Extracellular ATP drives systemic inflammation, tissue damage and mortality.
about
Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular PathwaysThe interplay between inflammation and metabolism in rheumatoid arthritisBioenergetic dysfunction and inflammation in Alzheimer's disease: a possible connectionP2X ion channel receptors and inflammationP2X3 purinergic receptor overexpression is associated with poor recurrence-free survival in hepatocellular carcinoma patientsCanonical and Novel Non-Canonical Cholinergic Agonists Inhibit ATP-Induced Release of Monocytic Interleukin-1β via Different Combinations of Nicotinic Acetylcholine Receptor Subunits α7, α9 and α10.Oxidants in Acute and Chronic Lung DiseaseClassification of current anticancer immunotherapiesP2X3 receptors mediate visceral hypersensitivity during acute chemically-induced colitis and in the post-inflammatory phase via different mechanisms of sensitization.Mass Spectrometry Imaging Reveals Elevated Glomerular ATP/AMP in Diabetes/obesity and Identifies Sphingomyelin as a Possible MediatorModulation of radiochemoimmunotherapy-induced B16 melanoma cell death by the pan-caspase inhibitor zVAD-fmk induces anti-tumor immunity in a HMGB1-, nucleotide- and T-cell-dependent manner.Neutrophil P2X7 receptors mediate NLRP3 inflammasome-dependent IL-1β secretion in response to ATP.Purinergic receptors as potential therapeutic targets in Alzheimer's disease.Purinergic Signaling and the Immune Response in Sepsis: A ReviewATP-Induced Inflammasome Activation and Pyroptosis Is Regulated by AMP-Activated Protein Kinase in Macrophages.Molecular regulation of cell fate in cerebral ischemia: role of the inflammasome and connected pathways.Redox stress unbalances the inflammatory cytokine network: role in autoinflammatory patients and healthy subjects.Pannexins Are Potential New Players in the Regulation of Cerebral Homeostasis during Sleep-Wake Cycle.Transcriptomic profiling of a chicken lung epithelial cell line (CLEC213) reveals a mitochondrial respiratory chain activity boost during influenza virus infection.Purinergic A2b Receptor Activation by Extracellular Cues Affects Positioning of the Centrosome and Nucleus and Causes Reduced Cell Migration.Mitochondrial dysfunction in inflammatory responses and cellular senescence: pathogenesis and pharmacological targets for chronic lung diseases.Mitochondrial lysates induce inflammation and Alzheimer's disease-relevant changes in microglial and neuronal cells.Gene Expression and Activity Profiling Reveal a Significant Contribution of Exo-Phosphotransferases to the Extracellular Nucleotides Metabolism in HUVEC Endothelial Cells.Paradoxical roles of dual oxidases in cancer biology.Systemic Adenosine Triphosphate Impairs Neutrophil Chemotaxis and Host Defense in Sepsis.Total tanshinones exhibits anti-inflammatory effects through blocking TLR4 dimerization via the MyD88 pathway.Alkaline Phosphatase, an Unconventional Immune Protein.Antimicrobial cathelicidin peptide LL-37 inhibits the pyroptosis of macrophages and improves the survival of polybacterial septic mice.Extracellular ATP activates eNOS and increases intracellular NO generation in Red Blood Cells.Consensus guidelines for the detection of immunogenic cell death.The NLPR3 inflammasome and obesity-related kidney disease.ATP-degrading ENPP1 is required for survival (or persistence) of long-lived plasma cells.Maternal Glucose Supplementation in a Murine Model of Chorioamnionitis Alleviates Dysregulation of Autophagy in Fetal Brain.A high concentration of fatty acids induces TNF-α as well as NO release mediated by the P2X4 receptor, and the protective effects of puerarin in RAW264.7 cells.Unfolding Role of a Danger Molecule Adenosine Signaling in Modulation of Microbial Infection and Host Cell Response.TBK1 at the Crossroads of Inflammation and Energy Homeostasis in Adipose Tissue.The P2X7 Receptor in Inflammatory Diseases: Angel or Demon?Burn-Related Dysregulation of Inflammation and Immunity in Experimental and Clinical Studies.Mechanisms of ATP Release by Inflammatory Cells.Electrophysiological Evidence for Functional Astrocytic P2X3 Receptors in the Mouse Trigeminal Caudal Nucleus.
P2860
Q26745537-BB8048D3-1061-4DD5-B528-0521A9A09706Q26785751-74F464EB-EF5A-46D3-AFE4-F8D9CA3584EAQ26822410-3B7DCDBF-C9D9-409E-A916-8F07C8FDB1F7Q28066934-F55396E6-1488-459A-A850-A513F7B6EA23Q28386398-D55A9481-0222-4CC2-8B31-7E72D9B69C50Q33871018-FBF7BAD7-17F8-4630-BAB9-988143E9EEC6Q35104183-1AFD56EF-3518-4760-A281-90BD221B5E8FQ35149637-A9A199DB-76FE-4780-87D3-517EB3D9F059Q35464242-20D6F9CB-51F0-49E5-9826-3461BC234ACAQ36056764-C8DFF9F2-592E-42B9-BF91-EF81746271A7Q36346964-6CFDC855-0BF1-46DE-B206-53C79F70C5E2Q36587518-994A9EB2-EBC0-467F-A633-45AD5D61A043Q36844881-50217A35-E620-4CB7-ADFC-2521F9336C27Q36923670-892BAB3A-1F13-4E31-B99E-351EC83A9CEAQ37492136-307BA4FB-862F-4615-A1B4-092F6ED78C84Q38250196-287C4B12-0A57-4AF5-A5BC-66B59CC69474Q38551809-69724FC4-99D1-4809-8DED-5A4BC79614BEQ38645696-397A4E60-0791-46BF-A841-E35724835D61Q38706160-835CF463-D3FD-4E57-B018-A83FAE9E34A2Q38768350-99058753-99EB-4CCF-B7CF-D05FD4F5F62CQ38836727-DEF8B21F-FD62-409E-A63D-3855CD00735BQ38925765-3475BCD3-772C-4E89-8906-E795AD92D3F1Q39177365-667A60D8-1414-4B74-9403-B4EEE5152A44Q39348472-9EAB75B9-2C2D-46D5-BB1B-71121C8350C2Q40567390-B6522E0C-D6DC-48BE-9AD3-3F1BD0684869Q41168560-38AE9D27-74EC-457D-808E-46372CF526A3Q41221628-D5A0A529-48B0-4ACA-BA91-811A30298ADCQ41949365-A4C9157E-D4E5-4A09-890A-43C4B2D23C6EQ42516919-03257077-745A-489E-8C27-4A73B35BE974Q42553556-1641EFF4-B7B7-434B-91D1-0798A42D249EQ47130640-3BEFF938-8DAF-423C-909C-EB0F24B02E8DQ47153776-B5D145C5-86B7-4704-A88F-0BEAAE101330Q47583076-1CF07384-7414-4A2D-BBF2-EB33BA2C1009Q47942618-BAF37488-434D-461B-8734-CE64AB44754CQ48284423-06D8C312-11CD-4A38-8A2C-E68DEA41821FQ50098101-F706D08D-C616-4829-AFD3-41B22FA354B4Q50202418-03D1D1B3-66F7-4F8E-9D99-78280B5BC578Q52781949-0DCC9610-E0CE-44E5-B74A-51EE519C9A2CQ55026641-6FD9DA09-4B76-4B69-B6D2-EA90B9CC0804Q55350404-BA7FB5D1-5A46-4798-A118-229948E90977
P2860
Extracellular ATP drives systemic inflammation, tissue damage and mortality.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 06 March 2014
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Extracellular ATP drives systemic inflammation, tissue damage and mortality.
@en
Extracellular ATP drives systemic inflammation, tissue damage and mortality.
@nl
type
label
Extracellular ATP drives systemic inflammation, tissue damage and mortality.
@en
Extracellular ATP drives systemic inflammation, tissue damage and mortality.
@nl
prefLabel
Extracellular ATP drives systemic inflammation, tissue damage and mortality.
@en
Extracellular ATP drives systemic inflammation, tissue damage and mortality.
@nl
P2093
P2860
P356
P1476
Extracellular ATP drives systemic inflammation, tissue damage and mortality
@en
P2093
P2860
P356
10.1038/CDDIS.2014.70
P577
2014-03-06T00:00:00Z