The origin of interstitial myofibroblasts in chronic kidney disease. - Wikidata - awgldk - TriplyDB

The origin of interstitial myofibroblasts in chronic kidney disease.

The origin of interstitial myofibroblasts in chronic kidney disease.

http://www.wikidata.org/entity/Q37839779

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Epithelial-to-Mesenchymal Transition in Diabetic Nephropathy: Fact or Fiction?The FOXD1 lineage of kidney perivascular cells and myofibroblasts: functions and responses to injury Fibrosis Related Inflammatory Mediators: Role of the IL-10 Cytokine Family Host responses in tissue repair and fibrosis Stem cells: potential and challenges for kidney repair Regulation of autophagy by TGF-β: emerging role in kidney fibrosis Experimental systems to study the origin of the myofibroblast in peritoneal fibrosis Role of the TGF-β/BMP-7/Smad pathways in renal diseases Vitamin D deficiency aggravates chronic kidney disease progression after ischemic acute kidney injury The proximal tubule is the primary target of injury and progression of kidney disease: role of the glomerulotubular junction.Inflammatory macrophages can transdifferentiate into myofibroblasts during renal fibrosis Ligand-independent transforming growth factor-β type I receptor signalling mediates type I collagen-induced epithelial-mesenchymal transition.Silencing SMOC2 ameliorates kidney fibrosis by inhibiting fibroblast to myofibroblast transformation Matrix metalloproteinase 9-dependent Notch signaling contributes to kidney fibrosis through peritubular endothelial-mesenchymal transition Cellular and molecular mechanisms in kidney fibrosis.Bone morphogenetic proteins and their antagonists: current and emerging clinical uses.Molecular mechanisms and treatment of radiation-induced lung fibrosis Transgenic expression of nonclassically secreted FGF suppresses kidney repair.Role of moesin in renal fibrosis.Autophagy regulates TGF-β expression and suppresses kidney fibrosis induced by unilateral ureteral obstruction.Iron chelation by deferoxamine prevents renal interstitial fibrosis in mice with unilateral ureteral obstruction Urinary candidate biomarker discovery in a rat unilateral ureteral obstruction model.Deficient Autophagy Results in Mitochondrial Dysfunction and FSGS.Microvesicles derived from human umbilical cord mesenchymal stem cells facilitate tubular epithelial cell dedifferentiation and growth via hepatocyte growth factor induction.Identification of human epididymis protein-4 as a fibroblast-derived mediator of fibrosis Impact of AMP-Activated Protein Kinase α1 Deficiency on Tissue Injury following Unilateral Ureteral Obstruction.ERK/GSK3β/Snail signaling mediates radiation-induced alveolar epithelial-to-mesenchymal transition.Cells of renin lineage express hypoxia inducible factor 2α following experimental ureteral obstruction.Cellular and molecular mechanisms of renal fibrosis.Lefty-1 alleviates TGF-β1-induced fibroblast-myofibroblast transdifferentiation in NRK-49F cells Matrix metalloproteinase 9 induces endothelial-mesenchymal transition via Notch activation in human kidney glomerular endothelial cells Cdc42-interacting protein-4 promotes TGF-Β1-induced epithelial-mesenchymal transition and extracellular matrix deposition in renal proximal tubular epithelial cells.Vitamin D Attenuates Kidney Fibrosis via Reducing Fibroblast Expansion, Inflammation, and Epithelial Cell Apoptosis Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis Cellular mechanisms of tissue fibrosis. 3. Novel mechanisms of kidney fibrosis.Kidney tubular β-catenin signaling controls interstitial fibroblast fate via epithelial-mesenchymal communication.Targeting pericyte differentiation as a strategy to modulate kidney fibrosis in diabetic nephropathy.TGF-β/Smad3 signalling regulates the transition of bone marrow-derived macrophages into myofibroblasts during tissue fibrosis Human Wharton's jelly-derived mesenchymal stromal cells reduce renal fibrosis through induction of native and foreign hepatocyte growth factor synthesis in injured tubular epithelial cells.Wnt4/β-catenin signaling in medullary kidney myofibroblasts

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The origin of interstitial myofibroblasts in chronic kidney disease.

http://www.wikidata.org/entity/Q37839779

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 February 2011

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

The origin of interstitial myofibroblasts in chronic kidney disease.

@en

The origin of interstitial myofibroblasts in chronic kidney disease.

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type

label

The origin of interstitial myofibroblasts in chronic kidney disease.

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The origin of interstitial myofibroblasts in chronic kidney disease.

@nl

prefLabel

The origin of interstitial myofibroblasts in chronic kidney disease.

@en

The origin of interstitial myofibroblasts in chronic kidney disease.

@nl

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Pediatric Nephrology

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The origin of interstitial myofibroblasts in chronic kidney disease.

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Benjamin D Humphreys

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Ivica Grgic

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Jeremy S Duffield

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P2860

Epithelial-mesenchymal transition and its implications for fibrosis

Cellular and molecular mechanisms of fibrosis

The basics of epithelial-mesenchymal transition

The myofibroblast: one function, multiple origins

Epithelial-mesenchymal transitions in development and disease

A perivascular origin for mesenchymal stem cells in multiple human organs

Bone morphogenic protein-7 inhibits progression of chronic renal fibrosis associated with two genetic mouse models

Bone marrow cells regenerate infarcted myocardium

Myofibroblasts and mechano-regulation of connective tissue remodelling

Epithelial-mesenchymal transition: at the crossroads of development and tumor metastasis

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s00467-011-1772-6

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10.1007/S00467-011-1772-6

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