Activation of expression of latent Epstein-Barr herpesvirus after gene transfer with a small cloned subfragment of heterogeneous viral DNA
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Epstein-barr virus immediate-early protein BZLF1 is SUMO-1 modified and disrupts promyelocytic leukemia bodiesSignal Transduction and Transcription Factor Modification during Reactivation of Epstein-Barr Virus from LatencyThe Epstein-Barr virus replication protein BBLF2/3 provides an origin-tethering function through interaction with the zinc finger DNA binding protein ZBRK1 and the KAP-1 corepressorThe C-mer gene is induced by Epstein-Barr virus immediate-early protein BRLF1The cellular proteins that bind specifically to the Epstein-Barr virus origin of plasmid DNA replication belong to a gene familyZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virusMutual inhibition between Kaposi's sarcoma-associated herpesvirus and Epstein-Barr virus lytic replication initiators in dually-infected primary effusion lymphomaThe B-cell specific transcription factor, Oct-2, promotes Epstein-Barr virus latency by inhibiting the viral immediate-early protein, BZLF1Reactive oxygen species mediate Epstein-Barr virus reactivation by N-methyl-N'-nitro-N-nitrosoguanidineTpl2/AP-1 enhances murine gammaherpesvirus 68 lytic replication.Epstein-Barr virus immediate-early protein Zta co-opts mitochondrial single-stranded DNA binding protein to promote viral and inhibit mitochondrial DNA replication.Methylation-dependent binding of the epstein-barr virus BZLF1 protein to viral promotersA viral gene that activates lytic cycle expression of Kaposi's sarcoma-associated herpesvirus.The Epstein-Barr virus early protein EB1 activates transcription from different responsive elements including AP-1 binding sitesEpstein-Barr virus bicistronic mRNAs generated by facultative splicing code for two transcriptional trans-activators.AP-1 homolog BZLF1 of Epstein-Barr virus has two essential functions dependent on the epigenetic state of the viral genome.Genome rearrangements activate the Epstein-Barr virus gene whose product disrupts latency.The Epstein-Barr virus lytic transactivator Zta interacts with the helicase-primase replication proteins.Epstein-Barr virus immediate-early proteins BZLF1 and BRLF1 activate the ATF2 transcription factor by increasing the levels of phosphorylated p38 and c-Jun N-terminal kinasesHistone acetylation and reactivation of Epstein-Barr virus from latency.Rta of murine gammaherpesvirus 68 reactivates the complete lytic cycle from latency.The K-bZIP protein from Kaposi's sarcoma-associated herpesvirus interacts with p53 and represses its transcriptional activity.The Epstein-Barr virus protein BRLF1 activates S phase entry through E2F1 induction.The Epstein-Barr virus BZLF1 protein interacts physically and functionally with the histone acetylase CREB-binding protein.Sumoylation of the Epstein-Barr virus BZLF1 protein inhibits its transcriptional activity and is regulated by the virus-encoded protein kinase.Epstein-Barr virus WZhet DNA can induce lytic replication in epithelial cells in vitro, although WZhet is not detectable in many human tissues in vivo.Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8 RTA reactivates murine gammaherpesvirus 68 from latency.Identification of a novel element involved in regulation of the lytic switch BZLF1 gene promoter of Epstein-Barr virusOrigin-independent assembly of Kaposi's sarcoma-associated herpesvirus DNA replication compartments in transient cotransfection assays and association with the ORF-K8 protein and cellular PML.Epstein-Barr virus immediate-early protein BRLF1 induces the lytic form of viral replication through a mechanism involving phosphatidylinositol-3 kinase activation.Epstein-Barr virus immediate-early protein BRLF1 interacts with CBP, promoting enhanced BRLF1 transactivation.BZLF1 activation of the methylated form of the BRLF1 immediate-early promoter is regulated by BZLF1 residue 186.Either ZEB1 or ZEB2/SIP1 can play a central role in regulating the Epstein-Barr virus latent-lytic switch in a cell-type-specific manner.Both Epstein-Barr virus (EBV)-encoded trans-acting factors, EB1 and EB2, are required to activate transcription from an EBV early promoter.Epstein-Barr virus small nuclear RNAs are not expressed in permissively infected cells in AIDS-associated leukoplakiaActivation and repression of Epstein-Barr Virus and Kaposi's sarcoma-associated herpesvirus lytic cycles by short- and medium-chain fatty acidsKaposi's sarcoma-associated herpesvirus K-bZIP represses gene transcription via SUMO modification.The genetic approach to the Epstein-Barr virus: from basic virology to gene therapy.Evidence for coiled-coil dimer formation by an Epstein-Barr virus transactivator that lacks a heptad repeat of leucine residues.BHRF1 of Epstein-Barr virus, which is homologous to human proto-oncogene bcl2, is not essential for transformation of B cells or for virus replication in vitro.
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P2860
Activation of expression of latent Epstein-Barr herpesvirus after gene transfer with a small cloned subfragment of heterogeneous viral DNA
description
article científic
@ca
article scientifique
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articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 1985
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Activation of expression of la ...... ent of heterogeneous viral DNA
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Activation of expression of la ...... nt of heterogeneous viral DNA.
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type
label
Activation of expression of la ...... ent of heterogeneous viral DNA
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Activation of expression of la ...... nt of heterogeneous viral DNA.
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prefLabel
Activation of expression of la ...... ent of heterogeneous viral DNA
@en
Activation of expression of la ...... nt of heterogeneous viral DNA.
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P2860
P356
P1476
Activation of expression of la ...... ent of heterogeneous viral DNA
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P2093
J Countryman
P2860
P304
P356
10.1073/PNAS.82.12.4085
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P577
1985-06-01T00:00:00Z