way for predictive diagnostics and personalized treatment of invasive aspergillosis Genetics of inflammatory bowel disease from multifactorial to monogenic forms Modulation of inflammation by autophagy: Consequences for human disease Neutrophils: Between host defence, immune modulation, and tissue injury Successful hepatectomy for hepatic abscess with chronic granulomatous disease: a case report.European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Innate Lung Defense during Invasive Aspergillosis: New Mechanisms.Apoptotic neutrophils augment the inflammatory response to Mycobacterium tuberculosis infection in human macrophages.NOX2-dependent regulation of inflammation Autophagy protects C. elegans against necrosis during Pseudomonas aeruginosa infection.Applying Convergent Immunity to Innovative Vaccines Targeting Staphylococcus aureus.The complex roles of NADPH oxidases in fungal infection.Moderate hypoxia potentiates interleukin-1β production in activated human macrophages.Phagocyte NADPH oxidase, chronic granulomatous disease and mycobacterial infections.IL-1α signaling is critical for leukocyte recruitment after pulmonary Aspergillus fumigatus challenge.An expanding role for interleukin-1 blockade from gout to cancer.Small-molecule control of cytokine function: new opportunities for treating immune disorders Transcriptome analysis of cyclic AMP-dependent protein kinase A-regulated genes reveals the production of the novel natural compound fumipyrrole by Aspergillus fumigatus.The syndrome of hemophagocytic lymphohistiocytosis in primary immunodeficiencies: implications for differential diagnosis and pathogenesis.Extracellular Vesicles Derived from Bone Marrow Mesenchymal Stem Cells Protect against Experimental Colitis via Attenuating Colon Inflammation, Oxidative Stress and Apoptosis When the Fight against Fungi Goes Wrong ATG16L1 deficiency in macrophages drives clearance of uropathogenic E. coli in an IL-1β-dependent manner Maintaining intestinal health: the genetics and immunology of very early onset inflammatory bowel disease.Commensal Bacteria-Induced Inflammasome Activation in Mouse and Human Macrophages Is Dependent on Potassium Efflux but Does Not Require Phagocytosis or Bacterial Viability.Molecular characterization of LC3-associated phagocytosis reveals distinct roles for Rubicon, NOX2 and autophagy proteins Toward an Inclusive, Congruent, and Precise Definition of Autoinflammatory Diseases Commensal Bacteria-induced Interleukin 1β (IL-1β) Secreted by Macrophages Up-regulates Hepcidin Expression in Hepatocytes by Activating the Bone Morphogenetic Protein Signaling Pathway.NADPH oxidase controls neutrophilic response to sterile inflammation in mice by regulating the IL-1α/G-CSF axis.Detection of a microbial metabolite by STING regulates inflammasome activation in response to Chlamydia trachomatis infection.IL-1 receptor antagonist ameliorates inflammasome-dependent inflammation in murine and human cystic fibrosis The DNA methylation profile of activated human natural killer cells.Intestinal Autophagy and Its Pharmacological Control in Inflammatory Bowel Disease.The role of interleukin-1 family members in the host defence against Aspergillus fumigatus.Role of inflammasomes in intestinal inflammation and Crohn's disease.The inflammasome and IL-1β: implications for the treatment of inflammatory diseases.The interplay between inflammasome activation and antifungal host defense.

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IL-1 receptor blockade restores autophagy and reduces inflammation in chronic granulomatous disease in mice and in humans.

http://www.wikidata.org/entity/Q37627905

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article científic

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bilimsel makale

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scientific article published on 18 February 2014

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vetenskaplig artikel

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IL-1 receptor blockade restore ...... disease in mice and in humans.

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IL-1 receptor blockade restore ...... disease in mice and in humans.

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IL-1 receptor blockade restore ...... disease in mice and in humans.

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Proceedings of the National Academy of Sciences of the United States of America

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IL-1 receptor blockade restore ...... disease in mice and in humans.

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Andrea Casagrande

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Frank L van de Veerdonk

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Georgios Chamilos

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Jos W M van der Meer

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Kara L Conway

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Leo A B Joosten

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Mihai G Netea

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Ramnik J Xavier

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Rossana Iannitti

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Sanne P Smeekens

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P2860

Activation of autophagy by inflammatory signals limits IL-1β production by targeting ubiquitinated inflammasomes for destruction

Reactive oxygen species-independent activation of the IL-1beta inflammasome in cells from patients with chronic granulomatous disease

In vivo analysis of autophagy in response to nutrient starvation using transgenic mice expressing a fluorescent autophagosome marker

Reactive oxygen species are essential for autophagy and specifically regulate the activity of Atg4

IL-1 receptor antagonist ameliorates inflammasome-dependent alcoholic steatohepatitis in mice

Beyond oxidative stress: an immunologist's guide to reactive oxygen species

Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production

Toll-like receptor signalling in macrophages links the autophagy pathway to phagocytosis

Sequence variants in the autophagy gene IRGM and multiple other replicating loci contribute to Crohn's disease susceptibility

A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1

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3526-3531

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10.1073/PNAS.1322831111

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111

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Theo S Plantinga

Mark S Gresnigt

Antonella De Luca

Charles A. Dinarello

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2014-02-18T00:00:00Z

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