Defective DNA-damage repair induced by nuclear lamina dysfunction is a key mediator of smooth muscle cell aging.
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Lamins at a glance.Oxydative stress alters nuclear shape through lamins dysregulation: a route to senescence.Vein graft neointimal hyperplasia is exacerbated by CXCR4 signaling in vein graft-extrinsic cells.Mechanisms of vascular calcification in CKD-evidence for premature ageing?Disruption of PCNA-lamins A/C interactions by prelamin A induces DNA replication fork stalling.Prelamin A impairs 53BP1 nuclear entry by mislocalizing NUP153 and disrupting the Ran gradient
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Defective DNA-damage repair induced by nuclear lamina dysfunction is a key mediator of smooth muscle cell aging.
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artículo científico
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name
Defective DNA-damage repair in ...... r of smooth muscle cell aging.
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Defective DNA-damage repair in ...... r of smooth muscle cell aging.
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type
label
Defective DNA-damage repair in ...... r of smooth muscle cell aging.
@en
Defective DNA-damage repair in ...... r of smooth muscle cell aging.
@nl
prefLabel
Defective DNA-damage repair in ...... r of smooth muscle cell aging.
@en
Defective DNA-damage repair in ...... r of smooth muscle cell aging.
@nl
P2860
P356
P1476
Defective DNA-damage repair in ...... r of smooth muscle cell aging.
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P2093
Catherine M Shanahan
Derek T Warren
P2860
P304
P356
10.1042/BST20110703
P577
2011-12-01T00:00:00Z