Diabetes increases formation of advanced glycation end products on Sarco(endo)plasmic reticulum Ca2+-ATPase.
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How exercise may amend metabolic disturbances in diabetic cardiomyopathyInterplay of oxidative, nitrosative/nitrative stress, inflammation, cell death and autophagy in diabetic cardiomyopathyAdvanced glycation end products and diabetic complicationsDiabetic cardiomyopathy: signaling defects and therapeutic approachesCarbonylation contributes to SERCA2a activity loss and diastolic dysfunction in a rat model of type 1 diabetesCombined insulin treatment and intense exercise training improved basal cardiac function and Ca(2+)-cycling proteins expression in type 1 diabetic ratsAdvanced Glycation End Product (AGE)-AGE Receptor (RAGE) System Upregulated Connexin43 Expression in Rat Cardiomyocytes via PKC and Erk MAPK PathwaysDiabetic cardiomyopathy: pathophysiology and clinical featuresAdvanced glycation endproduct (AGE) accumulation and AGE receptor (RAGE) up-regulation contribute to the onset of diabetic cardiomyopathy.Metabolic and Biochemical Stressors in Diabetic Cardiomyopathy.Role of ER stress in ventricular contractile dysfunction in type 2 diabetesCarbonylation of myosin heavy chains in rat heart during diabetes.Analysis and prediction of mammalian protein glycation.Pyridoxamine protects against mechanical defects in cardiac ageing in rats: studies on load dependence of myocardial relaxation.[Diabetic cardiomyopathy].Causes and characteristics of diabetic cardiomyopathy.Cannabinoid 1 receptor promotes cardiac dysfunction, oxidative stress, inflammation, and fibrosis in diabetic cardiomyopathyRole of advanced glycation end products in cardiovascular disease.Ginkgo biloba extract EGB761 protects against aging-associated diastolic dysfunction in cardiomyocytes of D-galactose-induced aging rat.Aminoguanidine prevents fructose-induced deterioration in left ventricular-arterial coupling in Wistar rats.Advanced glycation end product cross-link breaker attenuates diabetes-induced cardiac dysfunction by improving sarcoplasmic reticulum calcium handling.The discovery of glycated hemoglobin: a major event in the study of nonenzymatic chemistry in biological systems.Cardiac fibrosis and dysfunction in experimental diabetic cardiomyopathy are ameliorated by alpha-lipoic acid.Aminoguanidine prevents the impairment of cardiac pumping mechanics in rats with streptozotocin and nicotinamide-induced type 2 diabetesExercise training initiated after the onset of diabetes preserves myocardial function: effects on expression of beta-adrenoceptorsProteomic analysis in diabetic cardiomyopathy using bioinformatics approach.Molecular mechanisms of diabetic cardiomyopathy.Sildenafil and FDP-Sr attenuate diabetic cardiomyopathy by suppressing abnormal expression of myocardial CASQ2, FKBP12.6, and SERCA2a in rats.Diabetic Cardiomyopathy: An Immunometabolic Perspective.Endoplasmic-reticulum calcium depletion and disease.Advanced glycation end products and diabetic retinopathy.Advanced glycation end products: role in pathology of diabetic cardiomyopathy.Impairment of calcium ATPases by high glucose and potential pharmacological protection.Non-enzymatic glycation and glycoxidation protein products in foods and diseases: an interconnected, complex scenario fully open to innovative proteomic studies.The role of post-translational modifications in acute and chronic cardiovascular disease.The regulation of sarco(endo)plasmic reticulum calcium-ATPases (SERCA).Damaging effects of hyperglycemia on cardiovascular function: spotlight on glucose metabolic pathways.Pathophysiological Fundamentals of Diabetic Cardiomyopathy.Cardiomyocyte Ca2+ homeostasis as a therapeutic target in heart failure with reduced and preserved ejection fraction.Advanced glycation end products (AGEs) and cardiovascular dysfunction: focus on high molecular weight AGEs.
P2860
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P2860
Diabetes increases formation of advanced glycation end products on Sarco(endo)plasmic reticulum Ca2+-ATPase.
description
2004 nî lūn-bûn
@nan
2004 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Diabetes increases formation o ...... plasmic reticulum Ca2+-ATPase.
@ast
Diabetes increases formation o ...... plasmic reticulum Ca2+-ATPase.
@en
type
label
Diabetes increases formation o ...... plasmic reticulum Ca2+-ATPase.
@ast
Diabetes increases formation o ...... plasmic reticulum Ca2+-ATPase.
@en
prefLabel
Diabetes increases formation o ...... plasmic reticulum Ca2+-ATPase.
@ast
Diabetes increases formation o ...... plasmic reticulum Ca2+-ATPase.
@en
P2093
P1433
P1476
Diabetes increases formation o ...... plasmic reticulum Ca2+-ATPase.
@en
P2093
Chun Hong Shao
Henry R Besch
Kaushik P Patel
Keshore R Bidasee
U Deniz Dincer
Yinong Zhang
P304
P356
10.2337/DIABETES.53.2.463
P407
P577
2004-02-01T00:00:00Z