Reciprocal leukemia-stroma VCAM-1/VLA-4-dependent activation of NF-κB mediates chemoresistance. - Wikidata - awgldk - TriplyDB

Reciprocal leukemia-stroma VCAM-1/VLA-4-dependent activation of NF-κB mediates chemoresistance.

Reciprocal leukemia-stroma VCAM-1/VLA-4-dependent activation of NF-κB mediates chemoresistance.

http://www.wikidata.org/entity/Q37723478

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A hostel for the hostile: the bone marrow niche in hematologic neoplasms Role of Microenvironment in Resistance to Therapy in AML Mesenchymal Stromal Cells Can Regulate the Immune Response in the Tumor Microenvironment Stromal CYR61 Confers Resistance to Mitoxantrone via Spleen Tyrosine Kinase Activation in Human Acute Myeloid Leukaemia PRL-3 engages the focal adhesion pathway in triple-negative breast cancer cells to alter actin structure and substrate adhesion properties critical for cell migration and invasion Small molecule inhibitors in acute myeloid leukemia: from the bench to the clinic Tetraspanin Family Member, CD82, Regulates Expression of EZH2 via Inactivation of p38 MAPK Signaling in Leukemia Cells.Mesenchymal stromal cells derived from acute myeloid leukemia bone marrow exhibit aberrant cytogenetics and cytokine elaboration.Residual Disease in a Novel Xenograft Model of RUNX1-Mutated, Cytogenetically Normal Acute Myeloid Leukemia Normal vs. Malignant hematopoiesis: the complexity of acute leukemia through systems biology.The CCL2/CCR2 Axis Affects Transmigration and Proliferation but Not Resistance to Chemotherapy of Acute Myeloid Leukemia Cells.Serum Exosome MicroRNA as a Minimally-Invasive Early Biomarker of AML.Alterations in acute myeloid leukaemia bone marrow stromal cell exosome content coincide with gains in tyrosine kinase inhibitor resistance.Saikosaponin A inhibits influenza A virus replication and lung immunopathology.Coordinate regulation of residual bone marrow function by paracrine trafficking of AML exosomes.Bone marrow niche-mediated survival of leukemia stem cells in acute myeloid leukemia: Yin and Yang.Anti-apoptotic ARC protein confers chemoresistance by controlling leukemia-microenvironment interactions through a NFκB/IL1β signaling network.Loss of the oncogenic phosphatase PRL-3 promotes a TNF-R1 feedback loop that mediates triple-negative breast cancer growth.Rationale and efficacy of proteasome inhibitor combined with arsenic trioxide in the treatment of acute promyelocytic leukemia.Nucleophosmin Mutations Induce Chemosensitivity in THP-1 Leukemia Cells by Suppressing NF-κB Activity and Regulating Bax/Bcl-2 Expression.Pro-inflammatory-Related Loss of CXCL12 Niche Promotes Acute Lymphoblastic Leukemic Progression at the Expense of Normal Lymphopoiesis.De novo AML exhibits greater microenvironment dysregulation compared to AML with myelodysplasia-related changes.Understanding of leukemic stem cells and their clinical implications.Targeting the CXCL12/CXCR4 axis in acute myeloid leukemia: from bench to bedside.Cordycepin disrupts leukemia association with mesenchymal stromal cells and eliminates leukemia stem cell activity.Inhibiting glutaminase in acute myeloid leukemia: metabolic dependency of selected AML subtypes.Role of integrin alpha4 in drug resistance of leukemia Caveats of mesenchymal stem cell therapy in solid organ transplantation.Anticancer activity of cryptotanshinone on acute lymphoblastic leukemia cells.Contribution of very late antigen-4 (VLA-4) integrin to cancer progression and metastasis.Normal and leukemic stem cell niches: insights and therapeutic opportunities.Reciprocal Interactions of Leukemic Cells with Bone Marrow Stromal Cells Promote Enrichment of Leukemic Stem Cell Compartments in Response to Curcumin and Daunorubicin Therapeutically targeting SELF-reinforcing leukemic niches in acute myeloid leukemia: A worthy endeavor?CD98-Mediated Adhesive Signaling Enables the Establishment and Propagation of Acute Myelogenous Leukemia.Tunneling nanotube (TNT) formation is downregulated by cytarabine and NF-κB inhibition in acute myeloid leukemia (AML).ASK1/JNK-mediated TAp63 activation controls the cell survival signal of baicalein-treated EBV-transformed B cells.Epigenetic drug combination overcomes osteoblast-induced chemoprotection in pediatric acute lymphoid leukemia.Myeloid malignancies and the microenvironment.Hedgehog inhibitors selectively target cell migration and adhesion of mantle cell lymphoma in bone marrow microenvironment.Mesenchymal stromal cells (MSC) from JAK2+ myeloproliferative neoplasms differ from normal MSC and contribute to the maintenance of neoplastic hematopoiesis.

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Reciprocal leukemia-stroma VCAM-1/VLA-4-dependent activation of NF-κB mediates chemoresistance.

http://www.wikidata.org/entity/Q37723478

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 05 March 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Reciprocal leukemia-stroma VCA ...... F-κB mediates chemoresistance.

@en

Reciprocal leukemia-stroma VCA ...... F-κB mediates chemoresistance.

@nl

type

label

Reciprocal leukemia-stroma VCA ...... F-κB mediates chemoresistance.

@en

Reciprocal leukemia-stroma VCA ...... F-κB mediates chemoresistance.

@nl

prefLabel

Reciprocal leukemia-stroma VCA ...... F-κB mediates chemoresistance.

@en

Reciprocal leukemia-stroma VCA ...... F-κB mediates chemoresistance.

@nl

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BLOOD-2013-06-511527

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Reciprocal leukemia-stroma VCA ...... F-κB mediates chemoresistance.

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Carlos E Bueso-Ramos

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Elizabeth J Shpall

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Erika L Spaeth

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Marina Konopleva

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Martin H Nguyen

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Michael Andreeff

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Min Zhang

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Peter Ruvolo

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Po Yee Mak

http://www.w3.org/2001/XMLSchema#string

R Eric Davis

http://www.w3.org/2001/XMLSchema#string

P2860

Origins of the tumor microenvironment: quantitative assessment of adipose-derived and bone marrow-derived stroma

Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

Human extramedullary bone marrow in mice: a novel in vivo model of genetically controlled hematopoietic microenvironment

Mesenchymal stem cell transition to tumor-associated fibroblasts contributes to fibrovascular network expansion and tumor progression

I-kappa-kinase-2 (IKK-2) inhibition potentiates vincristine cytotoxicity in non-Hodgkin's lymphoma.

Bone marrow-derived myofibroblasts contribute to the mesenchymal stem cell niche and promote tumor growth.

Bortezomib with chemotherapy is highly active in advanced B-precursor acute lymphoblastic leukemia: Therapeutic Advances in Childhood Leukemia & Lymphoma (TACL) Study.

Platelet-derived growth factor (PDGF)-PDGF receptor interaction activates bone marrow-derived mesenchymal stromal cells derived from chronic lymphocytic leukemia: implications for an angiogenic switch.

Signaling pathways activated by daunorubicin.

Triterpenoid CDDO-Me blocks the NF-kappaB pathway by direct inhibition of IKKbeta on Cys-179.

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2691-2702

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scholarly article

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10.1182/BLOOD-2013-06-511527

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Katharina Schallmoser

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2014-03-05T00:00:00Z

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260558477

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24599548

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