Antiangiogenic therapy for high-grade gliomas: current concepts and limitations.
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Current standards and new concepts in MRI and PET response assessment of antiangiogenic therapies in high-grade glioma patientsLoss of endothelial programmed cell death 10 activates glioblastoma cells and promotes tumor growth.Monoamine oxidase B levels are highly expressed in human gliomas and are correlated with the expression of HiF-1α and with transcription factors Sp1 and Sp3.Multi-targeting NGR-modified liposomes recognizing glioma tumor cells and vasculogenic mimicry for improving anti-glioma therapy.Recent advance in molecular angiogenesis in glioblastoma: the challenge and hope for anti-angiogenic therapy.Angiogenesis inhibitors in tackling recurrent glioblastoma.Monoamines in Glioblastoma: complex biology with therapeutic potential.In Vitro Assays for Endothelial Cell Functions Required for Angiogenesis: Proliferation, Motility, Tubular Differentiation, and Matrix Proteolysis.
P2860
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P2860
Antiangiogenic therapy for high-grade gliomas: current concepts and limitations.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年學術文章
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name
Antiangiogenic therapy for high-grade gliomas: current concepts and limitations.
@en
type
label
Antiangiogenic therapy for high-grade gliomas: current concepts and limitations.
@en
prefLabel
Antiangiogenic therapy for high-grade gliomas: current concepts and limitations.
@en
P2093
P2860
P1476
Antiangiogenic therapy for high-grade gliomas: current concepts and limitations.
@en
P2093
Angelo Pompucci
Annunziato Mangiola
Carmelo Anile
Simone Peraio
P2860
P304
P356
10.1586/14737175.2013.856264
P577
2013-11-01T00:00:00Z