Ca handling during excitation-contraction coupling in heart failure.
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Hyperphosphorylation of RyRs underlies triggered activity in transgenic rabbit model of LQT2 syndrome.The angiotensin receptor-associated protein Atrap is a stimulator of the cardiac Ca2+-ATPase SERCA2a.Cytosolic Ca²⁺ buffering determines the intra-SR Ca²⁺ concentration at which cardiac Ca²⁺ sparks terminateStudying dyadic structure-function relationships: a review of current modeling approaches and new insights into Ca2+ (mis)handling.Functional Impact of Ryanodine Receptor Oxidation on Intracellular Calcium Regulation in the Heart.Post-extrasystolic Potentiation: Link between Ca(2+) Homeostasis and Heart Failure?Calcium-dependent Nedd4-2 upregulation mediates degradation of the cardiac sodium channel Nav1.5: implications for heart failure.SK channel enhancers attenuate Ca2+-dependent arrhythmia in hypertrophic hearts by regulating mito-ROS-dependent oxidation and activity of RyR.A multidimensional sight on cardiac failure: uncovered from structural to molecular level.The cardiac calsequestrin gene transcription is modulated at the promoter by NFAT and MEF-2 transcription factors.Redox-sensitive residue in the actin-binding interface of myosinThe effect of PKA-mediated phosphorylation of ryanodine receptor on SR Ca2+ leak in ventricular myocytes.Gene therapy for heart failure: time to go back to the drawing board.HAX-1 regulates SERCA2a oxidation and degradation.Sensitivity analysis revealing the effect of modulating ionic mechanisms on calcium dynamics in simulated human heart failure.The antiapoptotic protein HAX-1 mediates half of phospholamban's inhibitory activity on calcium cycling and contractility in the heart.The contractile adaption to preload depends on the amount of afterload.The role of spatial organization of Ca2+ release sites in the generation of arrhythmogenic diastolic Ca2+ release in myocytes from failing hearts.New Insights in Cardiac Calcium Handling and Excitation-Contraction Coupling.Chronic low-intensity exercise attenuates cardiomyocyte contractile dysfunction and impaired adrenergic responsiveness in aortic-banded mini-swine.Sodium nitrate supplementation alters mitochondrial H2O2 emission but does not improve mitochondrial oxidative metabolism in the heart of healthy rats.The role of RyR2 oxidation in the blunted frequency-dependent facilitation of Ca2+ transient amplitude in rabbit failing myocytes.Sub-cellular Electrical Heterogeneity Revealed by Loose Patch Recording Reflects Differential Localization of Sarcolemmal Ion Channels in Intact Rat Hearts.Proarrhythmic Remodeling of Calcium Homeostasis in Cardiac Disease; Implications for Diabetes and Obesity
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Ca handling during excitation-contraction coupling in heart failure.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 11 February 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Ca handling during excitation-contraction coupling in heart failure.
@en
Ca handling during excitation-contraction coupling in heart failure.
@nl
type
label
Ca handling during excitation-contraction coupling in heart failure.
@en
Ca handling during excitation-contraction coupling in heart failure.
@nl
prefLabel
Ca handling during excitation-contraction coupling in heart failure.
@en
Ca handling during excitation-contraction coupling in heart failure.
@nl
P2093
P2860
P1433
P1476
Ca handling during excitation-contraction coupling in heart failure.
@en
P2093
Aleksey V Zima
Dmitry Terentyev
Elisa Bovo
Jennifer A Rochira
Stefan R Mazurek
P2860
P2888
P304
P356
10.1007/S00424-014-1469-3
P577
2014-02-11T00:00:00Z