Opa proteins and CEACAMs: pathways of immune engagement for pathogenic Neisseria.
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Intruders below the radar: molecular pathogenesis of Bartonella sppHow the Knowledge of Interactions between Meningococcus and the Human Immune System Has Been Used to Prepare Effective Neisseria meningitidis VaccinesThe role of variable DNA tandem repeats in bacterial adaptationRho GTPases as pathogen targets: Focus on curable sexually transmitted infectionsStructure of the Neisserial outer membrane protein Opa₆₀: loop flexibility essential to receptor recognition and bacterial engulfment.Host-pathogen interactions in bacterial meningitisCEACAM1 negatively regulates IL-1β production in LPS activated neutrophils by recruiting SHP-1 to a SYK-TLR4-CEACAM1 complexGlobal analysis of neutrophil responses to Neisseria gonorrhoeae reveals a self-propagating inflammatory programStreptococcus pneumoniae Cell-Wall-Localized Phosphoenolpyruvate Protein Phosphotransferase Can Function as an Adhesin: Identification of Its Host Target Molecules and Evaluation of Its Potential as a VaccineExperimental Gonococcal Infection in Male Volunteers: Cumulative Experience with Neisseria gonorrhoeae Strains FA1090 and MS11mkCNeisserial Opa Protein-CEACAM Interactions: Competition for Receptors as a Means of Bacterial Invasion and Pathogenesis.An improved method for differentiating cell-bound from internalized particles by imaging flow cytometry.Opa+ Neisseria gonorrhoeae exhibits reduced survival in human neutrophils via Src family kinase-mediated bacterial trafficking into mature phagolysosomesNeisseria gonorrhoeae elicits extracellular traps in primary neutrophil culture while suppressing the oxidative burst.Assembly of NADPH oxidase in human neutrophils is modulated by the opacity-associated protein expression State of Neisseria gonorrhoeaeConstitutively Opa-expressing and Opa-deficient neisseria gonorrhoeae strains differentially stimulate and survive exposure to human neutrophilsA bacterial siren song: intimate interactions between Neisseria and neutrophils.Resistance of Neisseria gonorrhoeae to neutrophils.Neisseria gonorrhoeae infects the human endocervix by activating non-muscle myosin II-mediated epithelial exfoliation.Vaccines against gonorrhea: current status and future challenges.Seminal plasma initiates a Neisseria gonorrhoeae transmission stateAssociation of Neisseria gonorrhoeae Opa(CEA) with dendritic cells suppresses their ability to elicit an HIV-1-specific T cell memory responsePhase variation leads to the misidentification of a Neisseria gonorrhoeae virulence geneThe Pathobiology of Neisseria gonorrhoeae Lower Female Genital Tract Infection.Estradiol-Treated Female Mice as Surrogate Hosts for Neisseria gonorrhoeae Genital Tract Infections.Expression of Opacity Proteins Interferes with the Transmigration of Neisseria gonorrhoeae across Polarized Epithelial CellsCharacterisation of the Immunomodulatory Effects of Meningococcal Opa Proteins on Human Peripheral Blood Mononuclear Cells and CD4+ T Cells.Secretory leukocyte protease inhibitor binds to Neisseria gonorrhoeae outer membrane opacity protein and is bactericidalConstruction and characterization of a derivative of Neisseria gonorrhoeae strain MS11 devoid of all opa genes.Fur-mediated global regulatory circuits in pathogenic Neisseria species.Antigenic Variation in Bacterial Pathogens.Is gonococcal disease preventable? The importance of understanding immunity and pathogenesis in vaccine development.Neisseria meningitidis NalP cleaves human complement C3, facilitating degradation of C3b and survival in human serum.Neisseria gonorrhoeae survives within and modulates apoptosis and inflammatory cytokine production of human macrophagesThe biology of Neisseria adhesins.Vaccine research for gonococcal infections: where are we?Bacterial receptors for host transferrin and lactoferrin: molecular mechanisms and role in host-microbe interactions.The Long Road to an Effective Vaccine for Meningococcus Group B (MenB).Roles of CEACAM1 in cell communication and signaling of lung cancer and other diseases.Both MisR (CpxR) and MisS (CpxA) Are Required for Neisseria gonorrhoeae Infection in a Murine Model of Lower Genital Tract Infection.
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P2860
Opa proteins and CEACAMs: pathways of immune engagement for pathogenic Neisseria.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 17 January 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Opa proteins and CEACAMs: pathways of immune engagement for pathogenic Neisseria.
@en
Opa proteins and CEACAMs: pathways of immune engagement for pathogenic Neisseria.
@nl
type
label
Opa proteins and CEACAMs: pathways of immune engagement for pathogenic Neisseria.
@en
Opa proteins and CEACAMs: pathways of immune engagement for pathogenic Neisseria.
@nl
prefLabel
Opa proteins and CEACAMs: pathways of immune engagement for pathogenic Neisseria.
@en
Opa proteins and CEACAMs: pathways of immune engagement for pathogenic Neisseria.
@nl
P2860
P1476
Opa proteins and CEACAMs: pathways of immune engagement for pathogenic Neisseria.
@en
P2093
Andrew J Pollard
P2860
P304
P356
10.1111/J.1574-6976.2010.00260.X
P577
2011-01-17T00:00:00Z