IFN-alpha beta-dependent, IFN-gamma secretion by bone marrow-derived macrophages controls an intracellular bacterial infection.
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Nitric Oxide Protects against Infection-Induced Neuroinflammation by Preserving the Stability of the Blood-Brain BarrierCritical and independent role for SOCS3 in either myeloid or T cells in resistance to Mycobacterium tuberculosisThe NOD/RIP2 pathway is essential for host defenses against Chlamydophila pneumoniae lung infection.Evolution to a chronic disease niche correlates with increased sensitivity to tryptophan availability for the obligate intracellular bacterium Chlamydia pneumoniaeInterleukin 10 overexpression alters survival in the setting of gram-negative pneumonia following lung contusionAge alterations in extent and severity of experimental intranasal infection with Chlamydophila pneumoniae in BALB/c miceInduction of type I and type III interferons by Borrelia burgdorferi correlates with pathogenesis and requires linear plasmid 36.Gene expression analysis of macrophages derived from ankylosing spondylitis patients reveals interferon-gamma dysregulationMacrophage control of phagocytosed mycobacteria is increased by factors secreted by alveolar epithelial cells through nitric oxide independent mechanismsToll-like receptors 2 and 4 regulate the frequency of IFNγ-producing CD4+ T-cells during pulmonary infection with Chlamydia pneumoniaeLPS regulates SOCS2 transcription in a type I interferon dependent autocrine-paracrine loop.Role of endogenous IFN-gamma in macrophage programming induced by IL-12 and IL-18.CD44-regulated intracellular proliferation of Listeria monocytogenesInfection of human monocytes by Chlamydia pneumoniae and Chlamydia trachomatis: an in vitro comparative studyAnalyses of the pathways involved in early- and late-phase induction of IFN-beta during C. muridarum infection of oviduct epithelial cellsInterferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infectionChlamydia muridarum infection of macrophages elicits bactericidal nitric oxide production via reactive oxygen species and cathepsin B.The role of type I interferons in non-viral infections.FAM19A4 is a novel cytokine ligand of formyl peptide receptor 1 (FPR1) and is able to promote the migration and phagocytosis of macrophages.TLR9 regulates Th1 responses and cooperates with TLR2 in mediating optimal resistance to Mycobacterium tuberculosis.Alveolar epithelial cells are critical in protection of the respiratory tract by secretion of factors able to modulate the activity of pulmonary macrophages and directly control bacterial growth.Type I interferon signaling exacerbates Chlamydia muridarum genital infection in a murine model.In situ IL-12/23p40 production during mycobacterial infection is sustained by CD11bhigh dendritic cells localized in tissue sites distinct from those harboring bacilli.The infecting dose of Chlamydia muridarum modulates the innate immune response and ascending infection.Exploiting induced pluripotent stem cell-derived macrophages to unravel host factors influencing Chlamydia trachomatis pathogenesisInduction of iNOS by Chlamydophila pneumoniae requires MyD88-dependent activation of JNK.Host cell responses to Chlamydia pneumoniae in gamma interferon-induced persistence overlap those of productive infection and are linked to genes involved in apoptosis, cell cycle, and metabolism.Inflammatory responses following Chlamydia pneumoniae infection of glial cells.Phosphoinositide 3-kinase in nitric oxide synthesis in macrophage: critical dimerization of inducible nitric-oxide synthase.Effect of nitric oxide on the growth of Chlamydophila pneumoniae.Silencing or permanent activation: host-cell responses in models of persistent Chlamydia pneumoniae infection.The Role of IFN-β during the Course of Sepsis Progression and Its Therapeutic Potential.Identification of interferon-γ as a new molecular target of liver X receptor.Replication or death: distinct fates of pathogenic Leptospira strain Lai within macrophages of human or mouse origin.Innate IFN-γ-Producing Cells Developing in the Absence of IL-2 Receptor Common γ-Chain.TLR2 but not TLR4 signalling is critically involved in the inhibition of IFN-gamma-induced killing of mycobacteria by murine macrophages.Impact of toll-like receptor 2 deficiency on immune responses to mycobacterial antigens.Macrophage polarization and MRSA infection in burned mice.Cooperative role of macrophages and neutrophils in host Antiprotozoan resistance in mice acutely infected with Cryptosporidium parvum.Nonhematopoietic cells control the outcome of infection with Listeria monocytogenes in a nucleotide oligomerization domain 1-dependent manner.
P2860
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P2860
IFN-alpha beta-dependent, IFN-gamma secretion by bone marrow-derived macrophages controls an intracellular bacterial infection.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2001
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
IFN-alpha beta-dependent, IFN- ...... acellular bacterial infection.
@en
IFN-alpha beta-dependent, IFN- ...... acellular bacterial infection.
@nl
type
label
IFN-alpha beta-dependent, IFN- ...... acellular bacterial infection.
@en
IFN-alpha beta-dependent, IFN- ...... acellular bacterial infection.
@nl
prefLabel
IFN-alpha beta-dependent, IFN- ...... acellular bacterial infection.
@en
IFN-alpha beta-dependent, IFN- ...... acellular bacterial infection.
@nl
P2093
P1476
IFN-alpha beta-dependent, IFN- ...... acellular bacterial infection.
@en
P2093
Gigliotti D
Palmblad K
Rothfuchs AG
Rottenberg ME
P304
P356
10.4049/JIMMUNOL.167.11.6453
P407
P577
2001-12-01T00:00:00Z