Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects.
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The role of titin and extracellular matrix remodelling in heart failure with preserved ejection fractionAlways cleave up your mess: targeting collagen degradation to treat tissue fibrosisTargeting myocardial remodelling to develop novel therapies for heart failure: a position paper from the Working Group on Myocardial Function of the European Society of CardiologyTorsemide Versus Furosemide in Patients With Acute Heart Failure (from the ASCEND-HF Trial)Hepatic stellate cells and portal fibroblasts are the major cellular sources of collagens and lysyl oxidases in normal liver and early after injuryCellular mechanisms of tissue fibrosis. 6. Purinergic signaling and response in fibroblasts and tissue fibrosisDownregulation of Lysyl Oxidase Protects Retinal Endothelial Cells From High Glucose-Induced ApoptosisCopper sulfate affects Nile tilapia (Oreochromis niloticus) cardiomyocytes structure and contractile function.N-acetyl-seryl-aspartyl-lysyl-proline reduces cardiac collagen cross-linking and inflammation in angiotensin II-induced hypertensive ratsmicroRNA-122 regulates hypoxia-inducible factor-1 and vimentin in hepatocytes and correlates with fibrosis in diet-induced steatohepatitis.Transforming growth factor-beta induces extracellular matrix protein cross-linking lysyl oxidase (LOX) genes in human trabecular meshwork cells.The copper chelator tetrathiomolybdate regressed bleomycin-induced pulmonary fibrosis in mice, by reducing lysyl oxidase expressions.Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature.Building a better infarct: Modulation of collagen cross-linking to increase infarct stiffness and reduce left ventricular dilation post-myocardial infarction.Systemic sclerosis disease modification clinical trials design: quo vadis?Integrated glycoprotein immobilization method for glycopeptide and glycan analysis of cardiac hypertrophy.Action of SNAIL1 in Cardiac Myofibroblasts Is Important for Cardiac Fibrosis following Hypoxic InjuryExcess Linoleic Acid Increases Collagen I/III Ratio and "Stiffens" the Heart Muscle Following High Fat Diets.Biomechanics of Cardiac FunctionThe Living Scar--Cardiac Fibroblasts and the Injured Heart.Matrix metalloproteinase-28 deletion exacerbates cardiac dysfunction and rupture after myocardial infarction in mice by inhibiting M2 macrophage activation.Tissue mechanics and fibrosis.Left and right ventricle late remodeling following myocardial infarction in rats.LOXL4 is induced by transforming growth factor β1 through Smad and JunB/Fra2 and contributes to vascular matrix remodeling.Aging and the cardiac collagen matrix: Novel mediators of fibrotic remodellingCardioprotective effects of lysyl oxidase inhibition against volume overload-induced extracellular matrix remodeling.Targeted inhibition of Focal Adhesion Kinase Attenuates Cardiac Fibrosis and Preserves Heart Function in Adverse Cardiac Remodeling.The myofibroblast matrix: implications for tissue repair and fibrosis.TGF-β1 Pretreatment Improves the Function of Mesenchymal Stem Cells in the Wound Bed.Thrombospondin-4, tumour necrosis factor-like weak inducer of apoptosis (TWEAK) and its receptor Fn14: novel extracellular matrix modulating factors in cardiac remodelling.Molecular pathogenesis of myocardial remodeling and new potential therapeutic targets in chronic heart failure.Translating an understanding of the pathogenesis of hepatic fibrosis to novel therapiesLeft ventricular remodeling: one small step for the extracellular matrix will translate to a giant leap for the myocardium.Effects of 6-months' Exercise on Cardiac Function, Structure and Metabolism in Female Hypertensive Rats-The Decisive Role of Lysyl Oxidase and Collagen III.Crohn's Strictures-Moving Away from the KnifeIn-Vivo Assessment of Regional Mechanics Post-Myocardial Infarction: A Focus on the Road Ahead.Discovery and characterization of a photo-oxidative histidine-histidine cross-link in IgG1 antibody utilizing ¹⁸O-labeling and mass spectrometry.Transforming growth factor β1 signaling coincides with epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis in mice.Apocynin influence on oxidative stress and cardiac remodeling of spontaneously hypertensive rats with diabetes mellitus.Global fibroblast activation throughout the left ventricle but localized fibrosis after myocardial infarction.
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Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects.
description
article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 14 May 2010
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vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects.
@en
Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects.
@nl
type
label
Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects.
@en
Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects.
@nl
prefLabel
Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects.
@en
Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects.
@nl
P2093
P1476
Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects.
@en
P2093
Arantxa González
Eduardo de Teresa
Félix Valencia
Javier Díez
Nerea Hermida
P356
10.1152/AJPHEART.00335.2010
P50
P577
2010-05-14T00:00:00Z