Sildenafil and FDP-Sr attenuate diabetic cardiomyopathy by suppressing abnormal expression of myocardial CASQ2, FKBP12.6, and SERCA2a in rats.
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Response to pulmonary arterial hypertension drug therapies in patients with pulmonary arterial hypertension and cardiovascular risk factorsHyperglycemia-induced protein kinase C β2 activation induces diastolic cardiac dysfunction in diabetic rats by impairing caveolin-3 expression and Akt/eNOS signaling.Depressed calcium-handling proteins due to endoplasmic reticulum stress and apoptosis in the diabetic heart are attenuated by argirein.
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Sildenafil and FDP-Sr attenuate diabetic cardiomyopathy by suppressing abnormal expression of myocardial CASQ2, FKBP12.6, and SERCA2a in rats.
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scientific article published on 28 March 2011
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vetenskaplig artikel
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Sildenafil and FDP-Sr attenuat ...... FKBP12.6, and SERCA2a in rats.
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Sildenafil and FDP-Sr attenuat ...... FKBP12.6, and SERCA2a in rats.
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Sildenafil and FDP-Sr attenuat ...... FKBP12.6, and SERCA2a in rats.
@en
Sildenafil and FDP-Sr attenuat ...... FKBP12.6, and SERCA2a in rats.
@nl
prefLabel
Sildenafil and FDP-Sr attenuat ...... FKBP12.6, and SERCA2a in rats.
@en
Sildenafil and FDP-Sr attenuat ...... FKBP12.6, and SERCA2a in rats.
@nl
P2093
P2860
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P1476
Sildenafil and FDP-Sr attenuat ...... FKBP12.6, and SERCA2a in rats.
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P2093
P2860
P2888
P304
P356
10.1038/APS.2010.226
P577
2011-03-28T00:00:00Z