Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia. - Wikidata - awgldk - TriplyDB

Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

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The role of maintenance therapy in acute promyelocytic leukemia in the first complete remission Arsenite selectively inhibits mouse bone marrow lymphoid progenitor cell development in vivo and in vitro and suppresses humoral immunity in vivo Sequential activation of Elk-1/Egr-1/GADD45α by arsenic Arsenic trioxide prevents osteosarcoma growth by inhibition of GLI transcription via DNA damage accumulation Elemental and chemically specific X-ray fluorescence imaging of biological systems Metabolism, toxicity and anticancer activities of arsenic compounds.Synergistic inhibition of leukemia WEHI-3 cell growth by arsenic trioxide and Hedyotis diffusa Willd extract in vitro and in vivo.Tetraarsenictetrasulfide and Arsenic Trioxide Exert Synergistic Effects on Induction of Apoptosis and Differentiation in Acute Promyelocytic Leukemia Cells.Arsenic Primes Human Bone Marrow CD34+ Cells for Erythroid Differentiation Utilization of arsenic trioxide as a treatment of cisplatin-resistant non-small cell lung cancer PC-9/CDDP and PC-14/CDDP cells.Addition of Arsenic Trioxide into Induction Regimens Could Not Accelerate Recovery of Abnormality of Coagulation and Fibrinolysis in Patients with Acute Promyelocytic Leukemia Compound Library Screening Identified Cardiac Glycoside Digitoxin as an Effective Growth Inhibitor of Gefitinib-Resistant Non-Small Cell Lung Cancer via Downregulation of α-Tubulin and Inhibition of Microtubule Formation.Arsenic trioxide inhibits cell growth and induces apoptosis through inactivation of notch signaling pathway in breast cancer.Dose-dependent effects of selenite (Se(4+)) on arsenite (As(3+))-induced apoptosis and differentiation in acute promyelocytic leukemia cells.Outcome of elderly patients with acute promyelocytic leukemia: results of the German Acute Myeloid Leukemia Cooperative Group.Antioxidant Potential of Spirulina platensis Mitigates Oxidative Stress and Reprotoxicity Induced by Sodium Arsenite in Male Rats.Arsenic trioxide disrupts glioma stem cells via promoting PML degradation to inhibit tumor growth Salvianolic Acid B prevents arsenic trioxide-induced cardiotoxicity in vivo and enhances its anticancer activity in vitro.From molecular interaction to acute promyelocytic leukemia: Calculating leukemogenesis and remission from endogenous molecular-cellular network.The evolution of arsenic in the treatment of acute promyelocytic leukemia and other myeloid neoplasms: Moving toward an effective oral, outpatient therapy.Extracellular DNA traps released by acute promyelocytic leukemia cells through autophagy Arsenic trioxide and other arsenical compounds inhibit the NLRP1, NLRP3, and NAIP5/NLRC4 inflammasomes.Management of elderly patients with acute promyelocytic leukemia: progress and problems.Treatment of acute promyelocytic leukemia with arsenic trioxide: clinical results and open questions.Treatment of an acute promyelocytic leukemia relapse using arsenic trioxide and all-trans-retinoic in a 6-year-old child.RARA fusion genes in acute promyelocytic leukemia: a review.What is the standard regimen for patients with acute promyelocytic leukemia?Progress in the treatment of acute promyelocytic leukemia: optimization and obstruction.Current standard treatment of adult acute promyelocytic leukaemia.Activating transcription factor 4 underlies the pathogenesis of arsenic trioxide-mediated impairment of macrophage innate immune functions.Curcumin reduces the expression of survivin, leading to enhancement of arsenic trioxide-induced apoptosis in myelodysplastic syndrome and leukemia stem-like cells Low-dose arsenic trioxide combined with aclacinomycin A synergistically enhances the cytotoxic effect on human acute myelogenous leukemia cell lines by induction of apoptosis.Autologous transplant remains the preferred therapy for relapsed APL in CR2.Arsenic trioxide-based therapy of relapsed acute promyelocytic leukemia: registry results from the European LeukemiaNet.Retinoid differentiation therapy for common types of acute myeloid leukemia.Role of cysteine 288 in nucleophosmin cytoplasmic mutations: sensitization to toxicity induced by arsenic trioxide and bortezomib.A single course of all-trans retinoic acid plus arsenic trioxide reached a long-term survival in a patient with newly diagnosed acute promyelocytic leukemia: a suggestion for reduction of treatment courses?Identification of metal-associated proteins in cells by using continuous-flow gel electrophoresis and inductively coupled plasma mass spectrometry.Metformin ameliorates arsenic trioxide hepatotoxicity via inhibiting mitochondrial complex I.Inhibition of STAT3/VEGF/CDK2 axis signaling is critically involved in the antiangiogenic and apoptotic effects of arsenic herbal mixture PROS in non-small lung cancer cells.

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Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

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Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

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Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

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type

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Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

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Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

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Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

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Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

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Impact of arsenic trioxide in the treatment of acute promyelocytic leukemia.

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P2860

Rearrangements of the retinoic acid receptor alpha and promyelocytic leukemia zinc finger genes resulting from t(11;17)(q23;q21) in a patient with acute promyelocytic leukemia

Missense mutations in PML-RARA are critical for the lack of responsiveness to arsenic trioxide treatment.

Chromosomal translocation t(15;17) in human acute promyelocytic leukemia fuses RAR alpha with a novel putative transcription factor, PML

United States multicenter study of arsenic trioxide in relapsed acute promyelocytic leukemia

Fusion of retinoic acid receptor alpha to NuMA, the nuclear mitotic apparatus protein, by a variant translocation in acute promyelocytic leukaemia

Proposals for the classification of the acute leukaemias. French-American-British (FAB) co-operative group

The "retinoic acid syndrome" in acute promyelocytic leukemia

Arsenic inhibition of telomerase transcription leads to genetic instability

Arsenic-induced PML targeting onto nuclear bodies: implications for the treatment of acute promyelocytic leukemia

Retinoic acid, but not arsenic trioxide, degrades the PLZF/RARalpha fusion protein, without inducing terminal differentiation or apoptosis, in a RA-therapy resistant t(11;17)(q23;q21) APL patient.

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433-442

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10.1038/LEU.2011.245

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3

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26

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21904379

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