Prenatal poly(i:C) exposure and other developmental immune activation models in rodent systems.
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Neuroinflammation in Autism: Plausible Role of Maternal Inflammation, Dietary Omega 3, and MicrobiotaMetabotropic Glutamate 2/3 Receptors and Epigenetic Modifications in Psychotic Disorders: A ReviewComprehensive Analysis of the 16p11.2 Deletion and Null Cntnap2 Mouse Models of Autism Spectrum Disorder.Maternal Immune Activation and Autism Spectrum Disorder: From Rodents to Nonhuman and Human Primates.Maternal immune activation: Implications for neuropsychiatric disorders.Dimensional assessment of behavioral changes in the cuprizone short-term exposure model for psychosisFunction and Dysfunction of Microglia during Brain Development: Consequences for Synapses and Neural CircuitsDifferent Paths to Core Pathology: The Equifinal Model of the Schizophrenia SyndromeAnimal models of gene-environment interaction in schizophrenia: A dimensional perspective.Down-Regulation of Hippocampal Genes Regulating Dopaminergic, GABAergic, and Glutamatergic Function Following Combined Neonatal Phencyclidine and Post-Weaning Social Isolation of Rats as a Neurodevelopmental Model for Schizophrenia.Nicotine ameliorates schizophrenia-like cognitive deficits induced by maternal LPS exposure: a study in rats.Maternal Immune Activation Disrupts Dopamine System in the OffspringPreventive effects of minocycline in a neurodevelopmental two-hit model with relevance to schizophrenia.Molecular substrates of schizophrenia: homeostatic signaling to connectivityBehavioral effects of the benzodiazepine-positive allosteric modulator SH-053-2'F-S-CH₃ in an immune-mediated neurodevelopmental disruption model.Continuous kynurenine administration during the prenatal period, but not during adolescence, causes learning and memory deficits in adult ratsHeightened fear in response to a safety cue and extinguished fear cue in a rat model of maternal immune activation.Allergic fetal priming leads to developmental, behavioral and neurobiological changes in miceMetabolic Profiling and Phenotyping of Central Nervous System Diseases: Metabolites Bring Insights into Brain Dysfunctions.Genome-Wide Transcriptional Profiling and Structural Magnetic Resonance Imaging in the Maternal Immune Activation Model of Neurodevelopmental Disorders.Evolution of structural abnormalities in the rat brain following in utero exposure to maternal immune activation: A longitudinal in vivo MRI study.Neuroimmune Interactions in Schizophrenia: Focus on Vagus Nerve Stimulation and Activation of the Alpha-7 Nicotinic Acetylcholine ReceptorBeyond the association. Toxoplasma gondii in schizophrenia, bipolar disorder, and addiction: systematic review and meta-analysis.Regulation of histone H3K4 methylation in brain development and disease.DNA methylation analysis of the autistic brain reveals multiple dysregulated biological pathways.Identifying novel interventional strategies for psychiatric disorders: integrating genomics, 'enviromics' and gene-environment interactions in valid preclinical models.Neurodevelopment, GABA system dysfunction, and schizophreniaCellular mechanisms of toll-like receptor-3 activation in the thalamus are associated with white matter injury in the developing brainPre- and neonatal exposure to lipopolysaccharide or the enteric metabolite, propionic acid, alters development and behavior in adolescent rats in a sexually dimorphic manner.Combined effect of TLR2 gene polymorphism and early life stress on the age at onset of bipolar disorders.The Association between Infections and General Cognitive Ability in Young Men - A Nationwide Study.Brain in flames - animal models of psychosis: utility and limitationsLPA signaling initiates schizophrenia-like brain and behavioral changes in a mouse model of prenatal brain hemorrhage.Cortisol and Inflammatory Biomarkers Predict Poor Treatment Response in First Episode Psychosis.Late prenatal immune activation causes hippocampal deficits in the absence of persistent inflammation across aging.Using human brain imaging studies as a guide toward animal models of schizophrenia.Induction of endogenous retroelements as a potential mechanism for mouse-specific drug-induced carcinogenicity.Maternal immune activation produces neonatal excitability defects in offspring hippocampal neurons from pregnant rats treated with poly I:CMaternal immune activation induces GAD1 and GAD2 promoter remodeling in the offspring prefrontal cortex.Maternal Inflammation Disrupts Fetal Neurodevelopment via Increased Placental Output of Serotonin to the Fetal Brain.
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Prenatal poly(i:C) exposure and other developmental immune activation models in rodent systems.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
@es
name
Prenatal poly(i:C) exposure an ...... tion models in rodent systems.
@en
type
label
Prenatal poly(i:C) exposure an ...... tion models in rodent systems.
@en
prefLabel
Prenatal poly(i:C) exposure an ...... tion models in rodent systems.
@en
P1476
Prenatal poly(i:C) exposure an ...... ation models in rodent systems
@en
P2093
P304
P356
10.1016/J.BIOPSYCH.2013.07.011
P407
P50
P577
2013-08-12T00:00:00Z