Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade. - Wikidata - awgldk - TriplyDB

Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

http://www.wikidata.org/entity/Q37984182

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AAV-mediated gene therapy for heart failure: enhancing contractility and calcium handling A compartmentalized mathematical model of the β1-adrenergic signaling system in mouse ventricular myocytes Modeling the effects of β1-adrenergic receptor blockers and polymorphisms on cardiac myocyte Ca2+ handling.G protein-coupled Receptor Kinase 2 as a Therapeutic Target for Heart Failure.Gene targeting in ischemic heart disease and failure: translational and clinical studies.Altered sarcoplasmic reticulum calcium cycling--targets for heart failure therapy.Gene therapy for the treatment of heart failure: promise postponed GRK 2 level in peripheral blood lymphocytes of elderly patients with acute myocardial infarction Pathological ventricular remodeling: therapies: part 2 of 2.Structural and functional plasticity in long-term cultures of adult ventricular myocytes GRK2 as a therapeutic target for heart failure.Peptide substrates for G protein-coupled receptor kinase 2.

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Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

http://www.wikidata.org/entity/Q37984182

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article científic

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article scientifique

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artikel ilmiah

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name

Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

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Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

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Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

@en

Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

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Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

@en

Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

@nl

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Targeting GRK2 by gene therapy for heart failure: benefits above β-blockade.

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H A Katus

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H Tscheschner

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J Reinkober

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P Most

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P W J Raake

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S T Pleger

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W J Koch

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P2860

Targeted beta-adrenergic receptor kinase (betaARK1) inhibition by gene transfer in failing human hearts

Myocardial adeno-associated virus serotype 6-betaARKct gene therapy improves cardiac function and normalizes the neurohormonal axis in chronic heart failure

Expression of a beta-adrenergic receptor kinase 1 inhibitor prevents the development of myocardial failure in gene-targeted mice

Alterations in cardiac adrenergic signaling and calcium cycling differentially affect the progression of cardiomyopathy

Keeping G proteins at bay: a complex between G protein-coupled receptor kinase 2 and Gbetagamma

Cardiac AAV9-S100A1 gene therapy rescues post-ischemic heart failure in a preclinical large animal model

Modulation of adrenal catecholamine secretion by in vivo gene transfer and manipulation of G protein-coupled receptor kinase-2 activity

G protein-coupled receptor kinase 2 ablation in cardiac myocytes before or after myocardial infarction prevents heart failure.

The inotropic peptide βARKct improves βAR responsiveness in normal and failing cardiomyocytes through G(βγ)-mediated L-type calcium current disinhibition.

Mechanisms and models in heart failure: A combinatorial approach.

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686-693

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scholarly article

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10.1038/GT.2012.9

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6

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19

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2012-02-16T00:00:00Z

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221834521

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22336718

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