The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy.
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Epithelial-to-Mesenchymal Transition in Diabetic Nephropathy: Fact or Fiction?N-acetyl-seryl-aspartyl-lysyl-proline: a valuable endogenous anti-fibrotic peptide for combating kidney fibrosis in diabetes'Special k' and a loss of cell-to-cell adhesion in proximal tubule-derived epithelial cells: modulation of the adherens junction complex by ketamineMicroRNA-590 is an EMT-suppressive microRNA involved in the TGFβ signaling pathway.Effects of Chinese Medicine Tong xinluo on Diabetic Nephropathy via Inhibiting TGF- β 1-Induced Epithelial-to-Mesenchymal Transition.HRD1-Mediated IGF-1R Ubiquitination Contributes to Renal Protection of Resveratrol in db/db Mice.Representing kidney development using the gene ontology.Defective CFTR leads to aberrant β-catenin activation and kidney fibrosis.Distinct mesenchymal alterations in N-cadherin and E-cadherin positive primary renal epithelial cellsThe effects of the water-extraction of Astragali Radix and Lycopi herba on the Pathway of TGF-smads-UPP in a rat model of Diabetic Nephropathy.Norcantharidin inhibits renal interstitial fibrosis by blocking the tubular epithelial-mesenchymal transition.c-Myc is essential to prevent endothelial pro-inflammatory senescent phenotype.Transition of mesothelial cell to fibroblast in peritoneal dialysis: EMT, stem cell or bystander?MiRNA 17 family regulates cisplatin-resistant and metastasis by targeting TGFbetaR2 in NSCLC.Hyperglycemia in the absence of cilia accelerates cystogenesis and induces renal damageMycophenolate mofetil ameliorates diabetic nephropathy through epithelial mesenchymal transition in ratsOxymatrine Inhibits Renal Tubular EMT Induced by High Glucose via Upregulation of SnoN and Inhibition of TGF-β1/Smad Signaling Pathway.Substrate-specific gene expression profiles in different kidney cell types are associated with Fabry diseaseMiR-30a Inhibits the Epithelial--Mesenchymal Transition of Podocytes through Downregulation of NFATc3SnoN upregulation ameliorates renal fibrosis in diabetic nephropathyThe insulin response integrates increased TGF-β signaling through Akt-induced enhancement of cell surface delivery of TGF-β receptors.Xiaokeping mixture inhibits diabetic nephropathy in streptozotocin-induced rats through blocking TGF-β1/Smad7 signalingDiabetic nephropathy: the role of inflammation in fibroblast activation and kidney fibrosis.Hypoxia-inducible factor prolyl-hydroxylase-2 mediates transforming growth factor beta 1-induced epithelial-mesenchymal transition in renal tubular cells.Transforming growth factor-β1 and diabetic nephropathyNegative regulatory responses to metabolically triggered inflammation impair renal epithelial immunity in diabetes mellitus.Novel Tubular Biomarkers Predict Renal Progression in Type 2 Diabetes Mellitus: A Prospective Cohort Study.γ-Secretase inhibition promotes fibrotic effects of albumin in proximal tubular epithelial cells.The coordinated roles of miR-26a and miR-30c in regulating TGFβ1-induced epithelial-to-mesenchymal transition in diabetic nephropathy.Prolyl oligopeptidase attenuates hepatic stellate cell activation through induction of Smad7 and PPAR-γ.Review and discussion of tubular biomarkers in the diagnosis and management of diabetic nephropathy.Sodium-Glucose Cotransporter 2 Inhibitors with Renoprotective Effects.Astragalus membranaceus ameliorates renal interstitial fibrosis by inhibiting tubular epithelial-mesenchymal transition in vivo and in vitro.Role of TGF-β1/p38 MAPK pathway in hepatitis B virus-induced tubular epithelial-myofibroblast transdifferentiationRegulation of transforming growth factor beta 1 gene expression by dihydropteridine reductase in kidney 293T cells.Recombinant fibromodulin has therapeutic effects on diabetic nephropathy by down-regulating transforming growth factor-β1 in streptozotocin-induced diabetic rat model.Mesenchymal stem cell therapy ameliorates diabetic nephropathy via the paracrine effect of renal trophic factors including exosomes.Blockade of Orai1 Store-Operated Calcium Entry Protects against Renal Fibrosis.Integrin β4 in EMT: an implication of renal diseases.ALA/LA ameliorates glucose toxicity on HK-2 cells by attenuating oxidative stress and apoptosis through the ROS/p38/TGF-β1 pathway.
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The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
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name
The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy.
@en
The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy.
@nl
type
label
The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy.
@en
The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy.
@nl
prefLabel
The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy.
@en
The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy.
@nl
P1476
The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy
@en
P2093
Claire E Hills
Paul E Squires
P304
P356
10.1016/J.CYTOGFR.2011.06.002
P577
2011-06-01T00:00:00Z