Genetic analysis of neuronal ionotropic glutamate receptor subunits.
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Deep molecular diversity of mammalian synapses: why it matters and how to measure itPresynaptic NMDA receptors - dynamics and distribution in developing axons in vitro and in vivoDeveloping and applying the adverse outcome pathway concept for understanding and predicting neurotoxicityPotentiation of NMDA receptor-mediated transmission in striatal cholinergic interneuronsAcoustic trauma slows AMPA receptor-mediated EPSCs in the auditory brainstem, reducing GluA4 subunit expression as a mechanism to rescue binaural functionLTD expression is independent of glutamate receptor subtype.CRISPR/Cas9-mediated gene knock-down in post-mitotic neurons.SAP102 mediates synaptic clearance of NMDA receptors.Alcohol and NMDA receptor: current research and future direction.Neuronal autoantigens--pathogenesis, associated disorders and antibody testingLTP requires a reserve pool of glutamate receptors independent of subunit type.Glutamate receptor abnormalities in schizophrenia: implications for innovative treatments.Expression mechanisms underlying long-term potentiation: a postsynaptic view, 10 years onRegulation of dendritic development by semaphorin 3A through novel intracellular remote signaling.Adverse outcome pathways: Application to enhance mechanistic understanding of neurotoxicity.Synaptic molecular imaging in spared and deprived columns of mouse barrel cortex with array tomography.Plasticity of calcium-permeable AMPA glutamate receptors in Pro-opiomelanocortin neurons.Nature's Electric Potential: A Systematic Review of the Role of Bioelectricity in Wound Healing and Regenerative Processes in Animals, Humans, and Plants.Subunit-specific role for the amino-terminal domain of AMPA receptors in synaptic targeting.Abstinence from Cocaine-Induced Conditioned Place Preference Produces Discrete Changes in Glutamatergic Synapses onto Deep Layer 5/6 Neurons from Prelimbic and Infralimbic Cortices.Synaptic Potentiation at Basal and Apical Dendrites of Hippocampal Pyramidal Neurons Involves Activation of a Distinct Set of Extracellular and Intracellular Molecular Cues.Development of the Adverse Outcome Pathway (AOP): Chronic binding of antagonist to N-methyl-d-aspartate receptors (NMDARs) during brain development induces impairment of learning and memory abilities of children.
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Genetic analysis of neuronal ionotropic glutamate receptor subunits.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 18 July 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Genetic analysis of neuronal ionotropic glutamate receptor subunits.
@en
Genetic analysis of neuronal ionotropic glutamate receptor subunits.
@nl
type
label
Genetic analysis of neuronal ionotropic glutamate receptor subunits.
@en
Genetic analysis of neuronal ionotropic glutamate receptor subunits.
@nl
prefLabel
Genetic analysis of neuronal ionotropic glutamate receptor subunits.
@en
Genetic analysis of neuronal ionotropic glutamate receptor subunits.
@nl
P2860
P1476
Genetic analysis of neuronal ionotropic glutamate receptor subunits
@en
P2093
Adam J Granger
Roger A Nicoll
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P304
P356
10.1113/JPHYSIOL.2011.213033
P407
P577
2011-07-18T00:00:00Z