Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death. - Wikidata - awgldk - TriplyDB

Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

http://www.wikidata.org/entity/Q37683265

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Matricellular protein CCN1 activates a proinflammatory genetic program in murine macrophages Functions and mechanisms of action of CCN matricellular proteins JNK Signaling: Regulation and Functions Based on Complex Protein-Protein Partnerships Novel Strategy for Treatment of Viral Central Nervous System Infection by Using a Cell-Permeating Inhibitor of c-Jun N-Terminal Kinase Profiling Subcellular Protein Phosphatase Responses to Coxsackievirus B3 Infection of Cardiomyocytes.ER stress, autophagy, and RNA viruses.CCN1/CYR61: the very model of a modern matricellular protein.Stress-activated protein kinases are involved in coxsackievirus B3 viral progeny release.Pregnancy loss following coxsackievirus b3 infection in mice during early gestation due to high expression of coxsackievirus-adenovirus receptor (CAR) in uterus and embryo Rotavirus activates JNK and p38 signaling pathways in intestinal cells, leading to AP-1-driven transcriptional responses and enhanced virus replication Uses for JNK: the many and varied substrates of the c-Jun N-terminal kinases.Extracellular matrix protein CCN1 limits oncolytic efficacy in glioma.Validity of Klotho, CYR61 and YKL-40 as ideal predictive biomarkers for acute kidney injury: review study.Oncolytic HSV-1 infection of tumors induces angiogenesis and upregulates CYR61 Fas-mediated apoptotic signaling in the mouse brain following reovirus infection BX-795 inhibits HSV-1 and HSV-2 replication by blocking the JNK/p38 pathways without interfering with PDK1 activity in host cells.Death waits for no man--does it wait for a virus? How enteroviruses induce and control cell death Emerging role of CCN family proteins in tumorigenesis and cancer metastasis (Review).A mechanism of immunoreceptor tyrosine-based activation motif (ITAM)-like sequences in the capsid protein VP2 in viral growth and pathogenesis of Coxsackievirus B3.Poliovirus induces Bax-dependent cell death mediated by c-Jun NH2-terminal kinase.Genomic expression profiling of human inflammatory cardiomyopathy (DCMi) suggests novel therapeutic targets.LY294002 and Rapamycin promote coxsackievirus-induced cytopathic effect and apoptosis via inhibition of PI3K/AKT/mTOR signaling pathway.Dysregulation of the ubiquitin-proteasome system by curcumin suppresses coxsackievirus B3 replication.Coxsackievirus B3 used as a gene therapy vector to express functional FGF2.SIRT2 regulates oxidative stress-induced cell death through deacetylation of c-Jun NH2-terminal kinase.The cytotoxicity of coxsackievirus B3 is associated with a blockage of autophagic flux mediated by reduced syntaxin 17 expression.Caveolin-1 regulates the secretion and cytoprotection of Cyr61 in hyperoxic cell death.

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Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

http://www.wikidata.org/entity/Q37683265

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

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scientific article published on December 2004

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

@en

Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

@nl

type

label

Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

@en

Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

@nl

prefLabel

Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

@en

Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

@nl

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JVI.78.24.13479-13488.2004

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Journal of Virology

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Coxsackievirus B3 infection induces cyr61 activation via JNK to mediate cell death.

@en

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Ha-Young Hwang

http://www.w3.org/2001/XMLSchema#string

Inho Jo

http://www.w3.org/2001/XMLSchema#string

Jae-Hwan Nam

http://www.w3.org/2001/XMLSchema#string

Joo-Young Kim

http://www.w3.org/2001/XMLSchema#string

Jung-Hyun Park

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Kwang-Chul Chung

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Sang-Ick Park

http://www.w3.org/2001/XMLSchema#string

Sun-Ku Chung

http://www.w3.org/2001/XMLSchema#string

Sun-Mi Kim

http://www.w3.org/2001/XMLSchema#string

P2860

Apoptosis in coxsackievirus B3-caused diseases: interaction between the capsid protein VP2 and the proapoptotic protein siva

A new type of papillomavirus DNA, its presence in genital cancer biopsies and in cell lines derived from cervical cancer

Expression of cyr61, a growth factor-inducible immediate-early gene

Promoter function and structure of the growth factor-inducible immediate early gene cyr61

Brefeldin A: insights into the control of membrane traffic and organelle structure

CYR61, a product of a growth factor-inducible immediate early gene, promotes angiogenesis and tumor growth

Interference of hepatitis C virus RNA replication by short interfering RNAs

Duplexes of 21-nucleotide RNAs mediate RNA interference in cultured mammalian cells

From receptors to stress-activated MAP kinases

Cyr61, product of a growth factor-inducible immediate-early gene, regulates chondrogenesis in mouse limb bud mesenchymal cells

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13479-13488

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10.1128/JVI.78.24.13479-13488.2004

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