Intraneuronal Aβ as a trigger for neuron loss: can this be translated into human pathology? - Wikidata - awgldk - TriplyDB

Intraneuronal Aβ as a trigger for neuron loss: can this be translated into human pathology?

Intraneuronal Aβ as a trigger for neuron loss: can this be translated into human pathology?

http://www.wikidata.org/entity/Q37905118

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Risk factor SORL1: from genetic association to functional validation in Alzheimer's disease An apolipoprotein E4 fragment affects matrix metalloproteinase 9, tissue inhibitor of metalloproteinase 1 and cytokine levels in brain cell lines.Alzheimer's disease-associated peptide Aβ42 mobilizes ER Ca(2+) via InsP3R-dependent and -independent mechanisms.The systemic iron-regulatory proteins hepcidin and ferroportin are reduced in the brain in Alzheimer's disease.Pyroglutamate-Modified Amyloid Beta Peptides: Emerging Targets for Alzheimer´s Disease Immunotherapy.Biomarkers in Sporadic and Familial Alzheimer's Disease.Diesel engine exhaust accelerates plaque formation in a mouse model of Alzheimer's disease.Role of LDL cholesterol and endolysosomes in amyloidogenesis and Alzheimer's disease.Early intraneuronal accumulation and increased aggregation of phosphorylated Abeta in a mouse model of Alzheimer's disease.

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Intraneuronal Aβ as a trigger for neuron loss: can this be translated into human pathology?

http://www.wikidata.org/entity/Q37905118

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Intraneuronal Aβ as a trigger ...... anslated into human pathology?

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Intraneuronal Aβ as a trigger ...... anslated into human pathology?

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Intraneuronal Aβ as a trigger ...... anslated into human pathology?

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Intraneuronal Aβ as a trigger ...... anslated into human pathology?

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Intraneuronal Aβ as a trigger ...... anslated into human pathology?

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Intraneuronal Aβ as a trigger ...... anslated into human pathology?

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Biochemical Society Transactions

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Intraneuronal Aβ as a trigger ...... anslated into human pathology?

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Oliver Wirths

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Thomas A Bayer

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A Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Disease

Amyloid plaque core protein in Alzheimer disease and Down syndrome

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Active and passive immunotherapy for neurodegenerative disorders

Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

Neuropathological stageing of Alzheimer-related changes

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

Globular amyloid beta-peptide oligomer - a homogenous and stable neuropathological protein in Alzheimer's disease

Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway

A specific amyloid-beta protein assembly in the brain impairs memory

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857-861

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scholarly article

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10.1042/BST0390857

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4

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39

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2011-08-01T00:00:00Z

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51519442

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21787313

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