From mitochondrial dysfunction to amyloid beta formation: novel insights into the pathogenesis of Alzheimer's disease.
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The Amyloid Precursor Protein-A Novel Player within the Molecular Array of Presynaptic NanomachinesThe Role of Oxidative Stress-Induced Epigenetic Alterations in Amyloid-β Production in Alzheimer's DiseasePhysicochemical properties of cells and their effects on intrinsically disordered proteins (IDPs)ROS and brain diseases: the good, the bad, and the uglyFormaldehyde in brain: an overlooked player in neurodegeneration?APP Is a Context-Sensitive Regulator of the Hippocampal Presynaptic Active ZoneMitochondrial DNA damage in a mouse model of Alzheimer's disease decreases amyloid beta plaque formationMitochondrial protection by the mixed muscarinic/σ1 ligand ANAVEX2-73, a tetrahydrofuran derivative, in Aβ25-35 peptide-injected mice, a nontransgenic Alzheimer's disease model.Deer bone extract prevents against scopolamine-induced memory impairment in miceIdentification of tetrahydrocarbazoles as novel multifactorial drug candidates for treatment of Alzheimer's disease.Centella asiatica Attenuates Amyloid-β-Induced Oxidative Stress and Mitochondrial DysfunctionA γ-Secretase Independent Role for Presenilin in Calcium Homeostasis Impacts Mitochondrial Function and Morphology in Caenorhabditis elegans.Prophylactic melatonin significantly reduces Alzheimer's neuropathology and associated cognitive deficits independent of antioxidant pathways in AβPP(swe)/PS1 mice.Functional microRNAs in Alzheimer's disease and cancer: differential regulation of common mechanisms and pathways.Alterations in mitochondrial number and function in Alzheimer's disease fibroblastsDysregulated relationship of inflammation and oxidative stress in major depression.Beneficial effect of diosgenin as a stimulator of NGF on the brain with neuronal damage induced by Aβ-42 accumulation and neurotoxicant injection.STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.APP Deletion Accounts for Age-Dependent Changes in the Bioenergetic Metabolism and in Hyperphosphorylated CaMKII at Stimulated Hippocampal Presynaptic Active Zones.Nutritional contributions to dementia prevention: main issues on antioxidant micronutrients.Mitochondrion-mediated cell death: dissecting yeast apoptosis for a better understanding of neurodegeneration.The intersection of amyloid β and tau in glutamatergic synaptic dysfunction and collapse in Alzheimer's disease.From inventory to functional mechanisms: regulation of the mitochondrial protein import machinery by phosphorylation.Inflammation and programmed cell death in Alzheimer's disease: comparison of the central nervous system and peripheral blood.Dissecting Complex and Multifactorial Nature of Alzheimer's Disease Pathogenesis: a Clinical, Genomic, and Systems Biology Perspective.Mitochondrial dysfunction: the missing link between aging and sporadic Alzheimer's disease.DADS Analogues Ameliorated the Cognitive Impairments of Alzheimer-Like Rat Model Induced by Scopolamine.Physical Exercise and Brain Mitochondrial Fitness: The Possible Role Against Alzheimer's Disease.Amyloid β regulates the expression and function of AIP1.Therapeutic efficacy of the Ginkgo special extract EGb761® within the framework of the mitochondrial cascade hypothesis of Alzheimer's disease.Cerebrospinal fluid lactate levels and brain [18F]FDG PET hypometabolism within the default mode network in Alzheimer's disease.Staging of cognitive deficits and neuropathological and ultrastructural changes in streptozotocin-induced rat model of Alzheimer's disease.Centella asiatica Attenuates Mitochondrial Dysfunction and Oxidative Stress in Aβ-Exposed Hippocampal Neurons.Inherited real risk of Alzheimer's disease: bedside diagnosis and primary prevention.Formaldehyde metabolism and formaldehyde-induced stimulation of lactate production and glutathione export in cultured neurons.Naringenin Attenuates H2O2-Induced Mitochondrial Dysfunction by an Nrf2-Dependent Mechanism in SH-SY5Y Cells.Mitochondrial Translocase of the Outer Membrane Alterations May Underlie Dysfunctional Oxidative Phosphorylation in Alzheimer's Disease.Physical and Cognitive Stimulation Using an Exergame in Subjects with Normal Aging, Mild and Moderate Cognitive Impairment.A Cystine-Rich Whey Supplement (Immunocal®) Provides Neuroprotection from Diverse Oxidative Stress-Inducing Agents In Vitro by Preserving Cellular Glutathione.Brain testosterone deficiency leads to down-regulation of mitochondrial gene expression in rat hippocampus accompanied by a decline in peroxisome proliferator-activated receptor-γ coactivator 1α expression.
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From mitochondrial dysfunction to amyloid beta formation: novel insights into the pathogenesis of Alzheimer's disease.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
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artigo científico
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artikel ilmiah
@id
artikull shkencor
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artículo científico
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name
From mitochondrial dysfunction ...... enesis of Alzheimer's disease.
@en
type
label
From mitochondrial dysfunction ...... enesis of Alzheimer's disease.
@en
prefLabel
From mitochondrial dysfunction ...... enesis of Alzheimer's disease.
@en
P2860
P1476
From mitochondrial dysfunction ...... genesis of Alzheimer's disease
@en
P2093
Kristina Leuner
Walter E Müller
P2860
P2888
P304
P356
10.1007/S12035-012-8307-4
P577
2012-07-26T00:00:00Z
P6179
1000237137