The senescence accelerated mouse (SAMP8) as a model for oxidative stress and Alzheimer's disease.
about
The 2013 SFRBM discovery award: selected discoveries from the butterfield laboratory of oxidative stress and its sequela in brain in cognitive disorders exemplified by Alzheimer disease and chemotherapy induced cognitive impairmentRecovery of Cognitive Dysfunction via Orally Administered Redox-Polymer Nanotherapeutics in SAMP8 MiceDynamics of Human Mitochondrial Complex I Assembly: Implications for Neurodegenerative DiseasesSenescence-accelerated OXYS rats: a model of age-related cognitive decline with relevance to abnormalities in Alzheimer disease.Rcor2 underexpression in senescent mice: a target for inflammaging?Electroacupuncture Treatment Improves Learning-Memory Ability and Brain Glucose Metabolism in a Mouse Model of Alzheimer's Disease: Using Morris Water Maze and Micro-PET.A comprehensive multiomics approach toward understanding the relationship between aging and dementia.Amyloid accumulation is a late event in sporadic Alzheimer's disease-like pathology in nontransgenic rats.Age-related alterations in the metabolic profile in the hippocampus of the senescence-accelerated mouse prone 8: a spontaneous Alzheimer's disease mouse model.Musical Electroacupuncture May Be a Better Choice than Electroacupuncture in a Mouse Model of Alzheimer's DiseaseSomatostatin receptor subtype-4 agonist NNC 26-9100 mitigates the effect of soluble Aβ(42) oligomers via a metalloproteinase-dependent mechanismNodes and biological processes identified on the basis of network analysis in the brain of the senescence accelerated mice as an Alzheimer's disease animal model.Neuroendocrine immunomodulation network dysfunction in SAMP8 mice and PrP-hAβPPswe/PS1ΔE9 mice: potential mechanism underlying cognitive impairment.Behavioral and omics analyses study on potential involvement of dipeptide balenine through supplementation in diet of senescence-accelerated mouse prone 8.Environmental Enrichment Modified Epigenetic Mechanisms in SAMP8 Mouse Hippocampus by Reducing Oxidative Stress and Inflammaging and Achieving NeuroprotectionMetabolic Dysfunction of Astrocyte: An Initiating Factor in Beta-amyloid Pathology?LW-AFC Effects on N-glycan Profile in Senescence-Accelerated Mouse Prone 8 Strain, a Mouse Model of Alzheimer's Disease.Epigenetic alterations in hippocampus of SAMP8 senescent mice and modulation by voluntary physical exercise.Longitudinal analysis of calorie restriction on rat taste bud morphology and expression of sweet taste modulatorsNeuroprotective effect of the Chinese medicine Tiantai No. 1 and its molecular mechanism in the senescence-accelerated mouse prone 8Oxidative stress in aging--matters of the heart and mind.From Neurodegeneration to Brain Health: An Integrated Approach.Activation of the 5'-AMP-Activated Protein Kinase in the Cerebral Cortex of Young Senescence-Accelerated P8 Mice and Association with GSK3β- and PP2A-Dependent Inhibition of p-tau₃₉₆ Expression.The Effects of LW-AFC on the Hippocampal Transcriptome in Senescence-Accelerated Mouse Prone 8 Strain, a Mouse Model of Alzheimer's Disease.The SAMP8 mouse for investigating memory and the role of insulin in the brain.An antioxidant specifically targeting mitochondria delays progression of Alzheimer's disease-like pathology.The Effects of LW-AFC on Intestinal Microbiome in Senescence-Accelerated Mouse Prone 8 Strain, a Mouse Model of Alzheimer's Disease.Proteome response at the edge of protein aggregation.Antisense directed against PS-1 gene decreases brain oxidative markers in aged senescence accelerated mice (SAMP8) and reverses learning and memory impairment: a proteomics studyAngiotensin-(1-7) is Reduced and Inversely Correlates with Tau Hyperphosphorylation in Animal Models of Alzheimer's Disease.Fisetin Reduces the Impact of Aging on Behavior and Physiology in the Rapidly Aging SAMP8 Mouse.SIRT1 Regulates Cognitive Performance and Ability of Learning and Memory in Diabetic and Nondiabetic Models.TDP-43 accelerates age-dependent degeneration of interneurons.The mitochondrial ATP synthase is a shared drug target for aging and dementia.Environmental Enrichment Improves Behavior, Cognition, and Brain Functional Markers in Young Senescence-Accelerated Prone Mice (SAMP8).Central Nervous System Delivery of Intranasal Insulin: Mechanisms of Uptake and Effects on Cognition.Astrocyte and Alzheimer's disease.Coenzyme Q10 as a therapeutic candidate for treating inherited photoreceptor degeneration.Editorial: Geriatrics in the 21st Century.Aging, inflammation and the environment.
P2860
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P2860
The senescence accelerated mouse (SAMP8) as a model for oxidative stress and Alzheimer's disease.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
The senescence accelerated mou ...... tress and Alzheimer's disease.
@en
The senescence accelerated mouse
@nl
type
label
The senescence accelerated mou ...... tress and Alzheimer's disease.
@en
The senescence accelerated mouse
@nl
prefLabel
The senescence accelerated mou ...... tress and Alzheimer's disease.
@en
The senescence accelerated mouse
@nl
P2093
P1476
The senescence accelerated mou ...... tress and Alzheimer's disease.
@en
P2093
Harvey James Armbrecht
John E Morley
Susan A Farr
Vijaya B Kumar
P304
P356
10.1016/J.BBADIS.2011.11.015
P407
P577
2011-11-26T00:00:00Z