Evidence of a common mechanism of disassembly of adherens junctions through Gα13 targeting of VE-cadherin. - Wikidata - awgldk - TriplyDB

Evidence of a common mechanism of disassembly of adherens junctions through Gα13 targeting of VE-cadherin.

Evidence of a common mechanism of disassembly of adherens junctions through Gα13 targeting of VE-cadherin.

http://www.wikidata.org/entity/Q37630213

about

Regulation of Endothelial Adherens Junctions by Tyrosine Phosphorylation Pyk2 phosphorylation of VE-PTP downstream of STIM1-induced Ca2+ entry regulates disassembly of adherens junctions.Protein tyrosine kinase 6 mediates TNFα-induced endothelial barrier dysfunction.Cytoskeletal mechanisms regulating vascular endothelial barrier function in response to acute lung injury Transient receptor potential channel 1 maintains adherens junction plasticity by suppressing sphingosine kinase 1 expression to induce endothelial hyperpermeability.Cancer Cell Invasion in Three-dimensional Collagen Is Regulated Differentially by Gα13 Protein and Discoidin Domain Receptor 1-Par3 Protein Signaling Mutant B-Raf(V600E) Promotes Melanoma Paracellular Transmigration by Inducing Thrombin-mediated Endothelial Junction Breakdown Gene control of tyrosine kinase TIE2 and vascular manifestations of infections.Rab5-mediated VE-cadherin internalization regulates the barrier function of the lung microvascular endothelium.p120-catenin regulates VE-cadherin endocytosis and degradation induced by the Kaposi sarcoma-associated ubiquitin ligase K5.Regulation of endothelial barrier function by p120-catenin∙VE-cadherin interaction.The pulmonary endothelium in acute respiratory distress syndrome: insights and therapeutic opportunities.HIF2α signaling inhibits adherens junctional disruption in acute lung injury.Endothelial Cell Junctional Adhesion Molecules: Role and Regulation of Expression in Inflammation.Regulation of pulmonary endothelial barrier function by kinases.Protein Interactions at Endothelial Junctions and Signaling Mechanisms Regulating Endothelial Permeability.Mind the gap: mechanisms regulating the endothelial barrier.Novel role of reactive oxygen species-activated Trp melastatin channel-2 in mediating angiogenesis and postischemic neovascularization.Integrin β1 controls VE-cadherin localization and blood vessel stability.Endothelial cell SHP-2 negatively regulates neutrophil adhesion and promotes transmigration by enhancing ICAM-1-VE-cadherin interaction.Deubiquitinase function of A20 maintains and repairs endothelial barrier after lung vascular injury.Hypercapnic acidosis attenuates pressure-dependent increase in whole-lung filtration coefficient (Kf).

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Evidence of a common mechanism of disassembly of adherens junctions through Gα13 targeting of VE-cadherin.

http://www.wikidata.org/entity/Q37630213

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 03 March 2014

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Evidence of a common mechanism ...... Gα13 targeting of VE-cadherin.

@en

Evidence of a common mechanism ...... Gα13 targeting of VE-cadherin.

@nl

type

label

Evidence of a common mechanism ...... Gα13 targeting of VE-cadherin.

@en

Evidence of a common mechanism ...... Gα13 targeting of VE-cadherin.

@nl

prefLabel

Evidence of a common mechanism ...... Gα13 targeting of VE-cadherin.

@en

Evidence of a common mechanism ...... Gα13 targeting of VE-cadherin.

@nl

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Journal of Experimental Medicine

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Evidence of a common mechanism ...... Gα13 targeting of VE-cadherin.

@en

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Alexander Garcia

http://www.w3.org/2001/XMLSchema#string

Asrar B Malik

http://www.w3.org/2001/XMLSchema#string

Dolly Mehta

http://www.w3.org/2001/XMLSchema#string

Haixia Gong

http://www.w3.org/2001/XMLSchema#string

M Rizwan Siddiqui

http://www.w3.org/2001/XMLSchema#string

Marcelo G Bonini

http://www.w3.org/2001/XMLSchema#string

Shaoting Feng

http://www.w3.org/2001/XMLSchema#string

Stephen M Vogel

http://www.w3.org/2001/XMLSchema#string

Xiaopei Gao

http://www.w3.org/2001/XMLSchema#string

Yulia Komarova

http://www.w3.org/2001/XMLSchema#string

P2860

Vascular system defects and impaired cell chemokinesis as a result of Galpha13 deficiency

Cellular levels of p120 catenin function as a set point for cadherin expression levels in microvascular endothelial cells

VEGF controls endothelial-cell permeability by promoting the beta-arrestin-dependent endocytosis of VE-cadherin

Protein kinase Calpha-induced p115RhoGEF phosphorylation signals endothelial cytoskeletal rearrangement

The G protein G alpha(13) is required for growth factor-induced cell migration

Src kinase regulation by phosphorylation and dephosphorylation

p115 RhoGEF, a GTPase activating protein for Galpha12 and Galpha13

CSK: a protein-tyrosine kinase involved in regulation of src family kinases

The blood-brain-barrier in multiple sclerosis: functional roles and therapeutic targeting

G alpha(i) and G alpha(o) are target proteins of reactive oxygen species

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579-591

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scholarly article

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10.1084/JEM.20131190

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3

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211

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2014-03-03T00:00:00Z

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260485902

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24590762

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3949568

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