Chlamydia pneumoniae-induced foam cell formation requires MyD88-dependent and -independent signaling and is reciprocally modulated by liver X receptor activation.
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Role of gut microbiota in the modulation of atherosclerosis-associated immune responseInnate immune responses to Chlamydia pneumoniae infection: role of TLRs, NLRs, and the inflammasomeAn innate immunity signaling process suppresses macrophage ABCA1 expression through IRAK-1-mediated downregulation of retinoic acid receptor alpha and NFATc2The NOD/RIP2 pathway is essential for host defenses against Chlamydophila pneumoniae lung infection.Manipulation of Host Cholesterol by Obligate Intracellular Bacteria.Activation of macrophages by P2X7-induced microvesicles from myeloid cells is mediated by phospholipids and is partially dependent on TLR4.CD4 T Cells Mediate Both Positive and Negative Regulation of the Immune Response to HIV Infection: Complex Role of T Follicular Helper Cells and Regulatory T Cells in PathogenesisAnalyses of the pathways involved in early- and late-phase induction of IFN-beta during C. muridarum infection of oviduct epithelial cellsInfection and Atherosclerosis DevelopmentTLR4 rs1927911, but not TLR2 rs5743708, is associated with atherosclerotic cerebral infarction in the Southern Han population: a case-control study.Toll-like receptors and human disease: lessons from single nucleotide polymorphisms.Role of liver X receptors in cholesterol efflux and inflammatory signaling (review)Transcription factor complex AP-1 mediates inflammation initiated by Chlamydia pneumoniae infection.The Impact of Serum Amyloid P-Component on Gene Expression in RAW264.7 Mouse MacrophagesMacrophage-activating lipopeptide-2 downregulates the expression of ATP-binding cassette transporter A1 by activating the TLR2/NF-кB/ZNF202 pathway in THP-1 macrophages.The expression and functions of toll-like receptors in atherosclerosis.Effects of gut microbiota on obesity and atherosclerosis via modulation of inflammation and lipid metabolism.IL-17A is proatherogenic in high-fat diet-induced and Chlamydia pneumoniae infection-accelerated atherosclerosis in mice.Toll-like receptor signaling: common pathways that drive cardiovascular disease and rheumatoid arthritis.Biological roles of liver X receptors in immune cells.Toll-like receptor signaling pathways in cardiovascular diseases: challenges and opportunities.Inflammation-induced foam cell formation in chronic inflammatory disease.LXRα regulates macrophage arginase 1 through PU.1 and interferon regulatory factor 8.Tannerella forsythia BspA increases the risk factors for atherosclerosis in ApoE(-/-) mice.Cross-talk between TLR4-MyD88-NF-κB and SCAP-SREBP2 pathways mediates macrophage foam cell formation.Coordinate stimulation of macrophages by microparticles and TLR ligands induces foam cell formationThe JAK2-Akt-glycogen synthase kinase-3β signaling pathway is involved in toll-like receptor 2-induced monocyte chemoattractant protein-1 regulation.A Toll-like receptor 9-mediated pathway stimulates perilipin 3 (TIP47) expression and induces lipid accumulation in macrophages.LincRNA DYN-LRB2-2 upregulates cholesterol efflux by decreasing TLR2 expression in macrophages.Role of MyD88-dependent and MyD88-independent signaling in Porphyromonas gingivalis-elicited macrophage foam cell formation.The activation of liver X receptors inhibits toll-like receptor-9-induced foam cell formation.DBZ blocks LPS-induced monocyte activation and foam cell formation via inhibiting nuclear factor-ĸB.Inhibition of TLR4 alleviates the inflammation and apoptosis of retinal ganglion cells in high glucose.Liver X Receptors Activation Attenuates Ischemia Reperfusion Injury of Liver Graft in Rats.Toll-like receptor 2 downregulates the cholesterol efflux by activating the nuclear factor-κB pathway in macrophages and may be a potential therapeutic target for the prevention of atherosclerosis.Microbial modulation of cardiovascular disease.Chlamydia and Lipids Engage a Common Signaling Pathway That Promotes Atherogenesis.A micro RNA-146a polymorphism is associated with coronary restenosis
P2860
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P2860
Chlamydia pneumoniae-induced foam cell formation requires MyD88-dependent and -independent signaling and is reciprocally modulated by liver X receptor activation.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Chlamydia pneumoniae-induced f ...... y liver X receptor activation.
@en
Chlamydia pneumoniae-induced f ...... y liver X receptor activation.
@nl
type
label
Chlamydia pneumoniae-induced f ...... y liver X receptor activation.
@en
Chlamydia pneumoniae-induced f ...... y liver X receptor activation.
@nl
prefLabel
Chlamydia pneumoniae-induced f ...... y liver X receptor activation.
@en
Chlamydia pneumoniae-induced f ...... y liver X receptor activation.
@nl
P2093
P2860
P1476
Chlamydia pneumoniae-induced f ...... y liver X receptor activation.
@en
P2093
Kenichi Shimada
Rosalinda Sorrentino
Shuang Chen
Terence M Doherty
Timothy R Crother
Yonca Bulut
P2860
P304
P356
10.4049/JIMMUNOL.181.10.7186
P407
P50
P577
2008-11-01T00:00:00Z