Amyotrophic lateral sclerosis--a model of corticofugal axonal spread. - Wikidata - awgldk - TriplyDB

Amyotrophic lateral sclerosis--a model of corticofugal axonal spread.

Amyotrophic lateral sclerosis--a model of corticofugal axonal spread.

http://www.wikidata.org/entity/Q38161789

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Neurodegenerative diseases: expanding the prion concept The value of magnetic resonance imaging as a biomarker for amyotrophic lateral sclerosis: a systematic review Spreading of pathology in neurodegenerative diseases: a focus on human studies Eye Movement Deficits Are Consistent with a Staging Model of pTDP-43 Pathology in Amyotrophic Lateral Sclerosis The diagnostic value of midbrain hyperechogenicity in ALS is limited for discriminating key ALS differential diagnoses.Neuropathology of Amyotrophic Lateral Sclerosis and Its Variants.Formation and spreading of TDP-43 aggregates in cultured neuronal and glial cells demonstrated by time-lapse imaging.Memory deficits in amyotrophic lateral sclerosis are not exclusively caused by executive dysfunction: a comparative neuropsychological study of amnestic mild cognitive impairment Widespread grey matter pathology dominates the longitudinal cerebral MRI and clinical landscape of amyotrophic lateral sclerosis.Cerebro-cerebellar connectivity is increased in primary lateral sclerosis.Improving single injection CSF delivery of AAV9-mediated gene therapy for SMA: a dose-response study in mice and nonhuman primates.Autonomic system and amyotrophic lateral sclerosis.Microstructural changes across different clinical milestones of disease in amyotrophic lateral sclerosis.Frontotemporal lobar degeneration: defining phenotypic diversity through personalized medicine.TDP-43 pathology and neuronal loss in amyotrophic lateral sclerosis spinal cord Spread of pathology in amyotrophic lateral sclerosis: assessment of phosphorylated TDP-43 along axonal pathways.Perception of Emotional Facial Expressions in Amyotrophic Lateral Sclerosis (ALS) at Behavioural and Brain Metabolic Level.TDP-43 is intercellularly transmitted across axon terminals Super-Resolution Microscopy Reveals Presynaptic Localization of the ALS/FTD Related Protein FUS in Hippocampal Neurons.Telomere shortening leads to earlier age of onset in ALS mice In vitro prion-like behaviour of TDP-43 in ALS.Functional recovery in new mouse models of ALS/FTLD after clearance of pathological cytoplasmic TDP-43.Longitudinal imaging in C9orf72 mutation carriers: Relationship to phenotype.TDP-43 in the olfactory bulb in Alzheimer's disease.Pathological TDP-43 changes in Betz cells differ from those in bulbar and spinal α-motoneurons in sporadic amyotrophic lateral sclerosis.Anti-α-synuclein immunotherapy reduces α-synuclein propagation in the axon and degeneration in a combined viral vector and transgenic model of synucleinopathy.Sequential distribution of pTDP-43 pathology in behavioral variant frontotemporal dementia (bvFTD).Calretinin and Neuropeptide Y interneurons are differentially altered in the motor cortex of the SOD1G93A mouse model of ALS.TDP-43 in amyotrophic lateral sclerosis - is it a prion disease?The Role of TDP-43 in Alzheimer's Disease.Functional reorganization during cognitive function tasks in patients with amyotrophic lateral sclerosis.Computer-based magnetic resonance imaging as a tool in clinical diagnosis in neurodegenerative diseases.Epidemiology of amyotrophic lateral sclerosis in Southern Germany.Molecular neuropathology of frontotemporal dementia: insights into disease mechanisms from postmortem studies.The Axon Guidance Protein Semaphorin 3A Is Increased in the Motor Cortex of Patients With Amyotrophic Lateral Sclerosis.Pathogenesis of amyotrophic lateral sclerosis.Prion-like propagation as a pathogenic principle in frontotemporal dementia.Management and therapeutic perspectives in amyotrophic lateral sclerosis.Neurodegeneration and RNA-binding proteins.Neuroimaging Endpoints in Amyotrophic Lateral Sclerosis.

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Amyotrophic lateral sclerosis--a model of corticofugal axonal spread.

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Amyotrophic lateral sclerosis--a model of corticofugal axonal spread.

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Johannes Brettschneider

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Kelly Del Tredici

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Where, when, and in what form does sporadic Alzheimerʼs disease begin?

Non-invasive mapping of connections between human thalamus and cortex using diffusion imaging

A harmonized classification system for FTLD-TDP pathology

Converging mechanisms in ALS and FTD: disrupted RNA and protein homeostasis

Self-propagation of pathogenic protein aggregates in neurodegenerative diseases

Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis

Staging of brain pathology related to sporadic Parkinson's disease

TDP-43 transgenic mice develop spastic paralysis and neuronal inclusions characteristic of ALS and frontotemporal lobar degeneration

Loss of murine TDP-43 disrupts motor function and plays an essential role in embryogenesis.

The ALS disease protein TDP-43 is actively transported in motor neuron axons and regulates axon outgrowth.

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