LGR4/GPR48 inactivation leads to aniridia-genitourinary anomalies-mental retardation syndrome defects. - Wikidata - awgldk - TriplyDB

LGR4/GPR48 inactivation leads to aniridia-genitourinary anomalies-mental retardation syndrome defects.

LGR4/GPR48 inactivation leads to aniridia-genitourinary anomalies-mental retardation syndrome defects.

http://www.wikidata.org/entity/Q37691705

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Lgr4 protein deficiency induces ataxia-like phenotype in mice and impairs long term depression at cerebellar parallel fiber-Purkinje cell synapses Targeted deletion of the murine Lgr4 gene decreases lens epithelial cell resistance to oxidative stress and induces age-related cataract formation.Dysgerminoma developing from an ectopic ovary in a patient with WAGR syndrome: A case report.Neuro-psychopharmacological perspective of Orphan receptors of Rhodopsin (class A) family of G protein-coupled receptors.Sustained endocrine profiles of a girl with WAGR syndrome LGR4 modulates breast cancer initiation, metastasis, and cancer stem cells.Leucine-rich repeat-containing G protein-coupled receptor 4 (Lgr4) is necessary for prostate cancer metastasis via epithelial-mesenchymal transition.The RNA-binding protein, ZC3H14, is required for proper poly(A) tail length control, expression of synaptic proteins, and brain function in mice.

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LGR4/GPR48 inactivation leads to aniridia-genitourinary anomalies-mental retardation syndrome defects.

http://www.wikidata.org/entity/Q37691705

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 February 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

LGR4/GPR48 inactivation leads ...... retardation syndrome defects.

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LGR4/GPR48 inactivation leads ...... retardation syndrome defects.

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type

label

LGR4/GPR48 inactivation leads ...... retardation syndrome defects.

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LGR4/GPR48 inactivation leads ...... retardation syndrome defects.

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prefLabel

LGR4/GPR48 inactivation leads ...... retardation syndrome defects.

@en

LGR4/GPR48 inactivation leads ...... retardation syndrome defects.

@nl

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JBC.M113.530816

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Journal of Biological Chemistry

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LGR4/GPR48 inactivation leads ...... l retardation syndrome defects

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Dali Li

http://www.w3.org/2001/XMLSchema#string

Jinsheng Weng

http://www.w3.org/2001/XMLSchema#string

Tingfang Yi

http://www.w3.org/2001/XMLSchema#string

P2860

R-Spondin potentiates Wnt/β-catenin signaling through orphan receptors LGR4 and LGR5

Requirement of Lim1 for female reproductive tract development

R-spondins function as ligands of the orphan receptors LGR4 and LGR5 to regulate Wnt/beta-catenin signaling

JMJD2A is a novel N-CoR-interacting protein and is involved in repression of the human transcription factor achaete scute-like homologue 2 (ASCL2/Hash2)

Lgr5 homologues associate with Wnt receptors and mediate R-spondin signalling

Reversal of histone lysine trimethylation by the JMJD2 family of histone demethylases

Brain-derived neurotrophic factor and obesity in the WAGR syndrome

Reversal of learning deficits in a Tsc2+/- mouse model of tuberous sclerosis

Specificity and mechanism of JMJD2A, a trimethyllysine-specific histone demethylase

Chromosomal localization of GPR48, a novel glycoprotein hormone receptor like GPCR, in human and mouse with radiation hybrid and interspecific backcross mapping

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8767-8780

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10.1074/JBC.M113.530816

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