TGF-β1 → SMAD/p53/USF2 → PAI-1 transcriptional axis in ureteral obstruction-induced renal fibrosis. - Wikidata - awgldk - TriplyDB

TGF-β1 → SMAD/p53/USF2 → PAI-1 transcriptional axis in ureteral obstruction-induced renal fibrosis.

TGF-β1 → SMAD/p53/USF2 → PAI-1 transcriptional axis in ureteral obstruction-induced renal fibrosis.

http://www.wikidata.org/entity/Q37884486

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Key fibrogenic mediators: old players. Renin–angiotensin system TGF-β/Smad signaling in renal fibrosis TGF-β signaling in tissue fibrosis: redox controls, target genes and therapeutic opportunities The role of the p53 tumor suppressor in metabolism and diabetes Oxidative stress in obesity: a critical component in human diseases Redox control of p53 in the transcriptional regulation of TGF-β1 target genes through SMAD cooperativity.Antifibrotic effects of KS370G, a caffeamide derivative, in renal ischemia-reperfusion injured mice and renal tubular epithelial cells.Role of microRNA 1207-5P and its host gene, the long non-coding RNA Pvt1, as mediators of extracellular matrix accumulation in the kidney: implications for diabetic nephropathy Tumor suppressor ataxia telangiectasia mutated functions downstream of TGF-β1 in orchestrating profibrotic responses.Impact of AMP-Activated Protein Kinase α1 Deficiency on Tissue Injury following Unilateral Ureteral Obstruction.Loss of tumour suppressor PTEN expression in renal injury initiates SMAD3- and p53-dependent fibrotic responses Glycogen synthase kinase-3 inhibition attenuates fibroblast activation and development of fibrosis following renal ischemia-reperfusion in mice.Activation of AMP-activated protein kinase prevents TGF-β1-induced epithelial-mesenchymal transition and myofibroblast activation.Differentially expressed microRNAs in bone marrow mesenchymal stem cell-derived microvesicles in young and older rats and their effect on tumor growth factor-β1-mediated epithelial-mesenchymal transition in HK2 cells.Comprehensive genomic profiling in diabetic nephropathy reveals the predominance of proinflammatory pathways.Transforming growth factor-β-induced cross talk between p53 and a microRNA in the pathogenesis of diabetic nephropathy Central Nodes in Protein Interaction Networks Drive Critical Functions in Transforming Growth Factor Beta-1 Stimulated Kidney Cells.Grape seed extract attenuates arsenic-induced nephrotoxicity in rats.Genome-wide transcriptional analysis of differentially expressed genes in diabetic, healing corneal epithelial cells: hyperglycemia-suppressed TGFβ3 expression contributes to the delay of epithelial wound healing in diabetic corneas.PHF14: an innate inhibitor against the progression of renal fibrosis following folic acid-induced kidney injury.Klotho as a potential biomarker and therapy for acute kidney injury.Klotho and renal fibrosis Knockdown of elF3a inhibits collagen synthesis in renal fibroblasts via Inhibition of transforming growth factor-β1/Smad signaling pathway Time-resolved dissection of early phosphoproteome and ensuing proteome changes in response to TGF-β.Gender-specific role of HDAC11 in kidney ischemia- and reperfusion-induced PAI-1 expression and injury.The molecular biology of pelvi-ureteric junction obstruction.Age-dependent shifts in renal response to injury relate to altered BMP6/CTGF expression and signaling.Effect of testosterone therapy on the urinary bladder in experimental hypogonadism of rats.Protective effect of rosuvastatin treatment by regulating oxidized low-density lipoprotein expression in a rat model of liver fibrosis Transforming growth factor-β1/Smad3-independent epithelial-mesenchymal transition in type I collagen glomerulopathy.Loss of expression of protein phosphatase magnesium-dependent 1A during kidney injury promotes fibrotic maladaptive repair.Complex Regulation of the Pericellular Proteolytic Microenvironment during Tumor Progression and Wound Repair: Functional Interactions between the Serine Protease and Matrix Metalloproteinase Cascades.Ureteral Obstruction-Induced Renal Fibrosis: An In Vivo Platform for Mechanistic Discovery and Therapeutic Intervention.Cell-Based Therapies for Tissue Fibrosis.A nonclassical vitamin D receptor pathway suppresses renal fibrosis.Bcl-3 is a novel biomarker of renal fibrosis in chronic kidney disease.Deregulation of Hippo-TAZ pathway during renal injury confers a fibrotic maladaptive phenotype.Roles of p21, p53, cyclin D1, CDK-4, estrogen receptor α in aflatoxin B1-induced cytotoxicity in testicular tissue of mice.Thermodynamic Aspects and Reprogramming Cellular Energy Metabolism during the Fibrosis Process.TGF-β1/p53 signaling in renal fibrogenesis.

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TGF-β1 → SMAD/p53/USF2 → PAI-1 transcriptional axis in ureteral obstruction-induced renal fibrosis.

http://www.wikidata.org/entity/Q37884486

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 04 June 2011

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

TGF-β1 → SMAD/p53/USF2 → PAI-1 ...... uction-induced renal fibrosis.

@en

TGF-β1 → SMAD/p53/USF2 → PAI-1 ...... uction-induced renal fibrosis.

@nl

type

label

TGF-β1 → SMAD/p53/USF2 → PAI-1 ...... uction-induced renal fibrosis.

@en

TGF-β1 → SMAD/p53/USF2 → PAI-1 ...... uction-induced renal fibrosis.

@nl

prefLabel

TGF-β1 → SMAD/p53/USF2 → PAI-1 ...... uction-induced renal fibrosis.

@en

TGF-β1 → SMAD/p53/USF2 → PAI-1 ...... uction-induced renal fibrosis.

@nl

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Cell and Tissue Research

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TGF-β1 → SMAD/p53/USF2 → PAI-1 ...... uction-induced renal fibrosis.

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Jessica M Overstreet

http://www.w3.org/2001/XMLSchema#string

Paul J Higgins

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Rohan Samarakoon

http://www.w3.org/2001/XMLSchema#string

Stephen P Higgins

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P2860

TGF-beta signal transduction

Epithelial-mesenchymal transition and its implications for fibrosis

Synergistic cooperation of TFE3 and smad proteins in TGF-beta-induced transcription of the plasminogen activator inhibitor-1 gene

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

Bone morphogenetic protein-7 (BMP7) in chronic kidney disease

Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE)

Kielin/chordin-like protein, a novel enhancer of BMP signaling, attenuates renal fibrotic disease

Imatinib mesylate blocks a non-Smad TGF-beta pathway and reduces renal fibrogenesis in vivo

Blinded by the Light: The Growing Complexity of p53

Direct binding of Smad3 and Smad4 to critical TGF beta-inducible elements in the promoter of human plasminogen activator inhibitor-type 1 gene.

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s00441-011-1181-y

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10.1007/S00441-011-1181-Y

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3188682

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